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The protein encoded by CTNNBIP1 binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. 再加上，我们可以发CTNNBIP1 抗体 (49) 和 CTNNBIP1 试剂盒 (5)和数多这个蛋白质的别的产品。
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miR (显示 MLXIP 蛋白)-215-5p is a negative regulator of adipocyte differentiation through post-transcriptional regulation of FNDC3B and CTNNBIP1 during early adipogenesis
miR (显示 MLXIP 蛋白)-29b plays a pivotal role in fetal mouse neurogenesis by regulating ICAT-mediated Wnt (显示 WNT2 蛋白)/beta-catenin (显示 CTNNB1 蛋白) signaling.
Mechanistic studies revealed that CTNNBIP1 suppresses Wnt (显示 WNT2 蛋白)/beta-catenin (显示 CTNNB1 蛋白) signaling and that miR (显示 MLXIP 蛋白)-215 promotes beta-catenin (显示 CTNNB1 蛋白) activation and upregulates alpha-SMA (显示 SMN1 蛋白) and fibronectin (显示 FN1 蛋白) expression in TGF-beta1 (显示 TGFB1 蛋白)-treated MMCs by targeting CTNNBIP1
Overexpression of Icat induces G(2) arrest and cell death in tumor cell mutants for adenomatous polyposis coli (显示 APC 蛋白), beta-catenin (显示 CTNNB1 蛋白), or Axin (显示 AXIN1 蛋白).
ICAT plays an important role in the anteriorization of neural cells by inhibiting the posteriorizing activity of Wnt (显示 WNT2 蛋白) signaling
These results suggest that the loss of ICAT gene function causes the arrest of UB branching and the apoptotic death of MM cells, resulting in renal agenesis.
Inhibition of beta-catenin (显示 CTNNB1 蛋白) signaling in articular chondrocytes causes increased cell apoptosis and articular cartilage destruction in Col2a1 (显示 COL2A1 蛋白)-ICAT- transgenic mice.
Data demonstrated that ICAT was highly expressed in cervical cancer tissues and had a role in control of epithelial-mesenchymal transition (EMT) in cervical cancer cells. Its overexpression promoted cell proliferation, migration, invasion and resulted in EMT by disrupting the stability of E-cadherin/betacatenin complex.
Low PLD1 expression and high ICAT expression were significantly associated with increased survival in colorectal cancer patients.
The present work aims to investigate the relationship between the expression of AEG-1 (显示 MTDH 蛋白)(astrocyte elevated gene-1), b-FGF(basic-fibroblast growth factor (显示 FGF2 蛋白)), beta-catenin (显示 CTNNB1 蛋白), Ki-67 (显示 MKI67 蛋白), TNF-alpha (tumor necrosis factor (显示 TNF 蛋白)-alfa) other prognostic parameters in DC (Ductal Carcinomas) and ductal intraepithelial neoplasm. We found a relationship between these factors.
Overexpression of ICAT promoted Caski cells' proliferation, arrested the cell cycle in the S phase and enhanced cell migration. Conclusion Overexpression of ICAT can promote the proliferation and migration of Caski cervical cancer cells.
Somatic mutation of beta-catenin (CTNNB1 (显示 CTNNB1 蛋白)) is known to be crucial for Wilms tumor development in up to 15% of cases.
CTNNBIP1 expression correlated with longer overall survival in LAC (显示 LCT 蛋白) patients. This study reveals that miR (显示 MLXIP 蛋白)-214 plays a critical role in CSLC self-renewal and stemness by targeting CTNNBIP1.
Data show that microRNA miR (显示 MLXIP 蛋白)-215 activates beta-catenin (显示 CTNNB1 蛋白) pathways by decreasing catenin beta interacting protein 1 (CTNNBIP1)expression in gliomas.
Simultaneous silencing of beta-catenin (显示 CTNNB1 蛋白) and STAT3 (显示 STAT3 蛋白) synergistically induces apoptosis and inhibits cell proliferation in HepG2 liver cancer cells.
Finally, pro-incubation with idebenone inhibited mitochondrial dysfunction induced by oxLDL through the mitochondrial-dependent apoptotic pathway and GSK3beta/beta-catenin (显示 CTNNB1 蛋白) signalling pathways.
A potent beta-catenin (显示 CTNNB1 蛋白) inhibitor, ICAT/CTNNBIP1 was a direct target of miR (显示 MLXIP 蛋白)-424-5p.
The protein encoded by this gene binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. The encoded protein is a negative regulator of the Wnt signaling pathway. Two transcript variants encoding the same protein have been found for this gene.
, beta-catenin-interacting protein 1
, catenin, beta interacting protein 1 a
, catenin beta interacting protein 1 b
, catenin, beta interacting protein 1
, inhibitor of beta-catenin and Tcf-4
, beta-catenin-interacting protein ICAT