Use your antibodies-online credentials, if available.
抗Human MAPK12 抗体:
抗Mouse (Murine) MAPK12 抗体:
抗Rat (Rattus) MAPK12 抗体:
Cow (Bovine) Polyclonal MAPK12 Primary Antibody for WB - ABIN611036
Fulda, Meyer, Debatin: Inhibition of TRAIL-induced apoptosis by Bcl-2 overexpression. in Oncogene 2002
Show all 3 Pubmed References
Human Polyclonal MAPK12 Primary Antibody for PLA, WB - ABIN520005
Liu, Chen, Chau, Jan, Chen, Hsu, Lin, Juang, Lu, Cheng, Chen, Chang, Ting, Kao, Hsiao, Huang: Analysis of protein-protein interactions in cross-talk pathways reveals CRKL protein as a novel prognostic marker in hepatocellular carcinoma. in Molecular & cellular proteomics : MCP 2013
Study propose a model for sequential roles of MPK12, HT1, and GHR1 in the ABA-independent regulation of SLAC1 during CO2-induced stomatal closure.
MPK9 (显示 MAPK9 抗体) and MPK12 are positive regulators of salicylic acid signaling in Arabidopsis guard cells.
MPK9 (显示 MAPK9 抗体) and MPK12 are key regulators mediating both abscisic acid (ABA) and Methyl jasmonate (MeJA) signalling in guard cells.
MPK9 (显示 MAPK9 抗体) and MPK12 function redundantly downstream of extracellular reactive oxygen production and intracellular accumulation, cytosolic alkalisation and Ca(2 (显示 CA2 抗体)+)cytosolic oscillation in yeast elcictor-induced stomatal closure
MPK9 (显示 MAPK9 抗体) and MPK12 act downstream of ROS (显示 ROS1 抗体) and cytosolic Ca2 (显示 CA2 抗体)+ and upstream of anion channels in the guard cell abscisic acid signaling cascade.
MAP kinases MPK9 (显示 MAPK9 抗体) and MPK12 are preferentially expressed in guard cells and positively regulate ROS (显示 ROS1 抗体)-mediated ABA signaling.
MPK12 is both a physiological substrate of IBR5 and a novel negative regulator of auxin signaling.
There was significant association between p38gamma expression and esophageal squamous cell carcinoma clinical stage, lymph nodes metastases, and tumor volume. p38delta overexpression can promote tumorigenesis in nude mice model xenografted with Eca109 cells whose basal level of p38delta was stably over-expressed and p38gamma was stably knocked down.
This study reveals a novel pathway that directly links ErbB4 (显示 ERBB4 抗体) and p38gamma to the transcriptional machinery of NKx2.5 (显示 NKX2-5 抗体)-GATA4 (显示 GATA4 抗体) complex which is critical for cardiomyocyte formation during mammalian heart development.
during interphase ERK3 (显示 MAPK4 抗体) is mainly resident in the nucleoplasm in association with ribonuclear proteins involved in early pre-mRNA splicing, it undergoes cell cycle-dependent redistribution and, during apoptosis
Taken together our data suggest that as cells initiate adhesion to matrix increasing levels of ERK3 (显示 MAPK4 抗体) at the cell periphery are required to orchestrate cell morphology changes which can then drive migratory behavior.
p38gamma and p38delta reprogram liver metabolism by modulating neutrophil infiltration and provide a potential target for NAFLD (显示 TSC2 抗体) therapy
analysis of how allosteric regulation of p38gamma and PTPN3 (显示 PTPN3 抗体) involves a PDZ domain (显示 INADL 抗体)-modulated complex formation
Thus, in endothelial cells p38alpha (显示 MAPK14 抗体) mediates apoptotic signaling, whereas p38beta (显示 MAPK11 抗体) and p38gamma transduce survival signaling
p38gamma Mitogen-activated protein kinase signals through phosphorylating its phosphatase PTPH1 in regulating ras protein oncogenesis and stress response.
SEPW1 (显示 SEPW1 抗体) silencing increases MKK4 (显示 MAP2K4 抗体), which activates p38gamma, p38delta, and JNK2 (显示 MAPK9 抗体) to phosphorylate p53 (显示 TP53 抗体) on Ser (显示 SIGLEC1 抗体)-33 and cause a transient G(1) arrest.
phosphorylation at Ser (显示 SIGLEC1 抗体)-118 is required for ER to bind both p38gamma and c-Jun (显示 JUN 抗体), thereby promoting ER relocation from ERE to AP-1 (显示 FOSB 抗体) promoter sites.
p38gamma mitogen-activated protein kinase (显示 MAPK1 抗体) mediates inflammatory signaling to promote colon tumorigenesis
p38gamma and p38delta control heart growth by modulating mTOR (显示 FRAP1 抗体) pathway through DEPTOR (显示 DEPTOR 抗体) phosphorylation and subsequent degradation.
Findings provide genetic evidence that p38gamma and p38delta have essential roles in skin tumour development.
Together, our results establish that p38gamma and p38delta are central to colitis-associated colon cancer formation through regulation of hematopoietic cell response to injury, and validate p38gamma and p38kappa as potential targets for cancer therapy.
An energetic signal may trigger phosphorylation of the p38-gamma isoform which may explain how contractions differentially activate signaling pathways.
p38gamma and p38delta are crucial regulators of inflammatory joint destruction in collagen-induced arthritis.
p38gamma and p38delta kinases regulate the Toll-like receptor 4 (TLR4 (显示 TLR4 抗体))-induced cytokine production by controlling ERK1/2 protein kinase (显示 CDK7 抗体) pathway activation
results indicate that p38gamma and p38delta have a role in the suppression of tumor development
Activation of members of the mitogen-activated protein kinase family is a major mechanism for transduction of extracellular signals. Stress-activated protein kinases are one subclass of MAP kinases. The protein encoded by this gene functions as a signal transducer during differentiation of myoblasts to myotubes.
mitogen-activated protein kinase 12
, MAP kinase 12
, MAPK 12
, extracellular signal-regulated kinase 6
, stress-activated protein kinase 3
, MAP kinase p38 gamma
, mitogen-activated protein kinase 3
, mitogen-activated protein kinase p38 gamma
, mitogen activated protein kinase 12
, stress activated protein kinase 3
, SAP kinase-3