AGT CLIA Kit
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- 抗原 See all AGT CLIA Kits
- AGT (Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))
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适用
- 大鼠
- 检测方法
- Chemiluminescent
- 实验类型
- Competition ELISA
- 检测范围
- 12.35 pg/mL - 1000 pg/mL
- 最低检测浓度
- 12.35 pg/mL
- 应用范围
- ELISA
- 原理
- Rat Angiotensin I CLIA Kit is a Competitive CLIA Kit for use with Serum, plasma and other biological fluids.
- 样品类型
- Plasma, Serum
- Analytical Method
- Quantitative
- 灵敏度
- < 4.25 pg/mL
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- Discover our top product AGT ELISA Kit
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- 应用备注
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Stability: The stability of the kit is determined by the rate of activity loss. The loss rate is less than 5 % within the expiration date under appropriate storage conditions. To minimize performance fluctuations, operation procedures and lab conditions should be strictly controlled. It is also strongly suggested that the whole assay is performed by the same user throughout.
Recommended dilutions: Optimal dilutions/concentrations should be determined by the end user.
Standard Form: Lyophilized
- 板类型
- Pre-coated
- 限制
- 仅限研究用
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- 储存条件
- 4 °C/-20 °C
- 储存方法
- Upon receipt, store the kit according to the storage instruction in the kit's manual.
- 有效期
- 6 months
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- 抗原 See all AGT CLIA Kits
- AGT (Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))
- 别名
- Angiotensin I (AGT 产品)
- 别名
- ANHU CLIA Kit, SERPINA8 CLIA Kit, AI265500 CLIA Kit, AngI CLIA Kit, AngII CLIA Kit, Aogen CLIA Kit, Serpina8 CLIA Kit, ANRT CLIA Kit, Ang CLIA Kit, PAT CLIA Kit, wu:fb62f06 CLIA Kit, wu:fj87b02 CLIA Kit, zgc:111892 CLIA Kit, AGT CLIA Kit, angt CLIA Kit, ANGT CLIA Kit, angiotensinogen CLIA Kit, angiotensinogen (serpin peptidase inhibitor, clade A, member 8) CLIA Kit, AGT CLIA Kit, Agt CLIA Kit, agt CLIA Kit
- 途径
- JAK/STAT Signaling, ACE Inhibitor Pathway, EGFR Signaling Pathway, Peptide Hormone Metabolism, Regulation of Systemic Arterial Blood Pressure by Hormones, Regulation of Lipid Metabolism by PPARalpha, Protein targeting to Nucleus, Feeding Behaviour, Monocarboxylic Acid Catabolic Process, Dicarboxylic Acid Transport, Positive Regulation of Response to DNA Damage Stimulus, Regulation of long-term Neuronal Synaptic Plasticity
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