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Insulin ELISA 试剂盒

INS 适用: 人 Colorimetric Sandwich ELISA
产品编号 ABIN1326860
发货至: 中国
  • 抗原 See all Insulin (INS) ELISA试剂盒
    Insulin (INS)
    适用
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    检测方法
    Colorimetric
    实验类型
    Sandwich ELISA
    应用范围
    ELISA
    Analytical Method
    Quantitative
    Top Product
    Discover our top product INS ELISA Kit
  • 板类型
    Pre-coated
    限制
    仅限研究用
  • 储存条件
    4 °C
  • Bakry, Shoeib, El Shafiee, Hassan: "Androgenetic alopecia, metabolic syndrome, and insulin resistance: Is there any association? A case-control study." in: Indian dermatology online journal, Vol. 5, Issue 3, pp. 276-81, (2014) (PubMed).

    Sui, Yi, Yao, Liang, Sun, Hu, Uc, Nelson, Ode, Philipson, Engelhardt, Norris: "Quantifying insulin sensitivity and entero-insular responsiveness to hyper- and hypoglycemia in ferrets." in: PLoS ONE, Vol. 9, Issue 3, pp. e90519, (2014) (PubMed).

  • 抗原 See all Insulin (INS) ELISA试剂盒
    Insulin (INS)
    别名
    Insulin (INS 产品)
    背景
    Insulin is the principal hormone responsible for the control of glucose metabolism. It is synthesized in the _-cells of the islets of Langerhans as the precursor, proinsulin, which is processed to form C-peptide and insulin. Both are secreted in equimolar amounts into the portal circulation. The mature insulin molecule comprises two polypeptide chains, the A chain and B chain (21 and 30 amino acids respectively). The two chains are linked together by two inter-chain disulphide bridges. There is also an intra-chain disulphide bridge in the A chain. Insulin concentrations are severely reduced in insulin-dependent diabetes mellitus (IDDM) and some other conditions such as hypopituitarism. Insulin levels are raised in non-insulin-dependent diabetes mellitus (NIDDM), obesity, insulinoma and some endocrine dysfunctions such as Cushing
    途径
    NF-kappaB Signaling, RTK signaling, Positive Regulation of Peptide Hormone Secretion, Peptide Hormone Metabolism, Hormone Activity, Carbohydrate Homeostasis, ER-Nucleus Signaling, Regulation of Carbohydrate Metabolic Process, Feeding Behaviour, Autophagy, Negative Regulation of intrinsic apoptotic Signaling, Brown Fat Cell Differentiation, Positive Regulation of fat Cell Differentiation
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