DDIT3 Protein (AA 1-169) (His tag)
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- 抗原 See all DDIT3 蛋白
- DDIT3 (DNA-Damage-Inducible Transcript 3 (DDIT3))
- 蛋白类型
- Recombinant
- 产品特性
- AA 1-169
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宿主
- 人
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资源
- 大肠杆菌(E. Coli)
- 标记
- This DDIT3 protein is labelled with His tag.
- 应用范围
- SDS-PAGE (SDS)
- 产品特性
- GADD153,1-169aa, Human, His-tagged, Recombinant, E.coli
- 纯度
- > 90 % by SDS - PAGE
- Top Product
- Discover our top product DDIT3 蛋白
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- 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 mg/ml (determined by Bradford assay)
- 缓冲液
- Liquid in 20mM Tris pH8.0, 20% Glycerol
- 储存条件
- 4 °C
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- 抗原
- DDIT3 (DNA-Damage-Inducible Transcript 3 (DDIT3))
- 别名
- GADD153 (DDIT3 产品)
- 别名
- chop Protein, Xchop Protein, cebpz Protein, chop10 Protein, gadd153 Protein, CHOP-10 Protein, CHOP10 Protein, CHOP Protein, Chop10 Protein, Gadd153 Protein, CEBPZ Protein, GADD153 Protein, DNA damage inducible transcript 3 Protein, DNA-damage inducible transcript 3 Protein, DNA damage inducible transcript 3 L homeolog Protein, ddit3 Protein, Ddit3 Protein, DDIT3 Protein, ddit3.L Protein
- 背景
- GADD153, also known as DNA-damage-inducible transcript 3 (DDIT3), is a basic domain-leucine zipper(bZIP) transcription factor of C/EBP family. This protein has been shown to be up-regulated by several stresses, such as amino acid or glucose starvation, endoplasmic reticulum (ER) stress, osmotic stress and hypoxia. GADD153 protein may play a role in ER stress-mediated apoptosis and in disease including diabetes, brain ischemia and neurodegenerative disease. Recombinant GADD153 fused with His-tag, was expressed in E.coli and purified by conventional chromatography techniques. Synonyms: CHOP, DDIT3, CEBPZ, CHOP10, MGC4154, Growth arrest-and DNA damage-inducible gene 153, GADD153, Growth arrest-and DNA damage-inducible gene 153 C/EBP homologous protein, C/EBP Homology Protein, CHOP 10, DDIT 3, DNA Damage Inducible Transcript 3, GADD 153, Growth Arrest and DNA Damage Inducible Protein 153,. NCBI no.: NP_004074
- 分子量
- 21.3kDa (189aa), confirmed by MALDI-TOF.
- 途径
- Regulation of Muscle Cell Differentiation, ER-Nucleus Signaling, Skeletal Muscle Fiber Development, Cell RedoxHomeostasis
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