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抗Human RALB 抗体:
抗Rat (Rattus) RALB 抗体:
抗Mouse (Murine) RALB 抗体:
Human Monoclonal RALB Primary Antibody for IHC (p), IP - ABIN519627
Lee, Jethwaney, Schilling, Clemens, Gibson, Horwitz: The Mycobacterium bovis bacille Calmette-Guerin phagosome proteome. in Molecular & cellular proteomics : MCP 2010
our work provides new insight into the specific roles of Ras effector pathways in acute myeloid leukemia (显示 BCL11A 抗体) and has identified RALB signaling as a key survival pathway
This study identifies a novel regulatory crosstalk between Ral (显示 rala 抗体) and Arf6 (显示 ARF6 抗体) that controls Ral (显示 rala 抗体) function in cells.
High RALB expression is associated with acute myeloid leukemia (显示 BCL11A 抗体).
Inhibition of Ral (显示 rala 抗体) GTPases Using a Stapled Peptide Approach.
These findings suggest that RalB might be one of the targets for facilitating the invasive phenotype of malignant gliomas induced by GGTase-I.
striking isoform-specific consequences of distinct CAAX-signaled posttranslational modifications that contribute to the divergent subcellular localization and activity of RalA (显示 rala 抗体) and RalB.
expression of K-Ras (显示 HRAS 抗体) and RalB and possibly RalA (显示 rala 抗体) proteins is critical for maintaining low levels of p53 (显示 TP53 抗体), and down-regulation of these GTPases reactivates p53 (显示 TP53 抗体) by significantly enhancing its stability, contributing to suppression of malignant transformation
High RALB mRNA expression is associated with non-small-cell lung cancer growth and progression.
Integrin alpha(v (显示 ITGAV 抗体))beta expression and the resulting KRAS-RalB-NF-kappaB (显示 NFKB1 抗体) pathway were both necessary and sufficient for tumour initiation, anchorage independence, self-renewal and erlotinib resistance.
nutrient starvation induces RALB deubiquitylation by accumulation and relocalization of the deubiquitylase USP33 (显示 USP33 抗体) to RALB-positive vesicles
findings show either RALA (显示 rala 抗体) or RALB is sufficient for tumor growth; either RALA (显示 rala 抗体) or RALB is sufficient for cell proliferation; RALA (显示 rala 抗体) and RALB act in a redundant fashion
Endogenous RalB plays a negative role in the ingestion process of Fc gamma receptor (显示 FCGR1A 抗体)-mediated macrophage phagocytosis.
This gene encodes a GTP-binding protein that belongs to the small GTPase superfamily and Ras family of proteins. GTP-binding proteins mediate the transmembrane signaling initiated by the occupancy of certain cell surface receptors.
RAS-like protein B
, ras related GTP binding protein
, ras-related protein Ral-B
, v-ral simian leukemia viral oncogene homolog B (ras related; GTP binding protein)
, GTP binding protein)
, v-ral simian leukemia viral oncogene homolog B (ras related)