Influenza A Virus H1N1 抗体 (FITC)
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- 抗原
- Influenza A Virus H1N1
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适用
- Influenza A Virus H1N1
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宿主
- 山羊
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标记
- FITC
- 应用范围
- 请咨询
- 特异性
- Purified virions. Specific to H1N1 by IHA. Does not react with Influenza B, RSV, Para 1-3 or Adeno. Does not react with HEp-2 cells. May react with chicken cellular proteins.
- 产品特性
- Goat Anti-Influenza A H1N1 (virions)
- 免疫原
- Influenza A Strain USSR (H1N1)
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anti-Influenza A Virus H1N1 antibody (AA 18-344)
适用: Influenza A Virus H1N1 WB, EIA 宿主: 小鼠 Monoclonal AT1G7 unconjugated
anti-Influenza A Virus H1N1 antibody (Influenza Virus) (AA 35-48)适用: Influenza Virus WB 宿主: 兔 Polyclonal unconjugated
anti-Influenza A Virus H1N1 antibody (Influenza Virus) (AA 238-253)适用: Influenza Virus WB 宿主: 兔 Polyclonal unconjugated
anti-Influenza A Virus H1N1 antibody适用: Influenza A Virus H1N1 EIA 宿主: 兔 Polyclonal unconjugated
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- 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 4-5 mg/ml (OD280nm, E^0.1% = 1.4)
- 缓冲液
- 0.01M PBS, pH 7.2 containing 10mg/ml BSA
- 储存液
- Sodium azide
- 注意事项
- This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- 储存条件
- 4 °C
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- 抗原
- Influenza A Virus H1N1
- 物质类
- Influenza Virus
- 背景
- Actin polymerization is required for a variety of cell functions. Cells trigger Actin polymerization through either the de novo nucleation of filaments from monomeric Actin, the severing of existing filaments to create uncapped barbed ends, or the uncapping existing barbed ends. The nucleation of Actin is a rate-limiting and unfavorable reaction in Actin polymerization and therefore requires the involvement of the Arp2/3 complex, which helps create new filaments and promotes the end-to-side cross-linking of Actin filaments into the branching meshwork. The Arp2/3 complex consists of the Actinrelated proteins Arp2 and Arp3, as well as p41-ARC, p34-ARC, p21-ARC, p20-ARC and p16-ARC. The Arp2/3 complex promotes Actin nucleation by binding the pointed end of Actin filaments, or by associating with the side of an existing filament, and nucleates growth in the barbed direction. In addition, the Arp2/3 complex mediates Actin cytoskeletal outgrowths that are regulated by the Rho family of small GTPases. In response to GTPbinding Cdc42, the Arp2/3 complex binds the Cdc42 substrates, namely the WASP proteins, and initiates the formation of lamellipodia and filopodia.
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