Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 (SLC17A7) ELISA试剂盒

The protein encoded by SLC17A7 is a vesicle-bound, sodium-dependent phosphate transporter that is specifically expressed in the neuron-rich regions of the brain. 再加上,我们可以发Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 抗体 (133)Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 蛋白 (4)和数多这个蛋白质的别的产品。

list all ELISA KIts 基因 基因ID UniProt
SLC17A7 72961 Q3TXX4
SLC17A7 57030 Q9P2U7
大鼠 SLC17A7 SLC17A7 116638 Q62634
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小鼠 0.124 ng/mL 0.31 ng/mL - 20 ng/mL 96 Tests Log in to see 13至16个工作日
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4.68 pg/mL 18.75-1200 pg/mL Typical standard curve 96 Tests Log in to see 15至18个工作日
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小鼠 < 0.124 ng/mL 0.312 ng/mL - 20 ng/mL   96 Tests Log in to see 11至18个工作日
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适于 Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 相互作用对的更多 ELISA 试剂盒

Mouse (Murine) Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 (SLC17A7) interaction partners

  1. Mice heterozygous for the vesicular glutamate transporter 1 (VGLUT1+/-) were used. Firstly, mRNA expression of the different members of the HDAC superfamily in the prefrontal cortex (PFC) of VGLUT1+/- mice and WT littermates were studied by RT-PCR.

  2. These findings suggest that nitrosylation of VAChT and VGLUT1 may be associated with dysfunctional acetylcholinergic and glutamatergic neurotransmission in Alzheimer's disease.

  3. Study revealed susceptibility of glutamatergic nerve terminals to Abeta (显示 APP ELISA试剂盒) induced toxicity and underlined the importance of VGLUT1 in the progression of Alzheimer's disease, as the decrease of this protein levels could increase the susceptibility to subsequent deleterious inputs by exacerbating Abeta (显示 APP ELISA试剂盒) induced neuroinflammation and synaptic plasticity disruption.

  4. triple KO of Slc17a7, Slc17a6, and Slc17a8 diminished IIIS, which was rescued by exogenously introduced wild-type Slc17a7 or Slc17a6 genes

  5. In CA1 pyramidal neurons, a slow excitatory postsynaptic current is absent in the VGLUT1 knockout mouse.

  6. Data show that activation of nucleotide receptor P2Y4 (P2Y4) in the differentiating embryonic stem cells (ESCs) resulted in an increased proportion of neurons expressing vesicular glutamate transporter (vGluT).

  7. Results suggest that activation of JNK in Alzheimer's disease (AD) inhibits insulin signaling which could lead to a decreased expression of VGLUT1, therefore contributing to the glutamatergic deficit in AD

  8. Distribution of VGLUT1 and VGLUT2 in the CN, previously described for guinea pig, was replicated in mouse and showed similar changes in VGLUT1 and 2 distributions after unilateral cochlear insult

  9. Here, we show that VGluT1(+/-) mice acquired the initial visual discrimination at the same rate as controls. However, they failed to suppress responses to the previously rewarded stimulus following reversal of reward contingencies.

  10. our genetic analysis provides new in vivo evidence that VGluT1(+) glutamatergic signaling, mediated by the astroglial mGluR5 (显示 GRM5 ELISA试剂盒) receptor, regulates the functional maturation of cortical astroglia during development.

Human Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 (SLC17A7) interaction partners

  1. Study revealed susceptibility of glutamatergic nerve terminals to Abeta (显示 APP ELISA试剂盒) induced toxicity and underlined the importance of VGLUT1 in the progression of Alzheimer's disease, as the decrease of this protein levels could increase the susceptibility to subsequent deleterious inputs by exacerbating Abeta (显示 APP ELISA试剂盒) induced neuroinflammation and synaptic plasticity disruption.

  2. This study was the first to demonstrate an association between genetic polymorphism at rs7417284 SNP in the promoter region of the SLC17A7 gene and concussion severity and duration. Based upon these findings, rs74174284 is a potential predictive genetic marker for identifying athletes who are more susceptible for altered recovery times and worse motor speed ImPACT scores after sport-related concussion.

  3. Results suggest that activation of JNK in Alzheimer's disease (AD) inhibits insulin signaling which could lead to a decreased expression of VGLUT1, therefore contributing to the glutamatergic deficit in AD

  4. Loss of SLC17A7 expression is associated with glioblastoma.

  5. Depressed patients showed significant decreases in synaptophysin (SYN (显示 SYP ELISA试剂盒)) and VGLUT1 expression, whereas in bipolar patients, decreases in VGLUT1 expression have also been found.

  6. Data indicate that GABAergic axons were labeled with vesicular inhibitory aa transporter (VIAAT) antibodies, whereas glutamatergic axons were detected with antisera against the major vesicular glutamate transporter (VGLUT) isoforms, VGLUT1 and VGLUT2.

  7. We examined the ratio of excitatory to inhibitory vesicular neurotransmitter transporter (显示 SLC6A2 ELISA试剂盒) mRNAs (VGluT1 to VGAT (显示 SLC32A1 ELISA试剂盒)) and their ratio in the dorsolateral prefrontal cortex during normal human development and in people with schizophrenia

  8. this study suggests that the common genetic variants of the VGLUT1 gene appear not play a major role in conferring susceptibility to schizophrenia in Han population of Taiwan.

  9. In schizophrenia, VGLUT1 mRNA was decreased in hippocampal formation and dorsolateral prefrontal cortex. In the hippocampus, the loss of VGLUT1 mRNA supports data indicating that glutamatergic presynaptic deficits are prominent.

  10. Alterations in the pattern of vesicular glutamate transporter 1-immunoreactivity that perfectly matched the neuronal loss and gliosis, as well as the decrease in the number of asymmetrical synapses identified by electron microscopy in this tissue

Solute Carrier Family 17 (Vesicular Glutamate Transporter), Member 7 (SLC17A7) 抗原简介

Antigen Summary

The protein encoded by this gene is a vesicle-bound, sodium-dependent phosphate transporter that is specifically expressed in the neuron-rich regions of the brain. It is preferentially associated with the membranes of synaptic vesicles and functions in glutamate transport. The protein shares 82% identity with the differentiation-associated Na-dependent inorganic phosphate cotransporter and they appear to form a distinct class within the Na+/Pi cotransporter family.

Gene names and symbols associated with SLC17A7

  • solute carrier family 17 (sodium-dependent inorganic phosphate cotransporter), member 7 (Slc17a7) 抗体
  • solute carrier family 17 member 7 (SLC17A7) 抗体
  • solute carrier family 17 member 7 (Slc17a7) 抗体
  • 2900052E22Rik 抗体
  • AI851913 抗体
  • BNPI 抗体
  • VGLUT1 抗体

Protein level used designations for SLC17A7

Brain-specific Na(+)-dependent inorganic phosphate cotransporter , Solute carrier family 17 member 7 , brain-specific Na(+)-dependent inorganic phosphate cotransporter , solute carrier family 17 member 7 , vesicular glutamate transporter 1 , brain-specific Na-dependent inorganic phosphate cotransporter , solute carrier family 17 (sodium-dependent inorganic phosphate cotransporter), member 7 , solute carrier family 17 (vesicular glutamate transporter), member 7

GENE ID SPECIES
72961 Mus musculus
57030 Homo sapiens
116638 Rattus norvegicus
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