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The protein encoded by HABP2 is an extracellular serine protease that binds hyaluronic acid and is involved in cell adhesion. 再加上，我们可以发HABP2 蛋白 (17) 和 HABP2 试剂盒 (7)和数多这个蛋白质的别的产品。
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Human Monoclonal HABP2 Primary Antibody for IP, ELISA - ABIN516334
Altmäe, Kallak, Fridén, Stavreus-Evers: Variation in hyaluronan-binding protein 2 (HABP2) promoter region is associated with unexplained female infertility. in Reproductive sciences (Thousand Oaks, Calif.) 2011
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Human Polyclonal HABP2 Primary Antibody for IHC, IHC (p) - ABIN4316533
Bachmann, Burté, Pramana, Conte, Brown, Orimadegun, Ajetunmobi, Afolabi, Akinkunmi, Omokhodion, Akinbami, Shokunbi, Kampf, Pawitan, Uhlén, Sodeinde, Schwenk, Wahlgren, Fernandez-Reyes, Nilsson: Affinity proteomics reveals elevated muscle proteins in plasma of children with cerebral malaria. in PLoS pathogens 2014
Two of 20 probands from families with history of PTC (显示 F9 抗体) and breast carcinoma (BC) were evaluated by whole exome sequencing (WES) revealing HABP2 p.G534E.
No significant association was found between rs11196288 and early-onset ischemic stroke, large artery atherosclerotic stroke, or small vessel disease stroke. rs11196288 presented significant effect on late-onset SVD (显示 KCNJ13 抗体) stroke susceptibility in the older population.
study on a wide series of familial non-medullary thyroid cancers indicates that the HABP2(G534E) variant is frequent, but does not segregate with the disease
Letter: G534E variant in HABP2 is not associated with non-medullary thyroid cancer in the Spanish population.
Study showed that lower FSAP antigen plasma levels were associated with a higher chance of arterial recanalization after tissue plasminogen activator (显示 PLAT 抗体) treatment, suggesting an involvement of FSAP in tissue plasminogen activator (显示 PLAT 抗体)-induced clot (显示 TXNDC17 抗体) lysis. FSAP antigen determination might be useful in predicting tissue plasminogen activator (显示 PLAT 抗体) response in stroke patients.
HABP2 polymorphisms are not associated with thyroid cancer.
the promoter activity, which could phenocopy changes in Habp2 mRNA in response to TGF-beta (显示 TGFB1 抗体), was found to be located in the 177-bp region upstream of the transcription start site, and this region did not contain any SMAD (显示 SMAD1 抗体) binding sites.
Results show that G534E germline variant in HABP2 does not account for the familial nature of nonmedullary thyroid cancer in Australian kindreds but and is common in the general population.
omology modeling suggested that the Glu (显示 DCTN1 抗体)-221 side chain could sterically hinder insertion of the N terminus into the HABP2 protease domain, helping to explain the detrimental effects of Glu (显示 DCTN1 抗体)-221 substitution on HABP2 activity.
The data do not support the pathogenicity of the HABP2 c.1601G > A variant but highlight the existence of a new one that should be more extensively searched for in familial papillary thyroid carcinoma patients and its pathogenicity more carefully evaluated.
FSAP deficiency causes an increase in CCL2 (显示 CCL2 抗体) expression and CCL2 (显示 CCL2 抗体)-mediated infiltration of leukocytes into the injured vessel
Lack of endogenous FSAP impaired the formation of stable, occlusive thrombi in mice.
Results show that the lack of FSAP in mice worsens the outcome of stroke; in the absence of FSAP there was a stronger inflammatory response and lower cell survival due to insufficient activation of the PI3K/AKT (显示 AKT1 抗体) pathway
Lower FSAP expression is associated with enhanced liver fibrosis and inflammation in patients with chronic hepatic disorders and murine experimental liver injury.
Data indicate that FSAP mediates proteolytic cleavage and activation of bone morphogenetic protein-2 (BMP-2 (显示 BMP2 抗体)).
Hyaluronic acid binding protein 2 (HABP2) negatively regulates vascular integrity via activation of protease-activated receptor/RhoA (显示 RHOA 抗体)/Rho kinase (显示 ROCK2 抗体) signaling. It represents a potential therapeutic target for syndromes of increased vascular permeability.
The protein encoded by this gene is an extracellular serine protease that binds hyaluronic acid and is involved in cell adhesion. The encoded protein is synthesized as a single chain, but then undergoes an autoproteolytic event to form the functional heterodimer. Further autoproteolysis leads to smaller, inactive peptides. This protease is known to cleave urinary plasminogen activator, coagulation factor VII, and the alpha and beta chains of fibrinogen, but not prothrombin, plasminogen, or the gamma chain of fibrinogen. Two transcript variants encoding different isoforms have been found for this gene.
hyaluronan binding protein 2
, hyaluronan-binding protein 2-like
, factor VII activating protein
, factor VII-activating protease
, factor seven-activating protease
, hepatocyte growth factor activator-like protein
, hyaluronan-binding protein 2
, hyaluronic acid binding protein 2
, plasma hyaluronan binding protein
, plasma hyaluronan-binding protein
, hyaduronic acid-binding protein 2