TMC8 抗体
(Transmembrane Channel-Like 8 (TMC8))
Epidermodysplasia verruciformis (EV) is an autosomal recessive dermatosis characterized by abnormal susceptibility to human papillomaviruses (HPVs) and a high rate of progression to squamous cell carcinoma on sun-exposed skin. EV is caused by mutations in either of two adjacent genes located on chromosome 17q25.3. Both of these genes encode integral membrane proteins that localize to the endoplasmic reticulum and are predicted to form transmembrane channels. This gene encodes a transmembrane channel-like protein with 8 predicted transmembrane domains and 3 leucine zipper motifs. [provided by RefSeq, Jul 2008].
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Latest Publications for our TMC8 抗体
: "Regulation of cellular zinc balance as a potential mechanism of EVER-mediated protection against pathogenesis by cutaneous oncogenic human papillomaviruses." in: The Journal of experimental medicine, Vol. 205, Issue 1, pp. 35-42, (2008) (PubMed).: "Mutations in two adjacent novel genes are associated with epidermodysplasia verruciformis." in: Nature genetics, Vol. 32, Issue 4, pp. 579-81, (2002) (PubMed).
: "Evidence for a nonallelic heterogeneity of epidermodysplasia verruciformis with two susceptibility loci mapped to chromosome regions 2p21-p24 and 17q25." in: The Journal of investigative dermatology, Vol. 114, Issue 6, pp. 1148-53, (2000) (PubMed).
Aliases for TMC8 抗体
transmembrane channel like 8 (TMC8) 抗体transmembrane channel-like 8 (Tmc8) 抗体
transmembrane channel-like gene family 8 (Tmc8) 抗体
EV2 抗体
EVER2 抗体
Ever2 抗体
EVIN2 抗体
mFLJ00400 抗体
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