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CCL3 蛋白

CCL3 宿主: 小鼠 宿主: 大肠杆菌(E. Coli)
产品编号 ABIN6699627
发货至: 中国
  • 抗原 See all CCL3 蛋白
    CCL3 (Chemokine (C-C Motif) Ligand 3 (CCL3))
    宿主
    • 19
    • 5
    • 5
    • 3
    • 1
    • 1
    • 1
    小鼠
    资源
    • 21
    • 4
    • 3
    • 2
    • 2
    • 1
    • 1
    大肠杆菌(E. Coli)
    交叉反应
    小鼠
    产品特性
    Mouse Macrophage Inflammatory Protein-1-alpha, CCL3 cytokine, C-C motif chemokine 3, Small-inducible cytokine A3, Macrophage inflammatory protein 1-α, MIP-1-alpha, TY-5, SIS-alpha, Heparin-binding chemotaxis protein, L2G25B
    纯化方法
    Purity was determined to be greater than 95% by analysis by RP-HPLC and by reducing and non-reducing SDS-PAGE.
    内毒素水平
    Low endotoxin
    Top Product
    Discover our top product CCL3 蛋白
  • 应用备注

    Application Note: MIP-1α is suitable as a control for polyclonal or monoclonal anti--MIP-1α in immunological assays.

    Other Performance Data: Biological Activity: Mouse Macrophage Inflammatory Protein-1 is fully biologically active when compared to standard. The activity is calculated by the ability to chemoattract of Balb3/C splenocytes using 1-10 ng/mL. Endotoxin Level: Measured by LAL is <0.01 ng/μg or <0.1EU/μg.

    限制
    仅限研究用
  • 状态
    Lyophilized
    溶解方式

    Reconstitution Volume: 10 μL (10-100 μL)

    Reconstitution Buffer: Restore with deionized water (or equivalent)

    浓度
    0.1 mg/mL
    缓冲液

    Buffer: 0.1 % Trifluoroacetic acid

    储存液
    Without preservative
    储存条件
    RT,4 °C,-20 °C
    有效期
    6 months
  • 抗原
    CCL3 (Chemokine (C-C Motif) Ligand 3 (CCL3))
    别名
    MIP-1alpha (CCL3 产品)
    别名
    MIP-1alpha Protein, G0S19-1 Protein, LD78ALPHA Protein, MIP-1-alpha Protein, MIP1A Protein, SCYA3 Protein, MIP-1a Protein, Scya3 Protein, AI323804 Protein, LD78alpha Protein, MIP1-(a) Protein, MIP1-alpha Protein, Mip1a Protein, CCL3L1 Protein, CCL3L3 Protein, chemokine (C-C motif) ligand 3 Protein, C-C motif chemokine ligand 3 Protein, CCL3 Protein, Ccl3 Protein
    UniProt
    P10855
    途径
    Cellular Response to Molecule of Bacterial Origin, Autophagy
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