IRF5 抗体
Quick Overview for IRF5 抗体 (ABIN2666392)
抗原
See all IRF5 抗体适用
宿主
克隆类型
标记
应用范围
克隆位点
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亚型
- IgG2b kappa
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anti-Interferon Regulatory Factor 5 (IRF5) (AA 395-504) antibody
IRF5 适用: 人 WB, ELISA, IP, IF 宿主: 小鼠 Monoclonal 1H6 unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 395-504) antibodyIRF5 适用: 人 WB, ELISA, IF, IHC (p) 宿主: 小鼠 Monoclonal 2D4 unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (C-Term) antibodyVerified IRF5 适用: 人 WB, ELISA 宿主: 山羊 Polyclonal unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 81-108) antibodyIRF5 适用: 人 WB, ELISA, IF, FACS 宿主: 兔 Polyclonal unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 214-395) antibodyIRF5 适用: 人 WB, ELISA, IHC, IF 宿主: 兔 Polyclonal unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) antibodyIRF5 适用: 人, 小鼠 WB, ELISA, IHC 宿主: 兔 Polyclonal unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 176-240) antibodyIRF5 适用: 人 WB, ELISA, IHC, IF, ICC, FACS 宿主: 小鼠 Monoclonal 10T1 unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 1-498) antibodyIRF5 适用: 人 WB, ELISA, IHC (p) 宿主: 小鼠 Monoclonal 2G9 unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) (AA 1-200) antibodyIRF5 适用: 人 WB, IHC 宿主: 兔 Polyclonal unconjugated
anti-Interferon Regulatory Factor 5 (IRF5) antibodyIRF5 适用: 人 WB, IHC (p) 宿主: 兔 Monoclonal 3F11 unconjugated
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应用备注
- Optimal working dilution should be determined by the investigator.
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限制
- 仅限研究用
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浓度
- 0.5 mg/mL
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缓冲液
- phosphate-buffered solution, pH 7.2, containing 0.05 % sodium azide.
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储存液
- Sodium azide
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注意事项
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
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储存条件
- 4 °C
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储存方法
- Upon receipt, store undiluted between 2°C and 8°C.
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- IRF5 (Interferon Regulatory Factor 5 (IRF5))
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别名
- IRF5
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背景
- IRF-5 is a transcription factor belonging to the interferon regulatory factor family. It controls induction of type I IFN genes as well as inflammatory cytokines and chemokines through TLRs and the MyD88-TRAF6 pathway. Several genetic variations of IRF-5 have been reported as a risk factor of Systemic lupus erythematosus (SLE), which is caused by excess IFNα induction.
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途径
- TLR signaling, Autophagy
抗原
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