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CD25 抗体

IL2RA 适用: 人 FACS, IHC (fro) 宿主: 小鼠 Monoclonal B-F2 unconjugated
产品编号 ABIN1106138
发货至: 中国
  • 抗原 See all CD25 (IL2RA) 抗体
    CD25 (IL2RA) (Interleukin 2 Receptor, alpha (IL2RA))
    适用
    • 165
    • 111
    • 36
    • 10
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    • 2
    • 1
    • 1
    宿主
    • 137
    • 80
    • 77
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    小鼠
    克隆类型
    • 222
    • 70
    • 4
    单克隆
    标记
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    This CD25 antibody is un-conjugated
    应用范围
    • 218
    • 86
    • 54
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    • 44
    • 44
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    Flow Cytometry (FACS), Immunohistochemistry (Frozen Sections) (IHC (fro))
    特异性
    Recognises the Interleukin 2 Receptor a chain (IL-2 Ra), a 55 kDa protein
    纯化方法
    Ion exchange chromatography
    免疫原
    PHA activated T cells
    克隆位点
    B-F2
    亚型
    IgG1
    Top Product
    Discover our top product IL2RA Primary Antibody
  • 应用备注
    Optimal working dilution should be determined by the investigator.
    限制
    仅限研究用
  • 状态
    Liquid
    缓冲液
    Phosphate-buffered saline containing 1 % BSA as stabilizer and 0.09 % sodium azide as preservative
    储存液
    Sodium azide
    注意事项
    This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    储存条件
    4 °C
    储存方法
    Store the antibody undiluted at 2-8 °C.
  • 抗原
    CD25 (IL2RA) (Interleukin 2 Receptor, alpha (IL2RA))
    别名
    CD25 / IL2RA (IL2RA 产品)
    背景
    Genetic variations in IL2RA are associated with susceptibility to diabetes mellitus insulin-dependent type 10 (IDDM10) [MIM:601942]. A multifactorial disorder of glucose homeostasis that is characterized by susceptibility to ketoacidosis in the absence of insulin therapy. Clinical fetaures are polydipsia, polyphagia and polyuria which result from hyperglycemia-induced osmotic diuresis and secondary thirst. These derangements result in long-term complications that affect the eyes, kidneys, nerves, and blood vessels.Synonyms: IL-2 receptor alpha subunit, IL-2-RA, IL2-RA, Interleukin-2 receptor alpha chain, TAC antigen, p55
    基因ID
    3559
    NCBI登录号
    NP_000408
    UniProt
    P01589
    途径
    JAK/STAT Signaling, Growth Factor Binding, Activated T Cell Proliferation
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