Tumor Necrosis Factor (Ligand) Superfamily, Member 18 (TNFSF18) ELISA试剂盒

The protein encoded by TNFSF18 is a cytokine that belongs to the tumor necrosis factor (TNF) ligand family. 再加上,我们可以发Tumor Necrosis Factor (Ligand) Superfamily, Member 18 抗体 (171)Tumor Necrosis Factor (Ligand) Superfamily, Member 18 蛋白 (41)和数多这个蛋白质的别的产品。

list all ELISA KIts 基因 基因ID UniProt
TNFSF18 240873 Q7TS55
TNFSF18 8995 Q9UNG2
大鼠 TNFSF18 TNFSF18 364031  
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小鼠 10pg/mL 31.2-2000 pg/mL Mouse TNFSF18/GITRL PicoKine ELISA Kit standard curve 96 Tests Log in to see 4至6个工作日
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25 pg/mL 25-10000 pg/mL 96 Tests Log in to see 2至3个工作日
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小鼠 4 pg/mL 4.1 pg/mL - 1000 pg/mL   96 Tests Log in to see 5至8个工作日
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20 pg/mL 40.9 pg/mL - 10000 pg/mL   96 Tests Log in to see 5至8个工作日
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引用最多的Tumor Necrosis Factor (Ligand) Superfamily, Member 18 ELISA试剂盒

  1. Human TNFSF18 ELISA Kit for Sandwich ELISA - ABIN625249 : Gan, Feng, Gu, Tan, Sun, Lv, Zhang: Correlation of increased blood levels of GITR and GITRL with disease severity in patients with primary Sjögren's syndrome. in Clinical & developmental immunology 2013 (PubMed)
    Show all 2 Pubmed References

适于 Tumor Necrosis Factor (Ligand) Superfamily, Member 18 相互作用对的更多 ELISA 试剂盒

Human Tumor Necrosis Factor (Ligand) Superfamily, Member 18 (TNFSF18) interaction partners

  1. Cultured HCN-2 neurons were incubated at different times with GITRL and/or TRAIL, and thereafter nucleic acid and protein expression were measured. HCN-2 cells do not express GITRL mRNA, but the latter is induced after treatment with TRAIL. Cells did not express the GITRL receptor GITR mRNA, neither in control cultures, nor after treatment with TRAIL. TRAIL, when associated to GITRL, exerted additive toxic effects.

  2. GITRL levels are significantly elevated in rheumatoid arthritis serum and synovial fluid and are positively correlated with autoantibody production in rheumatoid arthritis, suggesting a role of GITRL in the development of rheumatoid arthritis.

  3. GITRL modulates the activities of p38 MAPK and STAT3 to promote Th17 cell differentiation in autoimmune arthritis.

  4. An increase in GITRL may impair the balance of Th17/Treg, and contribute to the pathopoiesis of Hashimoto's thyroiditis.

  5. Our findings indicate the possible involvement of GITR-GITRL pathway in the pathogenesis of pSS.

  6. Serum GITRL levels were higher in SLE patients.

  7. Glucocorticoid-induced TNF-related ligand (GITRL) confers pseudoexpression to tumor cells by platelets, which results in GITRL expression by megakaryocytes and their platelet progeny.

  8. observation suggests a link between cytokine-regulated keratinocyte GITRL expression and its role in inflammatory responses in AD

  9. GITRL upregulation induced by IFN-beta on dendritic cells downregulates CTLA-4 on regulatory T (Treg) cells, facilitating proliferation of anergic Treg cells in multiple sclerosis treatment of multiple sclerosis patients.

  10. Although GITR transgene costimulation can therapeutically enhance T helper (Th) type 2 cell responses, GITR-GITR ligand interactions are not required for development of Th2-mediated resistance or pathology.

  11. GITRL expression on Kupffer cells may mediate acute rejection in liver transplantation

  12. The incorporation of an isoleucine zipper motif could markedly improve the costimulation of hsGITRL.

  13. Upregulation by proinflammatory cytokines suggests GITRL may play important role in ocular immunity. High level of constitutive GITRL expression on photoreceptor inner segments suggests photoreceptors participate in regulation of ocular inflammation.

  14. Regulates ossteoclasst genersation and substantiate the major role played by the endothelium in bone physiology.

  15. Using a GITRL-transfected cell line, we demonstrate that GITRL promotes NK cell cytotoxicity and IFN-gamma production.

  16. GITRL could be a potential candidate for regulation of the ocular immune privilege and the balance between immune privilege and inflammation

  17. Since regulatory T-cells are localized in the vicinity of GITRL-expressing cells in atopic dermatitis skin, the GITR/GITRL interaction may serve to perpetuate the inflammation locally.

  18. Constitutive expression of GITRL by tumor cells diminishes natural killer cell antitumor immunity.

  19. although huGITRL is not capable of alleviating Treg suppression of responder T cells, huGITRL overexpression on monocyte-derived DC enhances their capacity to induce antigen-specific T cell responses

  20. These observations raise the possibility that the GITRL-mediated inflammatory activation of macrophages is involved in the pathogenesis of inflammatory diseases.

Tumor Necrosis Factor (Ligand) Superfamily, Member 18 (TNFSF18) 抗原简介

Antigen Summary

The protein encoded by this gene is a cytokine that belongs to the tumor necrosis factor (TNF) ligand family. This cytokine is a ligand for receptor TNFRSF18/AITR/GITR. It has been shown to modulate T lymphocyte survival in peripheral tissues. This cytokine is also found to be expressed in endothelial cells, and is thought to be important for interaction between T lymphocytes and endothelial cells.

Gene names and symbols associated with TNFSF18

  • tumor necrosis factor (ligand) superfamily, member 18 (Tnfsf18) 抗体
  • TNF superfamily member 18 (TNFSF18) 抗体
  • TNF superfamily member 18 (Tnfsf18) 抗体
  • AITRL 抗体
  • GITRL 抗体
  • hGITRL 抗体
  • TL6 抗体
  • TNLG2A 抗体

Protein level used designations for TNFSF18

GITR ligand , glucocorticoid-induced TNF-related ligand , glucocorticoid-induced-tumor necrosis factor receptor ligand , tumor necrosis factor ligand superfamily member 18 , AITR ligand , activation-inducible TNF-related ligand , glucocorticoid-induced TNFR-related protein ligand , tumor necrosis factor (ligand) superfamily, member 18 , tumor necrosis factor ligand 2A , tumor necrosis factor superfamily member 18

GENE ID SPECIES
240873 Mus musculus
8995 Homo sapiens
364031 Rattus norvegicus
100855912 Canis lupus familiaris
100060504 Equus caballus
768081 Bos taurus
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