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The protein encoded by TNFSF18 is a cytokine that belongs to the tumor necrosis factor (TNF) ligand family. 再加上，我们可以发Tumor Necrosis Factor (Ligand) Superfamily, Member 18 抗体 (178) 和 Tumor Necrosis Factor (Ligand) Superfamily, Member 18 蛋白 (41)和数多这个蛋白质的别的产品。
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Human TNFSF18 ELISA Kit for Sandwich ELISA - ABIN625249
Gan, Feng, Gu, Tan, Sun, Lv, Zhang: Correlation of increased blood levels of GITR and GITRL with disease severity in patients with primary Sjögren's syndrome. in Clinical & developmental immunology 2013
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the upregulation of IFN-beta (显示 IFNB1 ELISA试剂盒) in DCs induces the up-regulation of coinhibitory molecules B7H1 (显示 CD274 ELISA试剂盒) and GITRL, which cause an impaired activation of naive Ag-specific T cells
GITRL expressed on macrophages drives cytokine release and T cell activation, resulting in neuropathic pain via GITR (显示 TNFRSF18 ELISA试剂盒)-dependent actions. The GITRL-GITR (显示 TNFRSF18 ELISA试剂盒) pathway might represent a novel target for the treatment of neuropathic pain.
Down-regulation of GITRL enhances CD8 (显示 CD8A ELISA试剂盒) T cell responses to chronic lymphocytic choriomeningitis virus.
Sjogren's syndrome-like autoimmune sialadenitis in MRL-Faslpr mice is associated with expression of glucocorticoid-induced TNF receptor-related protein (GITR) ligand and 4-1BB ligand (显示 TNFSF9 ELISA试剂盒).
GITRL triggering in endothelial cells increases leukocyte adhesion and transmigration.
The B7H1 (显示 CD274 ELISA试剂盒) and GITRL molecules may play an important role in TGF-beta (显示 TGFB1 ELISA试剂盒)-induced Treg expansion of lung cancer microenvironment.
results demonstrate that enhanced GITR (显示 TNFRSF18 ELISA试剂盒)/GITRL interactions have a pleiotropic role on the regulation of T-cell responses
B cell expression of GITRL induced Treg proliferation, maintaining their numbers above a threshold required for the prevention of autoimmunity.
Findings suggest GITRL could enhance the immune stimulation of DC and might facilitate the potential development of DCs-based anti-tumor therapies.
GITRL-dependent expansion of Treg within the cornea is one mechanism underlying immune privilege in corneal allografts.
Cultured HCN-2 (显示 HCN2 ELISA试剂盒) neurons were incubated at different times with GITRL and/or TRAIL, and thereafter nucleic acid and protein expression were measured. HCN-2 (显示 HCN2 ELISA试剂盒) cells do not express GITRL mRNA, but the latter is induced after treatment with TRAIL. Cells did not express the GITRL receptor GITR (显示 TNFRSF18 ELISA试剂盒) mRNA, neither in control cultures, nor after treatment with TRAIL. TRAIL, when associated to GITRL, exerted additive toxic effects.
GITRL levels are significantly elevated in rheumatoid arthritis serum and synovial fluid and are positively correlated with autoantibody production in rheumatoid arthritis, suggesting a role of GITRL in the development of rheumatoid arthritis.
GITRL modulates the activities of p38 MAPK (显示 MAPK14 ELISA试剂盒) and STAT3 (显示 STAT3 ELISA试剂盒) to promote Th17 cell differentiation in autoimmune arthritis.
An increase in GITRL may impair the balance of Th17/Treg, and contribute to the pathopoiesis of Hashimoto's thyroiditis.
Our findings indicate the possible involvement of GITR (显示 TNFRSF18 ELISA试剂盒)-GITRL pathway in the pathogenesis of pSS (显示 CDSN ELISA试剂盒).
Serum GITRL levels were higher in SLE patients.
Glucocorticoid-induced TNF-related ligand (GITRL) confers pseudoexpression to tumor cells by platelets, which results in GITRL expression by megakaryocytes and their platelet progeny.
observation suggests a link between cytokine-regulated keratinocyte GITRL expression and its role in inflammatory responses in AD
GITRL upregulation induced by IFN-beta (显示 IFNB1 ELISA试剂盒) on dendritic cells downregulates CTLA-4 (显示 CTLA4 ELISA试剂盒) on regulatory T (Treg) cells, facilitating proliferation of anergic Treg cells in multiple sclerosis treatment of multiple sclerosis patients.
Although GITR (显示 TNFRSF18 ELISA试剂盒) transgene costimulation can therapeutically enhance T helper (Th) type 2 cell responses, GITR-GITR (显示 TNFRSF18 ELISA试剂盒) ligand interactions are not required for development of Th2-mediated resistance or pathology.
The protein encoded by this gene is a cytokine that belongs to the tumor necrosis factor (TNF) ligand family. This cytokine is a ligand for receptor TNFRSF18/AITR/GITR. It has been shown to modulate T lymphocyte survival in peripheral tissues. This cytokine is also found to be expressed in endothelial cells, and is thought to be important for interaction between T lymphocytes and endothelial cells.
, glucocorticoid-induced TNF-related ligand
, glucocorticoid-induced-tumor necrosis factor receptor ligand
, tumor necrosis factor ligand superfamily member 18
, AITR ligand
, activation-inducible TNF-related ligand
, glucocorticoid-induced TNFR-related protein ligand
, tumor necrosis factor (ligand) superfamily, member 18
, tumor necrosis factor ligand 2A
, tumor necrosis factor superfamily member 18