Follistatin-Like 3 (Secreted Glycoprotein) 蛋白 (FSTL3)

Follistatin-like 3 is a secreted glycoprotein of the follistatin-module-protein family. 再加上,我们可以发FSTL3 抗体 (66)FSTL3 试剂盒 (38)和数多这个蛋白质的别的产品。

列出全部蛋白 基因 基因ID UniProt
FSTL3 83554 Q9EQC7
FSTL3 10272 O95633
FSTL3 114031 Q99PW7
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Showing 9 out of 9 products:

产品编号 Origin 资源 标记 图像 规格 供应商 交付 价格 详细
人细胞 小鼠 His tag 50 μg Log in to see 14至16个工作日
$440.65
详细
Insect Cells His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 50 Days
$6,749.58
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Insect Cells 小鼠 His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 50 Days
$6,749.58
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小麦胚 GST tag 10 μg Log in to see 11至12个工作日
$340.00
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HEK-293 Cells Myc-DYKDDDDK Tag Validation with Western Blot 20 μg Log in to see 11 Days
$888.80
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酵母菌 大鼠 His tag   1 mg Log in to see 60至71个工作日
$2,588.67
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酵母菌 Cow His tag   1 mg Log in to see 60至71个工作日
$2,594.17
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大肠杆菌(E. Coli) 小鼠 S tag,His tag 100 μg Log in to see 15至18个工作日
$736.00
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大肠杆菌(E. Coli) 小鼠 非结合性   100 μg Log in to see 11至18个工作日
$710.67
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FSTL3 蛋白 by Origin and Source

Origin 在表达 标记
Mouse (Murine) , ,
,
Human , ,
, ,
Rat (Rattus)

引用最多的FSTL3 蛋白

  1. Mouse (Murine) FSTL3 Protein expressed in Human Cells - ABIN2007181 : Bartholin, Destaing, Forissier, Martel, Maguer-Satta, Jurdic, Rimokh: FLRG, a new ADAM12-associated protein, modulates osteoclast differentiation. in Biology of the cell / under the auspices of the European Cell Biology Organization 2005 (PubMed)
    Show all 5 Pubmed References

更多Follistatin-Like 3 (Secreted Glycoprotein) (FSTL3)互动伙伴

Mouse (Murine) Follistatin-Like 3 (Secreted Glycoprotein) (FSTL3) interaction partners

  1. FSTL3 KO lead to increased activin signaling, enhancing alpha- to beta-cell transdifferentiation in mouse islets.

  2. FSTL3 is a critical mediator of exercise-dependent bone formation

  3. Data indicate that deletion of activin antagonists follistatin-like-3 (FSTL3) results in hepatic steatosis

  4. Suggest FSTL3 has a role in paracrine communication between cardiomyocytes and fibroblasts.

  5. crucial roles for FSTL3 in limiting testis organ size and promoting age-related testicular regression

  6. These findings link FSTL3 and TGFbeta ligand signaling and a novel subset of the synexpression group of genes to organ/tissue-specific regulatory pathways.

  7. These results demonstrate that body composition and glucose homeostasis are differentially regulated by FST and FSTL3 and that their combined loss is associated with increased fat mass and insulin resistance despite elevated insulin production.

  8. Fstl3 as a stress-induced regulator of hypertrophy that controls myocyte size via regulation of Smad signaling.

  9. genomic organization and promoter analysis

  10. regulation and expression patterns of follistatin and FLRG differ; FLRG is distributed in spermatogenic cells of the testis, renal tubules, epithelial cells of the lung, and myocardium

  11. review with emphasis on authors' works on paracrine and autocrine regulation in activities of Fst and Fstl3, regulation of gonadal development, and structural elements necessary for biological activities

  12. Our study indicates that the FLRG protein may contribute to bone formation by inhibiting osteoclast differentiation.

  13. FSTL3 regulation of activin and myostatin is critical for normal adult metabolic homeostasis

  14. In situ hybridization and immunohistochemical analysis revealed that FLRG is expressed in smooth muscle of the aorta and pulmonary artery, valve leaflets of mitral and tricuspid valves, and cardiac muscles in the ventricle of embryonic heart.

  15. expression of follistatin-like 3 can be modulated in adipose tissue and skeletal muscle by chronic obesity

  16. Fstl3 is induced in heart by myocardial stress

Human Follistatin-Like 3 (Secreted Glycoprotein) (FSTL3) interaction partners

  1. Aberrant expression of FSTL3 in preeclampsia led to the dysfunction of trophoblast, indicating its involvement in the pathogenesis of preeclampsia.

  2. deficient FSTL3 expression by asthmatic Bronchial epithelial cells impairs epithelial regulation of human lung fibroblast and fibroblast-to-myofibroblast transition.

  3. FSTL3 expression in invasive breast cancer is inversely associated with tumor size and nuclear grade but it does not predict disease relapse in the short term.

  4. first trimester maternal serum levels not altered in women who develop gestational diabetes mellitus

  5. Human renal dysfunction is the strongest independent predictor of FSTL3 serum concentrations in multivariate analyses.

  6. The serum levels of IL-8, MIP-1 alpha, MIP-1 beta, MMP-8, Resistin, FLRG, and BCAM were significantly higher in breast cancer patients, but LAP and TSH-beta levels were lower.

  7. FSTL3 is a critical mediator of exercise-dependent bone formation

  8. Elevated FSTL-3 concentrations were attributable to preeclampsia and were associated with increased likelihood of later developing preeclampsia, suggesting further study as a biomarker prior to clinically evident disease.

  9. The elevation of Follistatin-like-3 was demonstrated in late second trimester of pregnancy destined to developing preeclampsia, suggesting its causal role in the pathogenesis and prediction of preeclampsia.

  10. Resistance exercise resulted in a significant downregulation of MSTN and FBXO32 mRNA expression and a significant upregulation in FSTL3 and SMURF1 mRNA expression, and carbohydrate and protein feeding have little influence on the these markers expression.

  11. Suggest FSTL3 has a role in paracrine communication between cardiomyocytes and fibroblasts.

  12. Data suggest that serum levels of FSTL3 are higher in pre-eclampsia than in controls; placental expression of FSTL3 is also increased in pre-eclampsia compared with controls; no differences were observed between mild and severe pre-eclampsia.

  13. After eccentric exercise, postmenopausal women not using hormone therapy (HT) expressed higher levels of follistatin-like protein 3 while postmenopausal women using HT showed a significant increased expression over controls.

  14. Maternal and placental FSTL3 concentrations were reduced in GDM women compared with normally pregnant women, suggesting FSTL3 may play an important role in the pathogenesis of gestational diabetes

  15. Structure of myostatin.follistatin-like 3: N-terminal domains of follistatin-type molecules exhibit alternate modes of binding.

  16. GDF9 decreases basal and activin A-induced FST and FSTL3 expression, and this explains, in part, its enhancing effects on activin A-induced inhibin beta(B)-subunit mRNA expression and inhibin B production in hGL cells

  17. FLRG can be successfully detected in maternal plasma in the first trimester of pregnancy but its levels are not significantly altered in the presence of Down syndrome fetuses.

  18. First-trimester FSTL3 levels are associated with glucose intolerance and gestational diabetes later in pregnancy.

  19. Transcription activation of FLRG and follistatin by activin A, through Smad proteins, participates in a negative feedback loop to modulate activin A function

  20. our present data suggest that protein kinase C and A signal transduction pathways differently regulate the expression of FLRG in human ovarian granulosa-luteal cells

蛋白简介FSTL3

蛋白简介

Follistatin-like 3 is a secreted glycoprotein of the follistatin-module-protein family. It may have a role in leukemogenesis.

Gene names and symbols associated with FSTL3

  • follistatin-like 3 (Fstl3)
  • follistatin like 3 (FSTL3)
  • follistatin like 3 (Fstl3)
  • E030038F23Rik 蛋白
  • FLRG 蛋白
  • FSRP 蛋白

Protein level used designations for FSTL3

follistatin-like protein 3 , follistatin-related gene protein , follistatin-related protein 3

GENE ID SPECIES
83554 Mus musculus
10272 Homo sapiens
114031 Rattus norvegicus
515367 Bos taurus
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