C-JUN (JUN) ELISA试剂盒产品概述

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Jun Proto-Oncogene ELISA试剂盒 (JUN)
在www.antibodies-online.cn可供102 Jun Proto-Oncogene (JUN) ELISA试剂盒的15不同的供货商。 再加上,我们可以发C-JUN 抗体 (876)C-JUN 蛋白 (24)和数多这个蛋白质的别的产品。 总共1063 C-JUN产品已列进来了。
AP-1, AP1, B230310J22Rik, Bsk, BSK/DJNK, c-jun, CG2275, CG5680, cJun, D-JNK, d-JRA, d-jun, D-jun/Jra, D-junk, dAP-1, DBSK/JNK, dJNK, DJNK/bsk, dJra, djun, dm-Jun, Dmel\\CG2275, Dmel\\CG5680, fj36h07, jnk, JNK/SAPK, jra, jun, Junc, Junk, l(2)46Ef, l(2)IA109, l(2R)IA109, p39, SAPKa, V, wu:fj36h07, zgc:65863

适于 C-JUN 相互作用对的更多 ELISA 试剂盒

Xenopus laevis Jun Proto-Oncogene (JUN) interaction partners

  1. AP-1(c-Jun/FosB (显示 FOSB ELISA试剂盒)) may play a role in neurogenesis via the induction of FoxD5b expression during early vertebrate development

  2. The cJun transcription factor bound to a variant cAMP response element in the promoter region of tlx3 (显示 TLX3 ELISA试剂盒) and modulated transcription and regulated neurotransmitter phenotype via its transactivation domain

Fruit Fly (Drosophila melanogaster) Jun Proto-Oncogene (JUN) interaction partners

  1. Mir (显示 MYLIP ELISA试剂盒)-8 modulates Drosophila C virus replication by negative regulation of dJun.

  2. Here we uncover a cell non-autonomous requirement for the Epidermal growth factor receptor (Egfr (显示 EGFR ELISA试剂盒)) pathway in the lateral epidermis for sustained dpp (显示 TGFb ELISA试剂盒) expression in the LE. Specifically, we demonstrate that Egfr (显示 EGFR ELISA试剂盒) pathway activity in the lateral epidermis prevents expression of the gene scarface (scaf), encoding a secreted antagonist of JNK (显示 MAPK8 ELISA试剂盒) signaling

  3. Tau and spectraplakin promote synapse formation and maintenance through Jun kinase (显示 MAPK9 ELISA试剂盒) and neuronal trafficking.

  4. n addition to significantly increasing the number of JNK (显示 MAPK8 ELISA试剂盒) target genes identified so far, our results reveal that the LE is a highly heterogeneous morphogenetic organizer, sculpted through crosstalk between JNK (显示 MAPK8 ELISA试剂盒), segmental and AP signalling. This fine-tuning regulatory mechanism is essential to coordinate morphogenesis and dynamics of tissue sealing

  5. malignant transformation of the ras(V12)scrib(1 (显示 SCRIB ELISA试剂盒)) tumors requires bZIP protein Fos, the ETS (显示 ETS1 ELISA试剂盒)-domain factor Ets21c and the nuclear receptor Ftz-F1 (显示 NR5A2 ELISA试剂盒), all acting downstream of Jun-N-terminal kinase (显示 MAPK8 ELISA试剂盒).

  6. Diminished MTORC1-dependent JNK (显示 MAPK8 ELISA试剂盒) activation underlies the neurodevelopmental defects associated with lysosomal dysfunction.

  7. ROS (显示 ROS1 ELISA试剂盒)/JNK (显示 MAPK8 ELISA试剂盒)/p38 (显示 MAPK14 ELISA试剂盒)/Upd (显示 UROD ELISA试剂盒) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  8. Significantly, the JNK (显示 MAPK8 ELISA试剂盒) pathway is responsible for the majority of the phenotypes and transcriptional changes downstream of Notch (显示 NOTCH1 ELISA试剂盒)-Src (显示 SRC ELISA试剂盒) synergy.

  9. This study demonstrated that the mechanism by which Bsk (显示 FRK ELISA试剂盒) is required for pruning is through reducing the membrane levels of the adhesion molecule (显示 NCAM1 ELISA试剂盒) Fasciclin II (显示 NCAM2 ELISA试剂盒) (FasII)

  10. Jra recruits the HP1a (显示 CBX5 ELISA试剂盒)/KDM4A (显示 KDM4A ELISA试剂盒) complex to its gene body region upon osmotic stress to reduce H3K36 methylation levels and disrupt H3K36 methylation-dependent histone deacetylation

Pig (Porcine) Jun Proto-Oncogene (JUN) interaction partners

  1. Porcine reproductive and respiratory syndrome virus -activated TAK-1 (显示 NR2C2 ELISA试剂盒) was essential for the activation of JNK (显示 MAPK8 ELISA试剂盒) and NF-kappaB (显示 NFKB1 ELISA试剂盒) pathways and IL-8 (显示 IL8 ELISA试剂盒) expression.

  2. The effects of prostaglandin F2alpha administration on transcription factor AP-1 expression and the expression of downstream genes involved in luteolysis are reported.

Rabbit Jun Proto-Oncogene (JUN) interaction partners

  1. ICAM1 (显示 ICAM1 ELISA试剂盒) and IL10 (显示 IL10 ELISA试剂盒) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 may be responsible for this upregulation.

Mouse (Murine) Jun Proto-Oncogene (JUN) interaction partners

  1. AP1 factors are important regulators of adult taste cell renewal and their downregulation negatively impacts taste maintenance.

  2. these data indicate that MEF2 (显示 MEF2C ELISA试剂盒) and AP-1 confer antagonistic regulation of Hspb7 (显示 HSPB7 ELISA试剂盒) gene expression in skeletal muscle, with implications for autophagy and muscle atrophy.

  3. results suggest that fibroblasts, c-Jun, and IGF-1 (显示 IGF1 ELISA试剂盒) play key roles in mediating stromal-epithelial interactions that are required for the therapeutic effects of finasteride in benign prostate epithelial cells

  4. These findings highlight a key role of the TLR4 (显示 TLR4 ELISA试剂盒)-NOS1 (显示 NOS1 ELISA试剂盒)-AP1 signaling axis in regulating macrophage polarization

  5. Data suggest that Sf1 and c-jun interact and cooperate to activate the Fdx1 promoter in MA-10 (tumorigenic cell line) and TM3 (non-tumorigenic cell line) Leydig cells; such activation requires different regulatory elements located between -124 and -306 bp of Fdx1 promoter and involves recruitment of Sf1 to this region. (Sf1 = splicing factor 1; c-jun = proto-oncogene c-jun; Fdx1 = ferredoxin 1)

  6. In this study, we discovered that selective upregulation of p39 (显示 ATP6V0D1 ELISA试剂盒) is the underlying mechanism that accommodates the increased functional requirement of Cdk5 (显示 CDK5 ELISA试剂盒) activation during neuronal differentiation. In addition, we demonstrated that p39 (显示 ATP6V0D1 ELISA试剂盒) selectively directs Cdk5 (显示 CDK5 ELISA试剂盒) to phosphorylate protein substrates essential for axonal development, dendritic spine formation, and synaptogenesis. Moreover, our studies suggest opposing roles o

  7. NF-kappaB (显示 NFKB1 ELISA试剂盒) and c-Jun coregulate lipopolysaccharide-induced Fra-1 (显示 FOSL1 ELISA试剂盒) transcription.

  8. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  9. this study shows that c-Jun regulates the activation state of macrophages and promotes arthritis via differentially regulating cyclooxygenase-2 (显示 PTGS2 ELISA试剂盒) and arginase-1 (显示 ARG1 ELISA试剂盒) levels

  10. PLIO treatment inhibited nuclear factorkappaB (NFkappaB) nuclear translocation in B16F10 cells. In addition, PLIO treatment inhibited the phosphorylation of c-Jun Nterminal kinases and AKT (显示 AKT1 ELISA试剂盒)

Human Jun Proto-Oncogene (JUN) interaction partners

  1. Our results indicate that assessing AP1 (显示 FOSB ELISA试剂盒) and PEA3 (显示 ETV4 ELISA试剂盒) transcription factor status might be a good indicator of OAC status. However, we could not detect any associations with disease stage or patient treatment regime. This suggests that the PEA3 (显示 ETV4 ELISA试剂盒)-AP1 (显示 FOSB ELISA试剂盒) regulatory module more likely contributes more generally to the cancer phenotype. In keeping with this observation, depletion of ETV1 (显示 ETV1 ELISA试剂盒) and/or ETV4 (显示 ETV4 ELISA试剂盒) causes an OAC cell growth defect

  2. shRNA-mediated inhibition of JUN decreases AML (显示 RUNX1 ELISA试剂盒) cell survival and propagation in vivo. These data uncover a previously unrecognized role of JUN as a regulator of the unfolded protein response

  3. These findings demonstrate an essential role for the ERK pathway together with c-JUN and c-FOS in the differentiation activity of LukS-PV.

  4. The present study defines the minimal TIM-3 (显示 HAVCR2 ELISA试剂盒) promoter region and demonstrates its interaction with c-Jun during TIM-3 (显示 HAVCR2 ELISA试剂盒) transcription in CD4 (显示 CD4 ELISA试剂盒)(+) T cells.

  5. Taken together, our data demonstrate that JNK (显示 MAPK8 ELISA试剂盒) regulates triple-negative breast cancer (TNBC)tumorigenesis by promoting CSC phenotype through Notch1 (显示 NOTCH1 ELISA试剂盒) signaling via activation of c-Jun and indicate that JNK/c-Jun/Notch1 (显示 NOTCH1 ELISA试剂盒) signaling is a potential therapeutic target for TNBC

  6. Regulation of osteosarcoma cell lung metastasis by the c-Fos/AP-1 target FGFR1

  7. c-jun promoted FOXK1 (显示 FOXK2 ELISA试剂盒)-mediated proliferation and metastasis via orthotopic implantation.

  8. Data provide evidence that AP-1 (显示 FOSB ELISA试剂盒) is a key determinant of endocrine resistance of breast cancer cells by mediating a global shift in the estrogen receptor (显示 ESR1 ELISA试剂盒) transcriptional program.

  9. Comparison of how AP-1 (Jun/Jun dimer) and Epstein-Barr virus Zta recognize methyl groups within their cognate response elements

  10. this study revealed the functional and mechanistic links between CDK5 (显示 CDK5 ELISA试剂盒) and the oncogenic ERK5 (显示 MAPK7 ELISA试剂盒)-AP-1 (显示 FOSB ELISA试剂盒) signaling pathway in the pathogenesis of colorectal cancer.

Zebrafish Jun Proto-Oncogene (JUN) interaction partners

  1. These results support a role for trim69 (显示 TRIM69 ELISA试剂盒) in the development of the zebrafish brain through ap-1 pathway.

  2. CPEB-1 (显示 CPEB1 ELISA试剂盒) control of c-Jun mRNA translation regulates GH gene expression and resulting downstream signaling events (e.g., synaptic plasticity) in the mouse hippocampus.

Cow (Bovine) Jun Proto-Oncogene (JUN) interaction partners

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

  2. dynamic compression stimulates cell proliferation and proteoglycan (显示 Vcan ELISA试剂盒) synthesis in the presence of IL-1beta (显示 IL1B ELISA试剂盒) and/or inhibitors of the MAPKs and NFkappaB and AP-1 signalling pathways

C-JUN (JUN) 抗原简介

Antigen Summary

This gene is the putative transforming gene of avian sarcoma virus 17. It encodes a protein which is highly similar to the viral protein, and which interacts directly with specific target DNA sequences to regulate gene expression. This gene is intronless and is mapped to 1p32-p31, a chromosomal region involved in both translocations and deletions in human malignancies.

Alternative names and synonyms associated with C-JUN (JUN)

  • jun oncogene (jun) Elisa Kit
  • basket (bsk) Elisa Kit
  • C-JUN protein (C-JUN) Elisa Kit
  • c-Jun protein (C-JUN) Elisa Kit
  • c-jun transcription factor (C-JUN) Elisa Kit
  • cyclin-dependent kinase 5, regulatory subunit 2 (p39) (Cdk5r2) Elisa Kit
  • Jun oncogene (Jun) Elisa Kit
  • jun proto-oncogene (JUN) Elisa Kit
  • jun proto-oncogene (jun) Elisa Kit
  • jun proto-oncogene (Jun) Elisa Kit
  • Jun-related antigen (Jra) Elisa Kit
  • AP-1 Elisa Kit
  • AP1 Elisa Kit
  • B230310J22Rik Elisa Kit
  • Bsk Elisa Kit
  • BSK/DJNK Elisa Kit
  • c-jun Elisa Kit
  • CG2275 Elisa Kit
  • CG5680 Elisa Kit
  • cJun Elisa Kit
  • D-JNK Elisa Kit
  • d-JRA Elisa Kit
  • d-jun Elisa Kit
  • D-jun/Jra Elisa Kit
  • D-junk Elisa Kit
  • dAP-1 Elisa Kit
  • DBSK/JNK Elisa Kit
  • dJNK Elisa Kit
  • DJNK/bsk Elisa Kit
  • dJra Elisa Kit
  • djun Elisa Kit
  • dm-Jun Elisa Kit
  • Dmel\\CG2275 Elisa Kit
  • Dmel\\CG5680 Elisa Kit
  • fj36h07 Elisa Kit
  • jnk Elisa Kit
  • JNK/SAPK Elisa Kit
  • jra Elisa Kit
  • jun Elisa Kit
  • Junc Elisa Kit
  • Junk Elisa Kit
  • l(2)46Ef Elisa Kit
  • l(2)IA109 Elisa Kit
  • l(2R)IA109 Elisa Kit
  • p39 Elisa Kit
  • SAPKa Elisa Kit
  • V Elisa Kit
  • wu:fj36h07 Elisa Kit
  • zgc:65863 Elisa Kit

Protein level used designations for Jun Proto-Oncogene (JUN) ELISA试剂盒

v-jun sarcoma virus 17 oncogene homolog , CG5680-PB , CG5680-PE , CG5680-PF , JNK , JUN kinase , Jun N-terminal kinase , Jun NH2-terminal kinase , Jun-N-terminal kinase , Jun-kinase , bsk-PB , bsk-PE , bsk-PF , c-Jun N-terminal kinase , c-Jun aminoterminal kinase , c-Jun-N-terminal kinase , drosophila JNK , AP1 , V-jun avian sarcoma virus 17 oncogene homolog , activator protein 1 , proto-oncogene c-jun , transcription factor AP-1 , CDK5 activator 2 , cyclin-dependent kinase 5 activator 2 , cyclin-dependent kinase 5 regulatory subunit 2 , p39I , AH119 , immediate early , jun A , Jun activation domain binding protein , enhancer-binding protein AP1 , jun oncogene , p39 , proto-oncogene c-Jun , v-jun avian sarcoma virus 17 oncogene homolog , Avian sarcoma virus 17 (v-jun) oncogene homolog , Jun oncogene , v-jun sarcoma virus 17 oncogene , CG2275-PA , CG2275-PB , CG2275-PC , Jra-PA , Jra-PB , Jra-PC , Jun related antigen , complementation group V , lethal(2)1A109 , lethal(2)IA109

379050 Xenopus laevis
44801 Drosophila melanogaster
396913 Sus scrofa
443219 Ovis aries
100008856 Oryctolagus cuniculus
12570 Mus musculus
16476 Mus musculus
3725 Homo sapiens
335916 Danio rerio
24516 Rattus norvegicus
399400 Xenopus laevis
424673 Gallus gallus
609429 Canis lupus familiaris
280831 Bos taurus
100170668 Ovis aries
36057 Drosophila melanogaster
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