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CD244 encodes a cell surface receptor expressed on natural killer (NK) cells (and some T cells) that mediate non-major histocompatibility complex (MHC) restricted killing. 再加上，我们可以发CD244 蛋白 (27) 和 CD244 试剂盒 (10)和数多这个蛋白质的别的产品。
Showing 10 out of 128 products:
Mouse (Murine) Polyclonal CD244 Primary Antibody for FACS, IHC - ABIN4898999
Cao, Wagers, Karsunky, Zhao, Reeves, Wong, Stevenson, Weissman, Contag: Heme oxygenase-1 deficiency leads to disrupted response to acute stress in stem cells and progenitors. in Blood 2008
Human Polyclonal CD244 Primary Antibody for IHC (p) - ABIN740730
Peiffer, Wang, Zimmerman, Ransom, Carmella, Kuo, Chen, Oshima, Huang, Hecht, Stoner: Dietary Consumption of Black Raspberries or Their Anthocyanin Constituents Alters Innate Immune Cell Trafficking in Esophageal Cancer. in Cancer immunology research 2016
Human Polyclonal CD244 Primary Antibody for FACS - ABIN4895306
Mattiola, Pesant, Tentorio, Molgora, Marcenaro, Lugli, Locati, Mavilio: Priming of Human Resting NK Cells by Autologous M1 Macrophages via the Engagement of IL-1β, IFN-β, and IL-15 Pathways. in Journal of immunology (Baltimore, Md. : 1950) 2015
Human Monoclonal CD244 Primary Antibody for FACS, IP - ABIN120311
Romero, Zapater, Calvo, Kalko, de la Fuente, Tovar, Ockeloen, Pizcueta, Engel: CD229 (Ly9) lymphocyte cell surface receptor interacts homophilically through its N-terminal domain and relocalizes to the immunological synapse. in Journal of immunology (Baltimore, Md. : 1950) 2005
2B4 expression is increased on CD4 (显示 CD4 抗体)+ T cells in septic patients
Self-iNKR(-) NK cells in X-linked lymphoproliferative disease 1 patients are functional even in resting conditions, suggesting a role of the inhibitory 2B4/CD48 (显示 CD48 抗体) pathway in the education process during NK-cell maturation.
our results provide strong evidence that CD244 co-operates with c-Kit (显示 KIT 抗体) to regulate leukemogenesis through SHP-2 (显示 PTPN11 抗体)/p27 (显示 PAK2 抗体) signaling.
SLAMF4 gene and surface protein expression was down-regulated in CD8 (显示 CD8A 抗体)+ T cells from SLE patients compared with that in cells obtained from healthy donors. SLAMF4+ effector memory cells produced more granzyme B (显示 Gzmb 抗体) and perforin (显示 PRF1 抗体) than SLAMF4- cells.
GSK-3 (显示 GSK3b 抗体) undergoes inhibitory phosphorylation at regulatory serine residues by the engagement of NKG2D (显示 KLRK1 抗体) and 2B4, either individually or in combination.
Cytomegalovirus-Induced Expression of CD244 after Liver Transplantation Is Associated with CD8 (显示 CD8A 抗体)+ T Cell Hyporesponsiveness to Alloantigen
CD8 (显示 CD8A 抗体)(+) T cells require 2B4 for Epstein-Barr virus-specific immune control, because 2B4 blockade after CD8 (显示 CD8A 抗体)(+) T-cell depletion did not further aggravate symptoms of the infection.
CD244 expression/signaling in tuberculosis correlates with high levels of a long noncoding RNA (lncRNA)-BC050410 [named as lncRNA-AS-GSTT1(1 (显示 GSTT1 抗体)-72) or lncRNA-CD244] in the CD244(+)CD8 (显示 CD8A 抗体)(+) T-cell subpopulation.
show that in addition to NKG2D (显示 KLRK1 抗体), human CEACAM1 (显示 CEACAM1 抗体) can inhibit NK-cell activation via NKp30 (显示 NCR3 抗体) or 2B4
this study investigates the structural requirements for the recruitment of the human-activating NK-cell receptors NKG2D (显示 KLRK1 抗体) and 2B4 to detergent-resistant membrane fractions in the murine BA/F3 cell line and in the human NK-cell line NKL (显示 GLIS2 抗体).
2B4 expression is increased on CD4 (显示 CD4 抗体)+ T cells in septic animals
2B4 dampens T-independent B cell responses.
DCs from CD244(-/-) mice elicited increased NK cell activation in vitro. These data add CD244 to a growing list of immuno-modulatory receptors found on DCs.
Data show that enforced expression of inhibitor of differentiation protein Id3 (显示 ID3 抗体) specifically increased the persistence of Cd244 protein 2B4(+) virus-specific CD8 (显示 CD8A 抗体)(+) T cells.
Molecular dynamic simulation explains unbinding and unfolding of CD48 (显示 CD48 抗体)-2B4 protein complexes.
Signaling via SLAMF4 controls expansion of cytotoxic CD8alphabeta(+) IELs, which regulate the reversible depletion of lamina propria phagocytes and inflammation in the small intestine.
CD244 (2B4) has a regulatory effect on mast cells and eosinophils in murine allergic inflammation.
NKG2D (显示 KLRK1 抗体) ligation relieves 2B4-mediated NK-cell self-tolerance in mice.
blockade of CD28 (显示 CD28 抗体) signals in the presence of preserved CTLA-4 (显示 CTLA4 抗体) signals results in the unique up-regulation of 2B4 on primary CD8 (显示 CD8A 抗体)(+) effectors, and that this 2B4 expression plays a critical functional role in controlling antigen-specific T8 cell responses
In the absence of SAP (显示 APCS 抗体), signaling through the SLAM (显示 SLAMF1 抗体) family members Ly108 (显示 SLAMF6 抗体) and 2B4 resulted in increased recruitment of the SHP-1 (显示 PTPN6 抗体) phosphatase, associated with altered SHP-1 (显示 PTPN6 抗体) localization and decreased activation of Src (显示 SRC 抗体) kinases at the synapse.
This gene encodes a cell surface receptor expressed on natural killer (NK) cells (and some T cells) that mediate non-major histocompatibility complex (MHC) restricted killing. The interaction between NK-cell and target cells via this receptor is thought to modulate NK-cell cytolytic activity. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
CD244 molecule, natural killer cell receptor 2B4
, natural killer cell receptor 2B4
, NK cell activation inducing ligand NAIL
, NK cell activation-inducing ligand
, NK cell type I receptor protein 2B4
, SLAM family member 4
, signaling lymphocytic activation molecule 4
, CD244 natural killer cell receptor 2B4
, non MHC restricted killing associated
, non-MHC restricted killing associated
, transmembrane NK cell receptor 2B4
, NK cell receptor 2B4