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抗Human S100A9 抗体:
抗Rat (Rattus) S100A9 抗体:
抗Mouse (Murine) S100A9 抗体:
Human Polyclonal S100A9 Primary Antibody for ICC, IF - ABIN409938
Sumi, Shimizu, Himeno: Involvement of Nrf2 activation in the upregulation of S100A9 by exposure to inorganic arsenite. in International journal of molecular medicine 2012
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Human Monoclonal S100A9 Primary Antibody for ELISA, WB - ABIN948434
Li, Riau, Setiawan, Mehta, Ti, Tong, Tan, Beuerman: S100A expression in normal corneal-limbal epithelial cells and ocular surface squamous cell carcinoma tissue. in Molecular vision 2011
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Human Polyclonal S100A9 Primary Antibody for WB - ABIN519967
Bianchi, Niemiec, Siler, Urban, Reichenbach: Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent. in The Journal of allergy and clinical immunology 2011
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Human Polyclonal S100A9 Primary Antibody for IHC (p), IHC - ABIN449656
McCormick, Rahimi, Bobryshev, Gaus, Zreiqat, Cai, Lord, Geczy: S100A8 and S100A9 in human arterial wall. Implications for atherogenesis. in The Journal of biological chemistry 2005
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Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN111889
Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. in Journal of clinical pathology 1989
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Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN111888
Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. in Clinical and experimental immunology 1990
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Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN238498
Mortezai, Harder, Schnabel, Moors, Gauly, Schlüter, Wagener, Buck: Tandem affinity depletion: a combination of affinity fractionation and immunoaffinity depletion allows the detection of low-abundance components in the complex proteomes of body fluids. in Journal of proteome research 2010
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Human Monoclonal S100A9 Primary Antibody for ELISA, WB - ABIN519968
Feng, Lee, Chang, Chan, Kuo, Lin, Chung, Kuo, Yu, Lin, Wang, Chou, Huang, Kuo: CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancer. in American journal of respiratory and critical care medicine 2012
Human Monoclonal S100A9 Primary Antibody for IA, FACS - ABIN2192037
Robinson, Tessier, Poulsom, Hogg: The S100 family heterodimer, MRP-8/14, binds with high affinity to heparin and heparan sulfate glycosaminoglycans on endothelial cells. in The Journal of biological chemistry 2002
In pediatric inflammatory bowel disease treatment groups, a significant inverse association was found between the quality of life and the levels of fecal calprotectin (显示 S100A8 抗体).
Fecal calprotectin (显示 S100A8 抗体) (FC) was elevated in two-thirds of stool specimens of patients with cystic fibrosis (显示 S100A8 抗体) (CF). Increased FC was more common in patients with pancreatic insufficiency. Whether increased FC reflects intestinal inflammation in patients with CF remains to be determined.
In healthy children, 95th Percentile values ranged from 1519 mg/kg at 0 months to 54.4 mg/kg at 144 months for fecal calprotectin (显示 S100A8 抗体) (fCP) and from 9.9 mg/kg at 0 months to 0.2 mg/kg at 144 months for fecal eosinophil-derived neurotoxin (显示 RNASE2 抗体) (fEDN). There was a significant association between age and fCP concentrations and age and fEDN concentrations and a significant association between fEDN and fCP.
Authors found that downregulation of S100A9 significantly reduced the expression of NF-kappaB (显示 NFKB1 抗体), phosphorylation of NF-kappaB (显示 NFKB1 抗体) and Bcl-2 (显示 BCL2 抗体), as well as the expression of MMP7 (显示 MMP7 抗体) and MMP2 (显示 MMP2 抗体). Restoration of NF-kappaB (显示 NFKB1 抗体) expression sufficiently reversed the inhibitory effects on cell proliferation and invasion induced by S100A9 downregulation in vitro and in vivo.
Urinary NGAL (显示 LCN2 抗体) is a good predictor of mortality in established pediatric acute kidney injury (AKI) of heterogeneous etiology and outperforms calprotectin (显示 S100A8 抗体) and KIM-1 (显示 HAVCR1 抗体). Urinary calprotectin (显示 S100A8 抗体) and KIM-1 (显示 HAVCR1 抗体) predict renal replacement therapy requirement in pediatric AKI.
Identify proteins that interact with calgranulin B to suppress the proliferation of colon cancer cells.
This study demonstrated that S100A9 was detected in the serums of Alzheimer disease patients
Expression levels of S100A9 and Tenascin-C (显示 TNC 抗体) were correlated with TNM (显示 ODZ1 抗体) stages and metastasis in colorectal cancer.
In Guatemalan preschoolers, the median fecal calprotectin (显示 S100A8 抗体) value was 58 mg/kg, with a mean of 98 +/- 136 mg/kg and a range from 10 to 950 mg/kg; 61% of values were above the manufacturer's cut-off for elevated concentration and 51% exceeded an age-adjusted criterion. The central tendency (mean or median) and distribution were generally shifted to the right in relation to comparable reports from children across the world.
Low S100A9 expression is associated with head and neck neoplasms.
Data suggest that up-regulation of S100A8 (显示 S100A8 抗体) and S100A9 is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.
Study verified porcine calprotectin (S100A8 (显示 S100A8 抗体)/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.
this study shows that S100A9 inhibits B lymphopoiesis by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta (显示 IL1B 抗体)
data suggest that S100A8 (显示 S100A8 抗体)/A9 acts directly on BV-2 microglial cells via binding to TLR4 (显示 TLR4 抗体) and RAGE (显示 AGER 抗体) on the membrane and then stimulates the secretion of proinflammatory cytokines through ERK (显示 EPHB2 抗体) and JNK (显示 MAPK8 抗体)-mediated NF-kappaB (显示 NFKB1 抗体) activity in BV-2 microglial cells.
Like S100A8 (显示 S100A8 抗体), S100A9 and S100A8 (显示 S100A8 抗体)/A9 reduced neutrophil influx in acute lung injury provoked by lipopolysaccharide (LPS (显示 TLR4 抗体)) challenge.
findings reveal unexpected gene expression differences between WT and KO mice at a young age (in the absence of physiological stress), and address the hypothesis that Mrp8 (显示 S100A8 抗体) and Mrp14 accumulation promotes age-related inflammation
S100A8 (显示 S100A8 抗体) and S100A9 aggravate Coxsackievirus B3-induced myocarditis and might serve as therapeutic targets in inflammatory cardiomyopathies.
An excessive expansion of monocytic-myeloid-derived suppressor cells in S100A9-/- neonates compared to wild-type neonates. This strong baseline expansion was associated with hyperinflammatory responses during endotoxemia and fatal septic courses
Data support a role of S100A9 to initiate and amplify the neutrophilic inflammation in asthma, possibly via inducing IL-1beta (显示 IL1B 抗体), IL-17 (显示 IL17A 抗体) and IFN-gamma (显示 IFNG 抗体).
The findings show that reduced PTP1B (显示 PTPN1 抗体) responses contribute to disease symptoms in part by enhancing S100A9 expression during viral-associated chronic obstructive pulmonary disease exacerbations.
Neutrophil-derived S100A8 (显示 S100A8 抗体)/A9 promotes thrombocytosis in diabetic mice
S100A9 knockdown almost completely abrogated the effects of IRF7 (显示 IRF7 抗体) deletion on granulocytic myeloid-derived suppressor cells development and tumor metastasis.
Perinatal alarmins S100A8 (显示 S100A8 抗体) and S100A9 specifically altered MyD88 (显示 MYD88 抗体)-dependent proinflammatory gene programs. S100 programming prevented hyperinflammatory responses without impairing pathogen defense. TRIF (显示 RNF138 抗体)-adaptor-dependent regulatory genes remained unaffected by perinatal S100 programming and responded strongly to lipopolysaccharide, but were barely expressed.
Calprotectin (显示 S100A8 抗体) expression in laminar epidermis occurs after extravasation of leukocytes indicating that leukocyte emigration may be an initiating factor in laminar epithelial stress and inflammation in black walnut extract-induced laminitis.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and altered expression of this protein is associated with the disease cystic fibrosis.
, S100 calcium-binding protein A9 (calgranulin B)
, calgranulin B
, calprotectin L1H subunit
, leukocyte L1 complex heavy chain
, migration inhibitory factor-related protein 14
, protein S100-A9
, S100 calcium binding protein A9 (calgranulin B)
, intracellular calcium-binding protein (MRP14)
, myeloid-related protein 14
, protein MRP-126
, S100 calcium binding protein A9
, S100 calcium-binding protein A9
, neutrophil cytosolic 23 kDa protein
, RNA-binding region containing protein 2-like