anti-ATP5B (ATP5B) 抗体产品概述

Horse (Equine) ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, beta Polypeptide (ATP5B) interaction partners

1. This study showed that beta-actin (显示 ACTB 抗体), L32 (显示 RPL32 抗体) ribosomal protein, and ATP5B proteins were the most stabily expressed genes in cryopreserved horse semen.

Mouse (Murine) ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, beta Polypeptide (ATP5B) interaction partners

1. The results of this study found that the expression levels of Cd68 (显示 CD68 抗体) and Atp5b were significantly correlated with the neurofibrillary tangle burden in the Alzheimer's Disease brain and with their cognition.

2. Results show that the relationship between ATPsyn-beta and insulin (显示 INS 抗体) secretion deficiency suggests that ATPsyn-beta potentially could serve as a marker for type 2 diabetes mellitus disease risk in women with polycystic ovary syndrome.

3. Data show that overexpressed ATP synthase subunit-beta (ATP5b) was significantly located on renal proximal tubules in db/db (显示 LEPR 抗体) mice.

4. inhibition of calmodulin (CaM) completely abolished ATPSbeta-induced Akt activation in liver cells

5. association of calcium channel alpha2/delta subunit 1 and ATP5b occurs in intracellular membranes and at the plasma membrane of developing muscle cells, where they form a signaling complex capable of accelerating the rate of decline of calcium transients

6. Adipocyte recycling of apoA-I (显示 APOA1 抗体) is a selective process that involves the ectopically expressed beta-subunit (显示 POLG 抗体) of ATP synthase.

7. Neuron-specific changes for F(1)-complex in the Ppt1 (显示 PPT1 抗体)-deficient cells and give clues for a possible link between lipid metabolism and neurodegeneration in Infantile neuronal ceroid lipofuscinosis (显示 CLN6 抗体).

8. Downregulated ATP5b also reduced ATP production in the murine macrophages infected with B. anthracis spores.

9. CyPD association to the lateral stalk of ATP synthase modulates the activity of the complex

Human ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, beta Polypeptide (ATP5B) interaction partners

1. findings show the T163S mutation affects the catalytic activity with a decrease in Ca2 (显示 CA2 抗体)+-dependent and an increase in Mg2 (显示 MUC7 抗体)+-dependent ATP hydrolysis and desensitizes the permeability transition pore to Ca2 (显示 CA2 抗体)+, resulting in increased resistance to Ca2 (显示 CA2 抗体)+-dependent mitochondrial depolarization and to cell death

2. experiments implicate circulating NEFA (显示 NUCB2 抗体) in obesity in suppressing muscle protein metabolism, and establish impaired beta-F1-ATPase translation as an important consequence of obesity

3. ATP5B, as a binding partner of a metastasis-related short peptide (B04) on prostate cancer cells, is involved in promoting prostate cancer metastasis.

4. Results show that the relationship between ATPsyn-beta and insulin (显示 INS 抗体) secretion deficiency suggests that ATPsyn-beta potentially could serve as a marker for type 2 diabetes mellitus disease risk in women with polycystic ovary syndrome.

5. These results suggested that increasing levels of ATP5B and ETFB (显示 ETFB 抗体) were associated with worsening renal injury.

6. In this instance, the ATP5B/CALR (显示 CALR 抗体)/HSP90B1 (显示 HSP90B1 抗体)/HSPB1 (显示 HSPB1 抗体)/HSPD1 (显示 HSPD1 抗体)-signaling network was revealed as the predominant target which was associated with the majority of the observed protein-protein interactions. As a result, the identified targets may be useful in explaining the anticancer mechanisms of ursolic acid and as potential targets for colorectal cancer therapy.

7. High mRNA levels of ATP5B are associated with glioblastoma.

8. Hypermethylation of ATPsyn-beta gene promoter is associated with a down-regulated mRNA expression and chemoresistance in AML (显示 RUNX1 抗体) patients.

9. Hemoglobin (显示 HBB 抗体) - a novel ligand of hepatocyte ectopic F1-ATPase (显示 DNAH8 抗体)

10. PKA phosphorylates the ATPase inhibitory factor 1 (显示 ATPIF1 抗体) and inactivates its capacity to bind and inhibit the mitochondrial H(+)-ATP synthase.

Pig (Porcine) ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, beta Polypeptide (ATP5B) interaction partners

1. ATP5B probably plays a key role in porcine skeletal muscle development.

Cow (Bovine) ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, beta Polypeptide (ATP5B) interaction partners

1. These data indicate that interaction with the alpha-phosphate is not crucial for efficient catalysis, but likely contributes to avoid formation of ADP-inhibited intermediates.

2. Molecular dynamics simulations show that the nucleotide-free beta subunit (显示 POLG 抗体), initially in the open, low-affinity state, undergoes a spontaneous closing transition to the half-open state in response to the gamma rotation in the synthesis direction.

3. analysis of a new catalytic intermediate in the pathway of ATP hydrolysis by F1-ATPase (显示 DNAH8 抗体) from bovine heart mitochondria

4. Molecular dynamics simulations of F1-ATPase (显示 DNAH8 抗体) suggest that the equilibrium conformation of a nucleotide-free beta-subunit (显示 POLG 抗体) is the open conformation and that the transition from the closed to the open conformation can occur in a few tens of nanoseconds.

5. molecular dynamic analysis of interaction between the gamma-subunit and a C-terminal fragment of the beta-subunit (显示 POLG 抗体) of F1-ATPase (显示 DNAH8 抗体)

6. Inhibition arises by IF(1 (显示 ATPIF1 抗体)) imposing the structure and properties of the beta(TP)-alpha(TP) interface on the beta(DP)-alpha(DP (显示 SRPR 抗体)) interface, thereby preventing it from hydrolyzing the bound ATP.

7. The catalytic sites of beef heart mitochondrial F1-ATPase (显示 DNAH8 抗体) were studied by electron spin echo envelope modulation (ESEEM) spectroscopy, using Mn(II) as a paramagnetic probe