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SKIL 抗体 (AA 539-588)

This 兔 多克隆 anti-SKIL antibody specifically detects SKIL in WB. The antibody is reactive with 人, 小鼠, 大鼠 和 豚鼠 samples.
产品编号 ABIN6745607
发货至: 中国
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Quick Overview for SKIL 抗体 (AA 539-588) (ABIN6745607)

抗原

See all SKIL 抗体
SKIL (SKI-Like Oncogene (SKIL))

适用

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人, 小鼠, 大鼠, 豚鼠

宿主

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克隆类型

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多克隆

标记

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This SKIL antibody is un-conjugated

应用范围

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Western Blotting (WB)
  • 抗原表位

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    AA 539-588

    特异性

    Human SKIL

    预测反应

    Percent identity by BLAST analysis: Human (100%) Dog, Bovine, Rabbit, Horse, Pig (92%) Chicken (90%) Mouse, Rat (85%).

    纯化方法

    Immunoaffinity purified

    免疫原

    Synthetic peptide located between aa539-588 of human SKIL (P12757-3, NP_001138569). Percent identity by BLAST analysis: Human, Chimpanzee, Gorilla, Orangutan, Gibbon, Monkey (100%), Galago, Marmoset, Hamster, Elephant, Panda, Dog, Bovine, Rabbit, Horse, Pig, Opossum, Zebra finch, Lizard (92%), Turkey, Chicken (90%), Mouse, Rat, Bat (85%).

    Type of Immunogen: Synthetic peptide
  • 应用备注

    Approved: WB (0.2 - 1 μg/mL)

    Usage: Western Blot: Suggested dilution at 1 μg/mL in 5 % skim milk / PBS buffer, and HRP conjugated anti-Rabbit IgG should be diluted in 1: 50,000 - 100,000 as secondary antibody.

    说明

    Target Species of Antibody: Human

    限制

    仅限研究用
  • 状态

    Lyophilized

    溶解方式

    Distilled water

    浓度

    Lot specific

    缓冲液

    Lyophilized from PBS with 2 % sucrose

    注意事项

    Avoid repeat freeze-thaw cycles.

    储存条件

    4 °C,-20 °C

    储存方法

    Long term: -20°C, the use of 50% glycerol is recommended if storing aliquots in -20°C for long term use (up to 1 year)
    Short term (less than 1 week): 4°C. Avoid freeze-thaw cycles.
  • 抗原

    SKIL (SKI-Like Oncogene (SKIL))

    别名

    SKIL / SNO / SnoN

    背景

    Name/Gene ID: SKIL

    Synonyms: SKIL, Ski-related oncogene, SnoI, SnoN, SKI-like oncogene, Sno, Ski-related oncogene snoN, Ski-related protein, Ski-like protein, SnoA

    基因ID

    6498

    NCBI登录号

    NP_001138569

    UniProt

    P12757

    途径

    Positive Regulation of Response to DNA Damage Stimulus
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