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Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. 再加上，我们可以发Retinoic Acid Receptor Responder (Tazarotene Induced) 3 蛋白 (4) 和 Retinoic Acid Receptor Responder (Tazarotene Induced) 3 试剂盒 (1)和数多这个蛋白质的别的产品。
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The current study establishes that hypoxia can profoundly influence the inducible RIG-I (显示 DDX58 抗体) protein expression in malignant cells of both human and murine origin, whereas this phenomenon was not identified in nonmalignant cell lines or primary cells.
our study demonstrates that the novel pathway lncRNA Ftx/miR (显示 MLXIP 抗体)-545/RIG-I (显示 DDX58 抗体) promotes hepatocellular carcinoma development
this review describes antiviral activities of RIG-I (显示 DDX58 抗体) against influenza viruses (standing on three legs)
Study uncovered a novel aspect of Rig-I (显示 DDX58 抗体) in monitoring gut (显示 GUSB 抗体) microbiota through regulating IgA (显示 IgA 抗体) and IL6 (显示 IL6 抗体)-STAT3 (显示 STAT3 抗体)-dependent Reg3gamma pathway. Besides, Rig-I (显示 DDX58 抗体) loss could also promote colorectal cancer progression both in the presence and absence of intestinal bacteria.
this study indicated that increased expression of TIG3 in primary glioblastoma is a novel biomarker for predicting poor outcome of patients. We then hypothesize that TIG3 may function in a different pattern in glioblastoma
Foot-and-mouth disease virus Viroporin 2B antagonizes RIG-I (显示 DDX58 抗体)-mediated antiviral effects by inhibition of its protein expression.
Overexpression of TIG3 suppresses tumor growth in hepatocellular carcinoma
miR (显示 MLXIP 抗体)-34a is an antioncogene in multiple tumors, in this study, RIG-I (显示 DDX58 抗体) and miR (显示 MLXIP 抗体)-34a suppressed cell growth, proliferation, migration, and invasion in cervical cancer cells in vitro. miR (显示 MLXIP 抗体)-34a was validated as a new regulator of RIG-I (显示 DDX58 抗体) by binding to its 3' untranslated region and upregulating its expression level.
Letter: Interleukin-22 (显示 IL22 抗体) inhibits tazarotene-induced gene 3 expression in keratinocytes via MAPK (显示 MAPK1 抗体)-ERK1/2 and JAK2 (显示 JAK2 抗体)/STAT3 (显示 STAT3 抗体) signaling.
TRIM25 (显示 TRIM25 抗体) plays an additional role in RIG-I (显示 DDX58 抗体)/MDA5 (显示 IFIH1 抗体) signaling other than RIG-I (显示 DDX58 抗体) ubiquitination via activation of NF-kappaB (显示 NFKB1 抗体).
Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. These effects are mediated by specific nuclear receptor proteins that are members of the steroid and thyroid hormone receptor superfamily of transcriptional regulators. RARRES1, RARRES2, and RARRES3 are genes whose expression is upregulated by the synthetic retinoid tazarotene. RARRES3 is thought act as a tumor suppressor or growth regulator.
retinoic acid receptor responder (tazarotene induced) 3
, retinoic acid receptor responder protein 3
, HRAS-like suppressor 4
, RAR-responsive protein TIG3
, retinoic acid-inducible gene 1
, retinoid-inducible gene 1 protein
, tazarotene-induced gene 3 protein