anti-CD69 (CD69) 抗体

CD69 encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. 再加上,我们可以发CD69 试剂盒 (25)CD69 蛋白 (20)和数多这个蛋白质的别的产品。

列出全部抗体 基因 基因ID UniProt
CD69 29187  
CD69 12515 P37217
CD69 969 Q07108
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antibodies-online.cn销售最多的anti-CD69 抗体

Showing 10 out of 304 products:

产品编号 适用 宿主 标记 应用范围 图像 规格 交付 价格 详细
小鼠 非结合性 Func, FACS, IHC (fro), WB 0.1 mg 6至8个工作日
非结合性 WB 100 μL 2至3个工作日
小鼠 非结合性 ELISA, FACS, IHC, IHC (p) Human Small Intestine: Formalin-Fixed, Paraffin-Embedded (FFPE) Human Tonsil: Formalin-Fixed, Paraffin-Embedded (FFPE) 50 μL 11至14个工作日
非结合性 ELISA, WB Western blot analysis of extracts from HuvEc cells, using CD69 Antibody. The lane on the right is treated with the synthesized peptide. 100 μg 2至3个工作日
小鼠 FITC FACS 100 Tests 6至8个工作日
小鼠 非结合性 FACS, Func, IHC (f) Surface staining of human peripheral blood using anti-CD69 antibody (clone FN50) after overnight activation of T cells by anti-CD3 (clone MEM-57). Surface staining of human PHA-activated peripheral blood using anti-CD69 antibody (clone FN50) purified, GAM-APC. 0.1 mg 5至7个工作日
非结合性 IF (p), IHC (p), WB Formalin-fixed and paraffin embedded human lung carcinoma labeled with Rabbit Anti CD69/CLEC2C/AIM Polyclonal Antibody, Unconjugated (ABIN741150) at 1:200 followed by conjugation to the secondary antibody and DAB staining Formalin-fixed and paraffin embedded mouse lung labeled with Rabbit Anti CD69/CLEC2C/AIM Polyclonal Antibody, Unconjugated (ABIN741150) at 1:200 followed by conjugation to the secondary antibody and DAB staining 100 μL 3至7个工作日
小鼠 APC FACS 100 Tests 6至8个工作日
小鼠 PE FACS 100 Tests 6至8个工作日
非结合性 ELISA, WB 100 μL Available

引用最多的anti-CD69 抗体

  1. Mouse (Murine) Monoclonal CD69 Primary Antibody for FACS - ABIN2689380 : Bendelac, Matzinger, Seder, Paul, Schwartz: Activation events during thymic selection. in The Journal of experimental medicine 1992 (PubMed)
    Show all 15 Pubmed References

  2. Human Monoclonal CD69 Primary Antibody for Func, FACS - ABIN263925 : Nielsen, Afzelius, Ersbøll, Nielsen, Hansen: Expression of the activation antigen CD69 predicts functionality of in vitro expanded peripheral blood mononuclear cells (PBMC) from healthy donors and HIV-infected patients. in Clinical and experimental immunology 1998 (PubMed)
    Show all 9 Pubmed References

  3. Human Monoclonal CD69 Primary Antibody for FACS, IHC - ABIN965813 : Iafrate, Bronson, Skowronski: Separable functions of Nef disrupt two aspects of T cell receptor machinery: CD4 expression and CD3 signaling. in The EMBO journal 1997 (PubMed)
    Show all 4 Pubmed References

  4. Human Monoclonal CD69 Primary Antibody for FACS, Func - ABIN302059 : Drbal, Moertelmaier, Holzhauser, Muhammad, Fuertbauer, Howorka, Hinterberger, Stockinger, Schütz: Single-molecule microscopy reveals heterogeneous dynamics of lipid raft components upon TCR engagement. in International immunology 2007 (PubMed)
    Show all 5 Pubmed References

  5. Human Monoclonal CD69 Primary Antibody for FACS, Func - ABIN302016 : Konjevi?, Jovi?, Vuleti?, Radulovi?, Jeli?, Spuzi?: CD69 on CD56+ NK cells and response to chemoimmunotherapy in metastatic melanoma. in European journal of clinical investigation 2007 (PubMed)
    Show all 5 Pubmed References

  6. Human Monoclonal CD69 Primary Antibody for FACS, Func - ABIN302038 : Pitsios, Dimitrakopoulou, Tsalimalma, Kordossis, Choremi-Papadopoulou: Expression of CD69 on T-cell subsets in HIV-1 disease. in Scandinavian journal of clinical and laboratory investigation 2008 (PubMed)
    Show all 5 Pubmed References

  7. Human Monoclonal CD69 Primary Antibody for FACS, Func - ABIN302090 : Hrdinka, Dráber, Stepánek, Ormsby, Otáhal, Angelisová, Brdicka, Paces, Horejsí, Drbal: PRR7 is a transmembrane adaptor protein expressed in activated T cells involved in regulation of T cell receptor signaling and apoptosis. in The Journal of biological chemistry 2011 (PubMed)
    Show all 5 Pubmed References

  8. Human Monoclonal CD69 Primary Antibody for FACS, Func - ABIN301987 : Tomescu, Chehimi, Maino, Montaner: NK cell lysis of HIV-1-infected autologous CD4 primary T cells: requirement for IFN-mediated NK activation by plasmacytoid dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2007 (PubMed)
    Show all 5 Pubmed References


Mouse (Murine) CD69 interaction partners

  1. This study reveals the importance of CD69 to prolong the thymic residency time of developing immature precursors for proper differentiation of a T cell subset, NKT2 cells.

  2. These studies showed that VCAM1 contributes not only to the initiation of myelination but also to its regulation through controlling the abundance of CD69, demonstrating that an intercellular molecule whose primary role is in the immune system can also play an unexpected role in the CNS.

  3. AIM plays an orchestrating role in the resolution process of inflammation by altering the profile of pulmonary lipid mediators

  4. Data show that a subset of inflammation-induced CD69(+) Kupffer cells which can feedback inhibit CD4 T cell response via cell surface TGF-beta1 at the late stage of immune response against infection.

  5. Specific niches for lung-resident memory CD8+ T cells at the site of tissue regeneration enable CD69-independent maintenance.

  6. this study shows that CD69 serves as a key mediator of the pathogenesis of psoriasis by controlling LAT1-CD98-mediated metabolic cues

  7. Loss of CD69 is associated with enhanced protection against vaccinia virus infection.

  8. The data highlight the contribution of CD69 as a nonredundant key regulator of BIC/miR-155-dependent regulatory T cell development and homeostasis.

  9. Data show that after tumor necrosis factor alpha-induced protein 8 like-2 (TIPE2) gene was down-regulated, the expression of the CD69 antigen was increased, and the proliferation of T lymphocytes and the secretion of cytokines IL-2 and IFN-gamma were enhanced.

  10. The generation of cancer-specific CD8(+) CD69(+)-expressing lymphocytes that inhibit colon cancer growth has been described.

  11. Maintenance of immune tolerance by Foxp3+ regulatory T cells requires CD69 expression.

  12. AIM(-/-) mice were highly susceptible to steatosis-associated HCC development, whereas no AIM(+/+) mouse developed the disease despite comparable liver inflammation and fibrosis in response to a long-term high-fat diet.

  13. our findings reveal potent strategies to safely increase AIM levels, which could form the basis for developing novel therapies for obesity

  14. CD69 interference with S1P1 function regulates peripheral T cell retention.

  15. The NKG2A expression was increased in hepatic CD69-NK cells.

  16. Report role of ZAP-70 kinase mutation/deletion on thymocyte-positive selection via CD69 signaling.

  17. The early activation marker CD69 regulates the expression of chemokines and CD4 T cell accumulation in intestine.

  18. Data indicate that CD69 is upregulated on endogenous dendritic cells (DC) upon activation.

  19. Intron I acts as an important regulatory element of CD69 expression.

  20. Data show that CD69 is critical for the generation and maintenance of professional memory T-helper (Th) lymphocytes, which can efficiently help humoral immunity in the late phase.

Human CD69 interaction partners

  1. In the presence of fusion protein-tyrosine kinases ITK-FER and ITK-SYK, signal transducer and activator of transcription 3 (STAT3) was highly phosphorylated and early activation antigen CD69 (CD69) was significantly elevated.

  2. CD69(+) T cells were associated with disease recurrence.

  3. CD69 is a direct target of miR-367-3p.

  4. The frequency of CD69+ T cells was significantly higher in CD8+ and CD4+ T cells in nasal polyps compared with the peripheral blood of patients with chronic rhinosinusitis.

  5. this paper shows that decrease of CD69 levels on TCR Valpha7.2(+)CD4(+) innate-like lymphocytes is associated with impaired cytotoxic functions in chronic hepatitis B virus-infected patients

  6. AIM expression in the kidney was associated with urinary protein and decline in kidney function.

  7. Higher CD69 expression were less sensitive to bendamustine and is associated with chronic lymphocytic leukemia.

  8. In vitro functional assays showed that CD69(+) Treg cells exerted an important suppressive effect on the activation of T effector cells

  9. results demonstrate the functional and mechanistic interplays between CD69 and S100A8/S100A9 in supporting Treg-cell differentiation

  10. Elevated expression of CD69 and CD161 on NK cells can be considered as immunological risk markers in RSA and IVF failure.

  11. Human tumor-infiltrating CD4+CD69+ T cells suppress T cell proliferation via membarene -bound TGF-beta1.

  12. these findings identify CD69 and galectin-1 to be a novel regulatory receptor-ligand pair that modulates Th17 effector cell differentiation and function.

  13. REVIEW: CD69 exerts a complex immunoregulatory role in humans, and that it could be considered as a target molecule for the therapy of immune-mediated diseases

  14. Following coculture with GTKO/CD46 pig mesenchymal stromal cells, it is possible that upregulation of CD69 on human T cells initiates signaling events that would regulate CD4+ and CD8+ T cell activation and differentiation.

  15. In patients with allergic rhinitis CD69 antigen is overexpressed on human peripheral blood natural killer cells reflecting their activation status.

  16. This is the first report of the regulation of CD69 expression by LMP-1, and this novel finding may, thus, represent an important link between the EBV oncoprotein LMP-1 and its critical role in the development of EBV-associated diseases.

  17. CD69 is induced by integrin alpha4beta1 outside-in signalling and T-cell receptor signalling.

  18. CD69 overexpression is associated with the human T-cell leukemia virus type 1 infection and adult T-cell leukemia.

  19. Intron I acts as an important regulatory element of CD69 expression.

  20. CD69 is significantly correlated with poor clinical and biological prognostic factors and is confirmed to be an independent disease prognosticator in chronic lymphocytic leukemia.

CD69 抗原简介


This gene encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. Expression of the encoded protein is induced upon activation of T lymphocytes, and may play a role in proliferation. Furthermore, the protein may act to transmit signals in natural killer cells and platelets.

Gene names and symbols associated with anti-CD69 (CD69) 抗体

  • Cd69 molecule (Cd69) 抗体
  • CD69 antigen (Cd69) 抗体
  • CD69 molecule (CD69) 抗体
  • 5830438K24Rik 抗体
  • AI452015 抗体
  • AIM 抗体
  • BL-AC/P26 抗体
  • CLEC2C 抗体
  • EA1 抗体
  • GP32/28 抗体
  • MLR-3 抗体
  • VEA 抗体

Protein level used designations for anti-CD69 (CD69) 抗体

CD69 antigen , early activation antigen CD69 , Very Early Activation Antigen , C-type lectin domain family 2, member C , CD69 antigen (p60, early T-cell activation antigen) , activation inducer molecule (AIM/CD69) , early T-cell activation antigen p60 , early lymphocyte activation antigen , leukocyte surface antigen Leu-23

29187 Rattus norvegicus
12515 Mus musculus
969 Homo sapiens
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