anti-MAPK14 (MAPK14) 抗体产品概述

Full name:
anti-Mitogen-Activated Protein Kinase 14 抗体 (MAPK14)
在www.antibodies-online.cn可供195 Mitogen-Activated Protein Kinase 14 (MAPK14) 抗体的25不同的供货商。 再加上,我们可以发MAPK14 试剂盒 (48)MAPK14 蛋白 (38)和数多这个蛋白质的别的产品。 总共312 MAPK14产品已列进来了。
别名:
186F5S, anon-sts23, AP22.98, AP22_98, ATMPK14, BG:DS00797.3, CG7393, CRK1, csbp, Csbp1, Csbp2, CSPB1, D-p38, D-p38 MAPK, D-p38b, Dmel\\CG7393, Dmp38b, Dm p38b, Dp38, dp38b, ESTS:186F5S, Exip, Hog, mapk14a, mitogen-activated protein kinase 14, Mpk34C, mxi2, p38, p38 beta, p38 MAPK, p38-alpha, p38a, p38alpha, p38B, p38beta, p38Hog, p38Kb, p38MAPK, Prkm14, Prkm15, RK, sapk2, sapk2a
列出全部抗体 基因 基因ID UniProt
MAPK14 1432 Q16539
MAPK14 26416 P47811
MAPK14 81649  

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  1. Human Polyclonal MAPK14 Primary Antibody for FACS, IF - ABIN1882176 : Cheung, Campbell, Nebreda, Cohen: Feedback control of the protein kinase TAK1 by SAPK2a/p38alpha. in The EMBO journal 2003 (PubMed)
    Show all 7 references for 1882176

  2. Human Polyclonal MAPK14 Primary Antibody for EIA, WB - ABIN359424 : Dean, Sarsfield, Tsounakou, Saklatvala: p38 Mitogen-activated protein kinase stabilizes mRNAs that contain cyclooxygenase-2 and tumor necrosis factor AU-rich elements by inhibiting deadenylation. in The Journal of biological chemistry 2003 (PubMed)
    Show all 5 references for 359424

  3. Dog (Canine) Monoclonal MAPK14 Primary Antibody for IF, WB - ABIN968769 : Brunet, Pouysségur: Identification of MAP kinase domains by redirecting stress signals into growth factor responses. in Science (New York, N.Y.) 1996 (PubMed)
    Show all 4 references for 968769

  4. Human Polyclonal MAPK14 Primary Antibody for IHC (p), WB - ABIN197012 : Zheng, Reynolds, Jo, Wersto, Han, Xiao: Intracellular acidosis-activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2005 (PubMed)
    Show all 2 references for 197012

  5. Human Polyclonal MAPK14 Primary Antibody for WB - ABIN197496 : van den Blink, Juffermans, ten Hove, Schultz, van Deventer, van der Poll, Peppelenbosch: p38 mitogen-activated protein kinase inhibition increases cytokine release by macrophages in vitro and during infection in vivo. in Journal of immunology (Baltimore, Md. : 1950) 2001 (PubMed)
    Show all 2 references for 197496

  6. Cow (Bovine) Polyclonal MAPK14 Primary Antibody for WB - ABIN2792184 : Choi, Kim, Kang, Bae, Cho, Soh, Kim, Kang, Chung, Lee, Lee: Activation of Bak and Bax through c-abl-protein kinase Cdelta-p38 MAPK signaling in response to ionizing radiation in human non-small cell lung cancer cells. in The Journal of biological chemistry 2006 (PubMed)

  7. Cow (Bovine) Polyclonal MAPK14 Primary Antibody for IHC, WB - ABIN2792183 : Alemán, Schierloh, de la Barrera, Musella, Saab, Baldini, Abbate, Sasiain: Mycobacterium tuberculosis triggers apoptosis in peripheral neutrophils involving toll-like receptor 2 and p38 mitogen protein kinase in tuberculosis patients. in Infection and immunity 2004 (PubMed)

  8. Human Polyclonal MAPK14 Primary Antibody for DB, WB - ABIN265093 : Choe, McGinty: N-Methyl-D-aspartate receptors and p38 mitogen-activated protein kinase are required for cAMP-dependent cyclase response element binding protein and Elk-1 phosphorylation in the striatum. in Neuroscience 2001 (PubMed)

  9. Human Polyclonal MAPK14 Primary Antibody for IHC (p), WB - ABIN197010 : Kim, Tannenbaum, White: Global phosphoproteome of HT-29 human colon adenocarcinoma cells. in Journal of proteome research 2005 (PubMed)

更多抗MAPK14的相互作用对抗体

Cow (Bovine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. results suggest that ET-1 (显示 EDN1 抗体)-induced activation of proMMP-2 is mediated via cross-talk between NADPH oxidase (显示 NOX1 抗体)-PKCalpha (显示 PKCa 抗体)-p(38)MAPK (显示 MAPK1 抗体) and NFkappaB-MT1MMP (显示 MMP14 抗体) signaling pathways along with a marked decrease in TIMP-2 (显示 TIMP2 抗体) expression in the cells

  2. cross-talk between p(38)MAPK (显示 MAPK1 抗体) and Gialpha play a pivotal role for full activation of cPLA2 (显示 PLA2G4A 抗体) during ET-1 (显示 EDN1 抗体) stimulation of pulmonary artery smooth muscle cells.

  3. MAPK14 signalling pathway is largely involved in heat-induced sperm damage.

  4. p38 MAPK is an early redox sensor in the laminar shear stress with hydrogen peroxide being a signaling mediator.

  5. Blockade of p38 enhances chondrocyte phenotype in monolayer culture and may promote more efficient cartilage tissue regeneration for cell-based therapies.

  6. p38 phosphorylation and MMP13 (显示 MMP13 抗体) expression are regulated by Rho/ROCK activation, and support the potential novel pathway that Rho/ROCK is in the upper part of the mechanical stress-induced matrix degeneration cascade in cartilage.

  7. These data suggest that the p38 and JNK (显示 MAPK8 抗体) signaling pathways play pivotal roles in PRRSV replication and may regulate immune responses during virus infection.

  8. findings support the hypothesis that ischemic factor stimulation of the blood-brain barrier Na-K-Cl cotransporter (显示 SLC12A1 抗体) involves activation of p38 and JNK (显示 MAPK8 抗体) MAPKs

  9. These data suggest a differential requirement of JNK1 (显示 MAPK8 抗体) and p38 MAPK in TNF (显示 TNF 抗体) regulation of E2F1 (显示 E2F1 抗体). Targeted inactivation of JNK1 (显示 MAPK8 抗体) at arterial injury sites may represent a potential therapeutic intervention for ameliorating TNF (显示 TNF 抗体)-mediated EC dysfunction.

  10. p38 MAPK (MAPK14) is redox-regulated in reactive oxygen species-dependent endothelial barrier dysfunction.

Fruit Fly (Drosophila melanogaster) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. ROS (显示 ROS1 抗体)/JNK (显示 MAPK8 抗体)/p38/Upd (显示 UROD 抗体) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  2. Taken together, our findings indicate that the p38 MAP Kinase is an integral component of the core circadian clock of Drosophila in addition to playing a role in stress-input pathways.

  3. Data show that the genetic interaction between p38b MAPK (显示 MAPK1 抗体) and Rack1 (显示 GNB2L1 抗体) controls muscle aggregate formation, locomotor function, and longevity.

  4. The interaction of any of several Drosophila Delta class glutathione transferases and p38b mitogen-activated protein kinase (显示 MAPK1 抗体) can affect the substrate specificity of either enzyme, which suggests induced conformational changes affecting catalysis.

  5. found a correlation between the depth of integration of individual p38 kinases into the protein interaction network and their functional significance; propose a central role of p38b in the p38 signaling module with p38a and p38c playing more peripheral auxiliary roles

  6. Loss of p38 MAPK causes early lethality and precipitates age-related motor dysfunction and stress sensitivity.

  7. The p38 pathway-mediated stress response contribute to Drosophila host defense against microbial infection.

  8. p38b MAPK (显示 MAPK1 抗体) plays a crucial role in the balance between intestinal stem cell proliferation and proper differentiation in the adult Drosophila midgut.

  9. the D-p38b gene is regulated by the DREF (显示 ZBED1 抗体) pathway and DREF (显示 ZBED1 抗体) is involved in the regulation of proliferation and differentiation of Drosophila ISCs (显示 NFS1 抗体) and progenitors

Horse (Equine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. p38 mitogen-activated protein kinase is crucial for bovine papillomavirus type-1 transformation of equine fibroblasts.

  2. p38 Mitogen-activated protein kinase (MAPK (显示 MAPK1 抗体)) is essential for drug-induced COX-2 (显示 PTGS2 抗体) expression in leukocytes, suggesting that p38 MAPK is a potential target for anti-inflammatory therapy.

  3. These findings support a function for p38 MAPK in equine neutrophil migration and suggest the potential for the ability of p38 MAPK inhibition to limit neutrophilic inflammation in the laminae during acute laminitis.

  4. Cultured equine digital vein endothelial cells were exposed to lipopolysaccharide and phosphorylation of p38 MAPK was assessed by Western blotting using phospho-specific antibodies.

Human Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. findings show that endothelial MAPKs ERK (显示 EPHB2 抗体), p38 (显示 CRK 抗体), and JNK (显示 MAPK8 抗体) mediate diapedesis-related and diapedesis-unrelated functions of ICAM-1 (显示 ICAM1 抗体) in cerebral and dermal microvascular endothelial cells

  2. Tetraarsenic hexoxide (As4O6) induced G2/M arrest, apoptosis and autophagic cell death through PI3K (显示 PIK3CA 抗体)/Akt (显示 AKT1 抗体) and p38 MAPK pathways alteration in SW620 colon cancer cells.

  3. The N-Terminal phosphorylation of RB by p38 (显示 CRK 抗体) bypasses its inactivation by cyclin (显示 PCNA 抗体)-dependent kinases and prevents proliferation in cancer cells.

  4. Inhibition of MAPK14 conclusively facilitates elucidation of the impact of the complex network of p38 MAPK signaling on atherogenesis.

  5. Collectively, this study provides more insights into RELT (显示 RELT 抗体) expression, RELT (显示 RELT 抗体) family member function, and the mechanism of RELT (显示 RELT 抗体)-induced death.

  6. Data, including data from studies conducted in cells from transgenic/knockout mice, suggest that p38alpha MAPK (显示 MAPK1 抗体) (MAPK14) activity is required for hypoxia-induced pro-angiogenic activity involving cardiomyocytes and vascular endothelial cells; p38 MAPK activation in cardiomyocyte is sufficient to promote paracrine signaling-mediated, pro-angiogenic activity/myocardial revascularization.

  7. The findings indicate that p38alpha and GADD45alpha (显示 GADD45A 抗体) are involved in an enhanced vitamin D signaling on TRPV6 (显示 TRPV6 抗体) expression.

  8. These results suggest that the p38 (显示 CRK 抗体)/NPM (显示 NPM1 抗体)/PP2A (显示 PPP2R4 抗体) complex acts as a dynamic sensor, allowing endothelial cells to react rapidly to acute oxidative stress.

  9. Inhibition of the inflammatory signaling intermediate p38 MAPK reduced tissue factor (显示 F3 抗体) (TF) mRNA by one third but increased tumor necrosis factor (TNF (显示 TNF 抗体)) and interleukin-1 beta (IL-1beta (显示 IL1B 抗体)) mRNA.

  10. These results suggest that the activation of endogenous levels of SFK renders the endothelial barrier more susceptible to low, physiologic doses of TNF-alpha (显示 TNF 抗体) through activation of p38 (显示 CRK 抗体) which leads to a loss of endothelial tight junctions.

Mouse (Murine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. Soluble epoxide hydrolase (显示 EPHX2 抗体) inhibitor AUDA decreases bleomycin-induced pulmonary toxicity in mice by inhibiting the p38 (显示 CRK 抗体)/Smad3 (显示 SMAD3 抗体) signaling pathway.

  2. These results provide evidence supporting a key role for the p38 MAPK signaling pathway which is involved in the regulation of Abeta1-42 internalization in the parietal cortex and hippocampus of mouse through LRP1 (显示 LRP1 抗体) in vivo.

  3. The N-Terminal phosphorylation of RB by p38 (显示 CRK 抗体) bypasses its inactivation by cyclin (显示 PCNA 抗体)-dependent kinases and prevents proliferation in cancer cells.

  4. Quantitative real-time polymerase chain reaction (qRT-PCR) and Western blotting were used to detect the mRNA and protein expressions of p-p38MAPK, AAT (显示 SERPINA1A 抗体), signal transducer and activator of transcription 1 (STAT1 (显示 STAT1 抗体)) and activating transcription factor2 (ATF2 (显示 ATF2 抗体))

  5. p38alpha is essential to maintain in actin dynamics with age in hepatocytes.

  6. Data, including data from studies conducted in cells from transgenic/knockout mice, suggest that p38alpha MAPK (显示 MAPK1 抗体) (Mapk14) activity is required for hypoxia-induced pro-angiogenic activity involving cardiomyocytes and vascular endothelial cells; p38 MAPK activation in cardiomyocyte is sufficient to promote paracrine signaling-mediated, pro-angiogenic activity/myocardial revascularization.

  7. blockage of NF-kappaB (显示 NFKB1 抗体) p65 (显示 NFkBP65 抗体) and/or MAPK p38 (显示 MAPK1 抗体) with their specific inhibitors strongly attenuated B7-H3 (显示 CD276 抗体)-amplified inflammatory response with significantly reduced proinflammatory cytokine and chemokine (显示 CCL1 抗体) production, and markedly ameliorated B7-H3 (显示 CD276 抗体)-exacerbated disruption of blood-brain barrier and severity of disease status in S. pneumoniae-infected mice.

  8. Data suggest that single muscle immobilization induces a shift of myosin heavy chain (MHC) isoforms composition toward a faster contractile phenotype and decreases the polymorphic profile of single fibres, and that activation of p38 and JNK could be a potential mechanism involved in these contractile phenotype modifications during muscle immobilization.

  9. The kinase TPL2 (显示 MAP3K8 抗体) activates ERK (显示 EPHB2 抗体) and p38 (显示 CRK 抗体) signaling to promote neutrophilic inflammation

  10. studies have therefore uncovered a p38alpha-mediated pathway that alters Hematopoietic Stem/Progenitor Cell metabolism to respond to stress and promote recovery.

Rabbit Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. These findings suggest that the TQ-induced production of ROS (显示 ROS1 抗体) causes dedifferentiation through the ERK (显示 MAPK1 抗体) pathway and inflammation through the PI3K and p38 pathways in rabbit articular chondrocytes.

  2. These results suggest that p38 MAPK signal transduction pathway is critical to NO-induced chondrocyte apoptosis, and p38 plays a role by way of stimulating NF-kappaB (显示 NFKB1 抗体), p53 (显示 TP53 抗体) and caspase-3 (显示 CASP3 抗体) activation.

Pig (Porcine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. Porcine reproductive and respiratory syndrome virus strain CH-1a could significantly up-regulate IL-10 (显示 IL10 抗体) production through p38 MAPK activation.

  2. JNK (显示 MAPK8 抗体) plays an active role in fragmentation of pig oocytes and p38 MAPK is not involved in this process.[p38MAPK]

  3. Retinal ischemia-reperfusion alters expression of mitogen-activated protein kinases, particularly ERK1/2 (显示 MAPK1/3 抗体), in the neuroretina and retinal arteries.

Xenopus laevis Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. cytochrome c (显示 CYCS 抗体) microinjection induces p38 phosphorylation through caspase-3 (显示 CASP3 抗体) activation, and caspase (显示 CASP3 抗体) inhibition reduces p38 activation induced by osmostress, indicating that a positive feedback loop is engaged by hyperosmotic shock

MAPK14 抗原简介

Antigen Summary

The protein encoded by this gene is a member of the MAP kinase family. MAP kinases act as an integration point for multiple biochemical signals, and are involved in a wide variety of cellular processes such as proliferation, differentiation, transcription regulation and development. This kinase is activated by various environmental stresses and proinflammatory cytokines. The activation requires its phosphorylation by MAP kinase kinases (MKKs), or its autophosphorylation triggered by the interaction of MAP3K7IP1/TAB1 protein with this kinase. The substrates of this kinase include transcription regulator ATF2, MEF2C, and MAX, cell cycle regulator CDC25B, and tumor suppressor p53, which suggest the roles of this kinase in stress related transcription and cell cycle regulation, as well as in genotoxic stress response. Four alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported.

Alternative names and synonyms associated with MAPK14

  • mitogen-activated protein kinase 14 (MPK14) 抗体
  • mitogen-activated protein kinase 14 (MAPK14) 抗体
  • mitogen-activated protein kinase 14 (Mapk14) 抗体
  • CG7393 gene product from transcript CG7393-RA (p38b) 抗体
  • mitogen activated protein kinase 14 (Mapk14) 抗体
  • mitogen-activated protein kinase 14 (mapk14) 抗体
  • 186F5S 抗体
  • anon-sts23 抗体
  • AP22.98 抗体
  • AP22_98 抗体
  • ATMPK14 抗体
  • BG:DS00797.3 抗体
  • CG7393 抗体
  • CRK1 抗体
  • csbp 抗体
  • Csbp1 抗体
  • Csbp2 抗体
  • CSPB1 抗体
  • D-p38 抗体
  • D-p38 MAPK 抗体
  • D-p38b 抗体
  • Dmel\\CG7393 抗体
  • Dmp38b 抗体
  • Dm p38b 抗体
  • Dp38 抗体
  • dp38b 抗体
  • ESTS:186F5S 抗体
  • Exip 抗体
  • Hog 抗体
  • mapk14a 抗体
  • mitogen-activated protein kinase 14 抗体
  • Mpk34C 抗体
  • mxi2 抗体
  • p38 抗体
  • p38 beta 抗体
  • p38 MAPK 抗体
  • p38-alpha 抗体
  • p38a 抗体
  • p38alpha 抗体
  • p38B 抗体
  • p38beta 抗体
  • p38Hog 抗体
  • p38Kb 抗体
  • p38MAPK 抗体
  • Prkm14 抗体
  • Prkm15 抗体
  • RK 抗体
  • sapk2 抗体
  • sapk2a 抗体

Protein level used designations for MAPK14

MAP kinase 14 , MAP kinase p38 alpha , MAPK 14 , mitogen-activated protein kinase p38 alpha , p38 mitogen activated protein kinase , CG7393-PA , p38 mitogen-activated protein kinase , p38b-PA , stress-activated p38b MAP kinase , p38 mitogen-activated kinase , cytokine suppressive anti-inflammatory drug binding protein 1 , mitogen activated protein kinase 14 , p38 MAP kinase alpha , p38 MAPK , p38 alpha , tRNA synthetase cofactor p38 , CSAIDS-binding protein 1 , mitogen-activated protein kinase 14A , stress-activated protein kinase 2a , Csaids binding protein , MAP kinase 2 , MAP kinase Mxi2 , MAX-interacting protein 2 , cytokine suppressive anti-inflammatory drug binding protein , cytokine-supressive anti-inflammatory drug binding protein , mitogen-activated protein kinase 14 , p38 MAP kinase , p38alpha Exip , reactive kinase , stress-activated protein kinase 2A , MAPK p38 , MPK2 , Mitogen-activated protein kinase 2 , mitogen-activated Mitogen-activated protein kinase 2

GENE ID SPECIES
829797 Arabidopsis thaliana
534492 Bos taurus
403856 Canis lupus familiaris
100723285 Cavia porcellus
34780 Drosophila melanogaster
100063532 Equus caballus
421183 Gallus gallus
1432 Homo sapiens
26416 Mus musculus
100341695 Oryctolagus cuniculus
450161 Pan troglodytes
81649 Rattus norvegicus
100156630 Sus scrofa
379992 Xenopus laevis
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