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Found that the calcium-modulating cyclophilin ligand (CAMLG), which is involved in Ca(2 (显示 CA2 抗体)+) signaling, was the major host cell PilA binding protein.
A novel interaction between calcium-modulating cyclophilin ligand and Basigin regulates calcium signaling and matrix metalloproteinase activities in human melanoma cells.
Results indicate calcium-modulating cyclophilin ligand (CAML) and WRB (显示 WRB 抗体) as components of the TRC40 (显示 ASNA1 抗体) receptor complex and a crucial mechanism for driving ER membrane insertion of TA proteins in mammalian cells.
CAML was found to play a crucial role in the PRL (显示 PRL 抗体)/PRLR (显示 PRLR 抗体)-dependent growth of breast cancer cells.
this study demonstrated that DENV manipulated the levels of CAML to subvert the apoptotic process which in turn favoured efficient virus production.
Data show that tetherin (显示 BST2 抗体) restricts particle release and does not require CAML for this effect.
CAMLG was more frequently significantly overexpressed in acute myeloid leukemia (显示 BCL11A 抗体) patients with CD56 (显示 NCAM1 抗体) immunophenoytpe
Kaposi's sarcoma-associated herpesvirus mitochondrial K7 protein targets cellular CAML to increase the cytosolic Ca(2 (显示 CA2 抗体)+) response, which consequently protects cells from mitochondrial damage and apoptosis
CAML is an important signal transducer for the actions of Ang II (显示 AGT 抗体) in regulating the calcineurin-NFAT (显示 NFATC1 抗体) pathway and the interaction of CAML with ATRAP (显示 AGTRAP 抗体) may mediate the Ang II (显示 AGT 抗体) actions in vascular physiology
The intracellular C-terminus of fibrocystin (显示 PKHD1 抗体) interacts with CAML, a protein with an intracellular distribution that is similar to that of PKD2 (显示 PKD2 抗体).
this study identified a novel role for B cell-specific, BAFF (显示 TNFSF13B 抗体)-dependent transmembrane activator and CAML interactor (显示 TNFRSF13B 抗体) signals in atherosclerosis pathogenesis
Deletion of calcium-modulating cyclophilin ligand causes deafness in mouse model.
Conditional deletion of CAML in CD19 (显示 CD19 抗体)-Cre transgenic mice reduced follicular B cell numbers & increased rates of homeostatic proliferation. They had increased cellular turnover & normal proliferative ability.
Loss of CAMLG protein failed to undergo normal T-cell development and exhibited dramatically increased rates of apoptosis.
results implicate Tmub1 (显示 TMUB1 抗体) in the regulation of locomotor activity and wakefulness and suggest that Tmub1 (显示 TMUB1 抗体) binds to and functions together with CAMLG
These data implicate calcium-modulating cyclophilin ligand in epidermal growth factor (EGF (显示 EGF 抗体)) receptor (显示 EGFR 抗体) signaling and suggest that it may play a role in receptor recycling during long-term proliferative responses to EGF (显示 EGF 抗体).
CAML as an essential mediator of T cell survival during thymopoiesis and its loss deregulates lymphocyte specific protein tyrosine kinase (显示 LCK 抗体) p56 (显示 IFIT1 抗体)(p56Lck (显示 LCK 抗体)) signaling.
specific role of CAML in functional expression and endocytic recycling of postsynaptic GABA(A) receptors
our data identify CAML as a novel chromosomal instability gene and suggest that CAML protein acts as a key regulator of mitotic spindle function and a modulator of SAC (显示 ADCY10 抗体) maintenance.
The immunosuppressant drug cyclosporin A blocks a calcium-dependent signal from the T-cell receptor (TCR) that normally leads to T-cell activation. When bound to cyclophilin B, cyclosporin A binds and inactivates the key signaling intermediate calcineurin. The protein encoded by this gene functions similarly to cyclosporin A, binding to cyclophilin B and acting downstream of the TCR and upstream of calcineurin by causing an influx of calcium. This integral membrane protein appears to be a new participant in the calcium signal transduction pathway, implicating cyclophilin B in calcium signaling, even in the absence of cyclosporin.
calcium modulating ligand
, calcium signal-modulating cyclophilin ligand
, calcium modulating cyclophilin ligand CAML
, calcium-modulating cyclophilin ligand
, calcium-signal modulating cyclophilin ligand
, cyclophilin B-binding protein