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抗Rat (Rattus) 抗体:
Human Polyclonal GCP2 Primary Antibody for WB - ABIN520075
Vaithilingam, Quayum, Joglekar, Jensen, Hardikar, Oberholzer, Guillemin, Tuch: Effect of alginate encapsulation on the cellular transcriptome of human islets. in Biomaterials 2011
These data identify suppression of CXCL2 (显示 CXCL2 抗体) and CXCL5 (显示 CXCL5 抗体) chemoattractant expression by 11beta-HSD1 (显示 HSD11B1 抗体) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (显示 IL17RA 抗体) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (显示 STAT3 抗体) is required for maximal OSM (显示 OSM 抗体)-induced CXCL5 (显示 CXCL5 抗体) expression.
CXCL5 (显示 CXCL5 抗体) has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 (显示 CXCL5 抗体) expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (显示 NXF1 抗体).
CXCL5 (显示 CXCL5 抗体) regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (显示 CXCL1 抗体) and CXCL5 (显示 CXCL5 抗体) are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2 (显示 TLR2 抗体)-induced epithelial-derived CXCL5 (显示 CXCL5 抗体) is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
CXCL5 (显示 CXCL5 抗体) modulated macrophage activation, increased expression of the cholesterol efflux regulatory protein ABCA1 (显示 ABCA1 抗体), and enhanced cholesterol efflux activity in macrophages.
CXCL6 level is high in the serum of chronic hepatitis B patients.CXCL6 promotes human hepatocyte proliferation through the CXCR1-NFkappaB pathway and inhibits collagen I secretion by hepatic stellate cells.
The role of PITX2 (显示 PITX2 抗体) in glaucoma may be mediated partly by regulating the expression of CXCL6 and BBS5 (显示 BBS5 抗体) and thus affecting immune functions and intraocular pressure.
This is the first study to note that elevated systemic CCL5 (显示 CCL5 抗体) and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
The neutrophil-recruiting chemokine (显示 CCL1 抗体) GCP-2/CXCL6 is expressed in cystic fibrosis (显示 S100A8 抗体) airways and retains its functional properties after binding to extracellular DNA.
HIF-1alpha (显示 HIF1A 抗体) promotes HCC (显示 FAM126A 抗体) progression and metastasis by upregulating CXCL6 transcription in HCC (显示 FAM126A 抗体) cells.
Expression of the CXCL8 (显示 IL8 抗体), CXCL6 and CXCL1 (显示 CXCL1 抗体) genes are under the primary control of 1,25-dihydroxyvitamin D3 and its receptor.
Data suggest that expression of CXCL6 in trophoblasts is up-regulated during pregnancy development/placentation; CXCL6 expression inhibits trophoblast cell migration and invasion by down-regulating activity of MMP2 (matrix metalloproteinase 2 (显示 MMP2 抗体)).
Overexpression of GCP-2 (显示 FOLH1 抗体) in mesenchymal stem cells has the potential to enhance their angiogenic and survival properties.
Studies show that functional blocking of GCP-2 (显示 FOLH1 抗体) inhibits tumor growth and metastases.
The aim of this study was to investigate the role of the transcription factors, AP-1 (显示 FOSB 抗体) and NF-kappaB (显示 NFKB1 抗体), in IL-6 (显示 IL6 抗体) and CXCL8 (显示 IL8 抗体) regulation in Jurkat T-cells.
mouse homolog is a member of the CXC chemokine family, is a neutrophil chemoattractant and is rapidly induced in response to muscle injury
C-X-C motif chemokine 5
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15
, C-X-C motif chemokine 6
, Small inducible cytokine subfamily B (Cys-X-Cys), member b
, chemokine alpha 3
, granulocyte chemotactic protein 2
, small inducible cytokine subfamily B (Cys-X-Cys), member 6 (granulocyte chemotactic protein 2)
, small-inducible cytokine B6
, chemokine (C-X-C motif) ligand 5
, inducible cytokine subfamily B (Cys-X-Cys), member 6