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抗Mouse (Murine) beta 2 Adrenergic Receptor 抗体:
抗Human beta 2 Adrenergic Receptor 抗体:
抗Rat (Rattus) beta 2 Adrenergic Receptor 抗体:
Human Monoclonal beta 2 Adrenergic Receptor Primary Antibody for Func, IA - ABIN2192080
Lebesgue, Wallukat, Mijares, Granier, Argibay, Hoebeke: An agonist-like monoclonal antibody against the human beta2-adrenoceptor. in European journal of pharmacology 1998
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Human Polyclonal beta 2 Adrenergic Receptor Primary Antibody for FACS, WB - ABIN513155
Lencesova, Sirova, Csaderova, Laukova, Sulova, Kvetnansky, Krizanova: Changes and role of adrenoceptors in PC12 cells after phenylephrine administration and apoptosis induction. in Neurochemistry international 2010
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Human Polyclonal beta 2 Adrenergic Receptor Primary Antibody for FACS, IF (p) - ABIN730153
Fan, Liao, Tang, Chen, Zhang, Liu, Zhong: Role of ?2-adrenoceptor-?-arrestin2-nuclear factor-?B signal transduction pathway and intervention effects of oxymatrine in ulcerative colitis. in Chinese journal of integrative medicine 2012
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Human Polyclonal beta 2 Adrenergic Receptor Primary Antibody for ELISA, WB - ABIN185478
Elefteriou, Ahn, Takeda, Starbuck, Yang, Liu, Kondo, Richards, Bannon, Noda, Clement, Vaisse, Karsenty: Leptin regulation of bone resorption by the sympathetic nervous system and CART. in Nature 2005
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Human Polyclonal beta 2 Adrenergic Receptor Primary Antibody for WB - ABIN2792089
Penco, Buscema, Patrosso, Marocchi, Grossi: New application of intelligent agents in sporadic amyotrophic lateral sclerosis identifies unexpected specific genetic background. in BMC bioinformatics 2008
Zebrafish larvae lacking beta1AR (显示 ADRB1 抗体) expression by morpholino knockdown displayed lower heart rates than control fish, whereas larvae deficient in both beta2aAR and beta2bAR expression exhibited significantly higher heart rates than controls.
S1928A KI mice failed to induce long-term potentiation in response to prolonged theta-tetanus (PTT-LTP (显示 SCP2 抗体)), a form of synaptic plasticity that requires Cav1.2 (显示 CACNA1C 抗体) and enhancement of its activity by the beta2-adrenergic receptor (beta2AR)-cAMP-PKA cascade.
activation of beta2AR decreased cerebral amyloid plaques through the up-regulation of alpha-secretase activity and by decreasing the phosphorylation of APP (显示 APP 抗体) at Thr668
Together, these data indicate that high-fat diet promotes phosphorylation of the beta2 AR, contributing to impairment of cardiac contractile reserve before cardiac structural and functional remodelling, suggesting that early intervention in the insulin (显示 INS 抗体)-adrenergic signalling network might be effective in prevention of cardiac complications in diabetes
beta2-AR deletion is associated to selective hepatic insulin (显示 INS 抗体) resistance and preserved skeletal muscle insulin (显示 INS 抗体) sensitivity.
The diurnal variation of the humoral immune response was dependent on beta2AR-mediated neural signals and was diminished when lymphocyte recirculation through lymph nodes was stopped.
knocking out of the beta1/2 (显示 TFAP2B 抗体) receptor significantly diminished the ST25 acupuncture-induced inhibition of gastric motility and jejunal motility without significantly altering the enhancement of colonic motility induced by acupuncture at ST25.
The authors show that phosphorylation of S1928 displaces the beta-2 adrenergic receptor from Cav1.2 (显示 CACNA1C 抗体) upon beta-adrenergic stimulation rendering Cav1.2 (显示 CACNA1C 抗体) refractory for several minutes from further beta-adrenergic stimulation.
The Galphas (显示 GNAS 抗体) and Galphaq (显示 GNAQ 抗体) peptides adopt different orientations in beta2-AR and V1AR (显示 AVPR1A 抗体), respectively. The beta2-AR/Galphas (显示 GNAS 抗体) peptide interface is dominated by electrostatic interactions, whereas the V1AR (显示 AVPR1A 抗体)/Galphaq (显示 GNAQ 抗体) peptide interactions are predominantly hydrophobic.
Thus the beta2AR acts as a double-edged sword: increasing TAS2R14 cell surface expression, but when activated by beta-agonist, partially offsetting the expression phenotype by direct receptor:receptor desensitization of TAS2R14 function.
Data show that inhibitory (Galphai2 (显示 GNAI2 抗体)) and stimulatory (GalphasL) G-protein subunits produced minor atrophic and hypertrophic changes in muscle mass, and Galphai2 (显示 GNAI2 抗体) over-expression prevented AAV:beta2-adrenoceptor (beta2-AR) mediated hypertrophy.
These results indicate dependence of hepatic glucocorticoid and adrenergic receptors on stage of maturation in neonatal calves and emphasize the association of alpha1-adrenergic receptor and glucocorticoid receptor (显示 NR3C1 抗体) with neonatal glucose and lipid metabolism.
ADRb2 shows lipid scrambling activity.
relationship between luteal concentrations of oxytocin (OT), noradrenaline (NA), progesterone (P4), oxytocin receptors (OT-R (显示 OXTR 抗体)) and beta(2)-adrenoreceptors (beta(2)-R) gene expression and their protein level throughout the estrous cycle
a substantial proportion of the beta2AR produced is non-functional and VCP (显示 vcp 抗体) plays a key role in the maturation and trafficking of the beta2AR.
The protein kinase (显示 CDK7 抗体) activity of PI3K (显示 PIK3CA 抗体) phosphorylates serine residue 70 on Src (显示 SRC 抗体) to enhance its activity and induce EGFR (显示 EGFR 抗体) transactivation following betaAR stimulation.
The data suggested that Arg/Arg homozygosity at codon 16 of the ADRB2 gene predisposes patients to a clinically more severe course of chronic obstructive pulmonary disease
using single-molecule fluorescence resonance energy transfer imaging, we examine TM6 movements in the beta2 adrenergic receptor (beta2AR) upon exposure to orthosteric ligands with different efficacies, in the absence and presence of the Gs heterotrimer
The frequency of the genotypes and alleles of rs1042711 in ADRB2 showed a significant difference between the COPD (显示 ARCN1 抗体) and control groups. TT genotype and TG and TC haplotypes of rs1042711 in ADRB2 are related to pulmonary function in COPD (显示 ARCN1 抗体) patients. TT genotype of rs1042711 in ADRB2 and smoking amount are risk factors for COPD (显示 ARCN1 抗体) development.
Results provide further evidence for the interaction of rs2400707, and other SNPs within the ADRB2 gene, with childhood trauma in relation to risk for posttraumatic stress disorder.
Gln27Glu polymorphism of ADRB2 influences exercise-induced vascular adaptation in patients with acute coronary syndrome.
Data suggest that ADRB2 (beta2 adrenergic receptor) activation (as illustrated by epinephrine and nor epinephrine) leads to robust calcium ion mobilization from intracellular stores in endoplasmic reticulum via activation of phosphoinositide phospholipase C (PLC) and opening of inositol trisphosphate receptor (IP3R).
Data suggest that post-translational modifications (phosphorylation, oxidation, and nitrosylation) of RyR2 (ryanodine receptor 2 (显示 RYR2 抗体)) occur downstream of production of amyloid beta-peptides through ADRB2 (beta2-adrenergic receptor) Ca2 (显示 CA2 抗体)+ signaling cascade that activates PKA (protein kinase A).
ADRB2 SNPs might be a genetic risk factor for dyslipidemia in the Chinese hypertensive patients. the A46G polymorphism was significantly associated with the elevated risk of hypertriglyceridemia. Haplotype analysis showed that the TAC (显示 IL2RA 抗体) haplotype carrying frequent alleles of the three SNPs played a reduced role in hypertriglyceridemia risk and the TGC (显示 TGM2 抗体) haplotype carrying rare allele of A46G expressed a significant risk effe
A persistent B2R (显示 BDKRB2 抗体)-beta2AR heterodimer was confirmed in bradykinin-stimulated and non-stimulated left ventricular myocardium. BK transactivation of beta2AR enhanced beta2AR-mediated release of tPA (显示 PLAT 抗体).
Prolactin (显示 PRL 抗体) infusion brought about the inhibition of a vasodilatory beta(2)-adrenergic receptor-mediated effect related to the NO intracellular pathway
Intracoronary intermedin (显示 ADM2 抗体) 1-47 augments cardiac perfusion and function in anesthetized pigs: role of calcitonin (显示 CALCA 抗体) receptors and beta-adrenoreceptor-mediated nitric oxide release.
The results imply ADRB-2-mediated actions in the development of primate follicles
This gene encodes beta-2-adrenergic receptor which is a member of the G protein-coupled receptor superfamily. This receptor is directly associated with one of its ultimate effectors, the class C L-type calcium channel Ca(V)1.2. This receptor-channel complex also contains a G protein, an adenylyl cyclase, cAMP-dependent kinase, and the counterbalancing phosphatase, PP2A. The assembly of the signaling complex provides a mechanism that ensures specific and rapid signaling by this G protein-coupled receptor. This gene is intronless. Different polymorphic forms, point mutations, and/or downregulation of this gene are associated with nocturnal asthma, obesity and type 2 diabetes.
adrenergic receptor beta-2
, adrenergic, beta-2-, receptor, surface
, beta-2 adrenergic receptor
, beta-2b-adrenergic receptor
, beta-2 adrenoceptor
, beta-2 adrenoreceptor
, beta 2-AR
, adrenergic receptor beta 2
, adrenergic, beta-2, receptor, surface
, catecholamine receptor
, beta2-adrenergic receptor
, Adrenergic beta 2- receptor surface
, adrenergic receptor, beta 2
, Beta-2 adrenergic receptor
, Beta-2 adrenoceptor
, Beta-2 adrenoreceptor
, beta 2 adrenergic receptor
, beta-2-adrenergic receptor