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TUSC2 is a highly conserved lung cancer candidate gene. 再加上，我们可以发Tumor Suppressor Candidate 2 蛋白 (3) 和 Tumor Suppressor Candidate 2 试剂盒 (1)和数多这个蛋白质的别的产品。
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Human Polyclonal TUSC2 Primary Antibody for ELISA, WB - ABIN1533546
Kondo, Ji, Kamibayashi, Tomizawa, Randle, Sekido, Yokota, Kashuba, Zabarovsky, Kuzmin, Lerman, Roth, Minna: Overexpression of candidate tumor suppressor gene FUS1 isolated from the 3p21.3 homozygous deletion region leads to G1 arrest and growth inhibition of lung cancer cells. in Oncogene 2001
Human Polyclonal TUSC2 Primary Antibody for ELISA - ABIN451824
Deng, Kawashima, Wu, Jayachandran, Xu, Minna, Roth, Ji et al.: Synergistic tumor suppression by coexpression of FUS1 and p53 is associated with down-regulation of murine double minute-2 and activation of the apoptotic protease-activating factor 1-dependent ... in Cancer research 2007
FOXP1 (显示 FOXP1 抗体), TP53INP1 (显示 TP53INP1 抗体), TNFAIP3 (显示 TNFAIP3 抗体), and TUSC2 were identified as miR (显示 MLXIP 抗体)-19a targets.
The therapeutic activity of TUSC2 could extend the use of erlotinib to lung cancer patients with wildtype EGFR (显示 EGFR 抗体).
TUSC2P and TUSC2 3'-UTR (显示 UTS2R 抗体) expression inhibits cell proliferation, survival, migration, invasion and colony formation, and increases tumor cell death. TUSC2P and TUSC2 3'-UTR (显示 UTS2R 抗体) bind to miRNAs and arrest their functions, leading to increased TUSC2 translation.
The tumor suppressor gene TUSC2 (FUS1) sensitizes NSCLC to the AKT (显示 AKT1 抗体) inhibitor MK2206 in LKB1 (显示 STK11 抗体)-dependent manner.
RNA sequence elements in FUS1 (显示 FUS 抗体) UTRs that regulate FUS1 (显示 FUS 抗体) protein express were identified.
Absence of tumor suppressor FUS1 (显示 FUS 抗体) protein expression is associated with bone and soft tissue sarcomas.
Data show that the recombinant FUS1 (显示 FUS 抗体) plasmid has been successfully cloned, which allows highly efficient FUS1 (显示 FUS 抗体) expression in E.coli strain Rosetta (DE3)2 plys.
The goal of this study was to analyze possible involvement of TUSC2 in malignant pleural mesothelioma.
Myristoylation is required for Fus1 (显示 FUS 抗体)-mediated tumor-suppressing activity and suggest a novel mechanism for the inactivation of tumor suppressors in lung cancer.
These results suggest that miR (显示 MLXIP 抗体)-378 enhances cell survival, tumor growth, and angiogenesis through repression of the expression of two tumor suppressors, Sufu (显示 SUFUH 抗体) and Fus-1 (显示 FUS 抗体).
Fus1 achieves its protective role in inflammation, autoimmunity, and cancer via the regulation of mitochondrial calcium and calcium-coupled parameters.
This gene is a highly conserved lung cancer candidate gene. No other information about this gene is currently available.
tumor suppressor candidate 2
, PDGFA associated protein 2
, PDGFA-associated protein 2
, fus-1 protein
, fusion 1 protein
, lung cancer candidiate, FUS