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MONDOA forms heterodimers with MLX (MIM 602976) that can bind to and activate transcription from CACGTG E boxes (Billin et al., 2000. 再加上，我们可以发MLX Interacting Protein 蛋白 (7) 和 MLX Interacting Protein 试剂盒 (2)和数多这个蛋白质的别的产品。
Showing 10 out of 31 products:
Human Polyclonal MLXIP Primary Antibody for EIA, IHC (p) - ABIN953462
Peterson, Stoltzman, Sighinolfi, Han, Ayer: Glucose controls nuclear accumulation, promoter binding, and transcriptional activity of the MondoA-Mlx heterodimer. in Molecular and cellular biology 2010
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Cow (Bovine) Polyclonal MLXIP Primary Antibody for WB - ABIN610684
Das, Lewis, Scherer, Lisanti: The membrane-spanning domains of caveolins-1 and -2 mediate the formation of caveolin hetero-oligomers. Implications for the assembly of caveolae membranes in vivo. in The Journal of biological chemistry 1999
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Dog (Canine) Polyclonal MLXIP Primary Antibody for IHC, WB - ABIN2776519
Stoltzman, Peterson, Breen, Muoio, Billin, Ayer: Glucose sensing by MondoA:Mlx complexes: a role for hexokinases and direct regulation of thioredoxin-interacting protein expression. in Proceedings of the National Academy of Sciences of the United States of America 2008
MondoA-directed programs have a key role in the coordinated control of myocyte lipid balance and insulin (显示 INS 抗体) signaling
Evaluation of the conservation of ChREBP (显示 MLXIPL 抗体) and MondoA sequences demonstrate that MondoA is better conserved and potentially mediates more ancient function in glucose metabolism.
These results suggest that C771G polymorphism of MLXIPL (显示 MLXIPL 抗体) gene is associated with coronary stenosis and its severity.
Knockdown of MondoA, or its dimerization partner Mlx (显示 MLX 抗体), blocks Myc (显示 MYC 抗体)-induced reprogramming of multiple metabolic pathways, resulting in apoptosis
regulatory relationship between mTOR (显示 FRAP1 抗体) and the MondoA-TXNIP (显示 TXNIP 抗体) axis that we propose contributes to glucose homeostasis
Suppression of Txnip (显示 TXNIP 抗体) by lipopolysaccharide is accompanied by a decrease of the glucose sensing transcription factor MondoA in the nuclei.
An important contribution of MondoA to leukemia aggressiveness, which makes MondoA a potential candidate for targeted treatment of acute lymphoblastic leukemia.
the MondoA-TXNIP (显示 TXNIP 抗体) regulatory circuit has a role in the hexose transport curb, although other redundant pathways also contribute
Induction of TXNIP (显示 TXNIP 抗体) under lactic acidosis is caused by the activation of the glucose-sensing helix-loop-helix transcriptional complex MondoA:Mlx, which is usually triggered upon glucose exposure.
Glucose is required at two additional steps to stimulate the transcription activation function of MondoA-Mlx (显示 MLX 抗体) complexes.
MondoA is a basic helix-loop-helix/leucine zipper transcription factor (显示 NRL 抗体) that is expressed predominantly in skeletal muscle. Mice deficient for MondoA excel in sprinting, as their skeletal muscles display an enhanced glycolytic capacity.
MondoA-Mlx (显示 MLX 抗体) complexes sense elevated levels of G6P and adenine nucleotides to trigger a TXNIP (显示 TXNIP 抗体)-dependent feedback inhibition of glycolysis
MONDOA forms heterodimers with MLX (MIM 602976) that can bind to and activate transcription from CACGTG E boxes (Billin et al., 2000
MLX interacting protein
, MLX-interacting protein-like
, MLX-interacting protein
, Mlx interactor
, class E basic helix-loop-helix protein 36
, transcriptional activator MondoA