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Probable adipokine. 再加上，我们可以发C1q and Tumor Necrosis Factor Related Protein 9 抗体 (30) 和 C1q and Tumor Necrosis Factor Related Protein 9 试剂盒 (16)和数多这个蛋白质的别的产品。
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CTRP9 levels are elevated in obesity and significantly decrease following weight loss surgery.
Plasma CTRP9 levels are associated with atherosclerosis in diabetic patients without CKD, independently of obesity, adiponectin, and traditional cardiovascular risk factors
The up-regulation of CTRP9 during hypertrophic heart disease facilitates maladaptive cardiac remodeling and left ventricular dysfunction.
Results demonstrate that CTRP9 alleviates hepatic steatosis through relief of endoplasmic reticulum stress via the AMPK (显示 PRKAA1 蛋白)-mediated induction of autophagy.
Study demonstrates that CTRP9 attenuates cytokine-induced vascular inflammation in endothelial cells mediated by AMPK (显示 PRKAA1 蛋白) activation.
Circulating and coronary CTRP9 plays an important role in the inflammation and coronary atherosclerosis of CAD patients. Serum CTRP9 is an independent protective factor of CAD
Serum CTRP9 concentration was significantly and positively associated with arterial stiffness in patients with type 2 diabetes
Serum CTRP9 concentrations were positively associated with favorable glucose or metabolic phenotypes and absence of metabolic syndrome, independent of serum total adiponectin concentrations.
CTRP9 is a cardiokine critical in maintaining a healthy microenvironment facilitating stem cell engraftment in infarcted myocardial tissue treated with mesenchymal stem cell transplantation.
Based on these findings, this study showed that CTRP9 might induce mitochondrial biogenesis and protect high glucose-induced endothelial oxidative damage via AdipoR1-SIRT1-PGC-1alpha signaling pathway.
These results suggested that gCTRP9 protected RAW 264.7 macrophages from oxLDL via AMPK (显示 PRKAA1 蛋白) activation in an AdipoR1 (显示 ADIPOR1 蛋白) dependent fashion.
Suggest reduced plasma CTRP9 concentration during diabetes plays a causative role in platelet hyper-activity, contributing to platelet-induced cardiovascular damage.
Enhancing cardiac CTRP9 production and/or its proteolytic posttranslational modification are of therapeutic potential, attenuating diabetic cardiac injury.
CTRP9 protects against acute cardiac damage in response to pathological stimuli by suppressing inflammatory reactions through AdipoR1 (显示 ADIPOR1 蛋白)/AMPK (显示 PRKAA1 蛋白)-dependent mechanisms.
CTRP9 reduces pro-inflammatory cytokines expression in atherosclerotic plaque.
Genetic evidence for a physiological role of CTRP9 in controlling energy balance via central and peripheral mechanisms.
Adipokine CTRP9 attenuates adverse cardiac remodeling after acute myocardial infarction, largely via a protein kinase A (PKA)-dependent pathway.
Probable adipokine. Activates AMPK, AKT, and p44/42 MAPK signaling pathways (By similarity).
complement C1q and tumor necrosis factor-related protein 9
, complement C1q and tumor necrosis factor-related protein 9A
, complement C1q tumor necrosis factor-related protein 9
, complement C1q tumor necrosis factor-related protein 9A