anti-TRPV2 (TRPV2) 抗体产品概述

Rabbit Transient Receptor Potential Cation Channel, Subfamily V, Member 2 (TRPV2) interaction partners

1. This study presents the atomic model of rabbit TRPV2 in its putative desensitized state, as determined by cryo-EM at a nominal resolution of approximately 4 A.

Human Transient Receptor Potential Cation Channel, Subfamily V, Member 2 (TRPV2) interaction partners

1. the expression of transient receptor potential vanilloid-1 (TRPV1 (显示 TRPV1 抗体)), transient receptor potential vanilloid-2 (TRPV2) and transient receptor potential vanilloid-3 (TRPV3 (显示 TRPV3 抗体)) channels in native human odontoblasts, was examined.

2. High TRPV2 expression is associated with breast cancer.

3. both TRPV2 and TRPV4 (显示 TRPV4 抗体) are involved in migration of human cardiac c-kit (显示 KIT 抗体)(+) progenitor cells.

4. The results of the study show that polymorphism of TRPV2 influenced the susceptibility to FM.

5. CGRP (显示 S100A12 抗体)- and TRPV2-containing trigeminal neurons probably innervate the paranasal sinus mucosae, and project into spinal and principal trigeminal sensory nuclei.

6. transient receptor potential vanilloid 2 is an intracellular Ca(2 (显示 CA2 抗体)+)-permeable transient receptor potential vanilloid channel upregulated by nerve growth factor via the mitogen-activated protein kinase (显示 MAPK1 抗体) signaling pathway to augment neurite outgrowth.

7. Using multiple sequence alignments as source for evolutionary, bioinformatics and statistical analysis, we have analyzed the evolutionary profiles for TRPV1 (显示 TRPV1 抗体), TRPV2, TRPV3 (显示 TRPV3 抗体) and TRPV4 (显示 TRPV4 抗体)

8. There appears to be TRPV1 (显示 TRPV1 抗体)- or TRPV2-IR sympathetic pathway in the human stellate ganglion and spinal cord.

9. This study evaluated the role of Trpv2 in arthritis and the pharmacological effects of Trpv2 agonists.

10. High expression of TRPV2 mRNA was closely associated with advanced pathological stage and worse survival of patients with esophageal squamous cell carcinoma.

Mouse (Murine) Transient Receptor Potential Cation Channel, Subfamily V, Member 2 (TRPV2) interaction partners

1. data suggest that knockout of the TRPV2 channel may attenuate macrophage-dependent pro-inflammatory processes and result in better cardiac recovery. TRPV2 may thus represent a novel therapeutic target for treatment of patients undergoing an acute MI

2. We conclude that TRPV2 function, as a stretch-activated channel, regulates the development of cardiomyocyte hypertrophy in response to increased afterload.

3. These findings suggest that TRPV2 activation via actin reorganization induced by Gq and G12 (显示 TCF3 抗体)/13 signaling is involved in LPI (显示 Slc7a7 抗体)-stimulated GLP-1 (显示 GCG 抗体) secretion in enteroendocrine L cells.

4. Expression of TRPV2 was dramatically increased during the differentiation of brown adipocytes. TRPV2 might be involved in the modulation of brown adipocyte differentiation partially via a calcineurin pathway.

5. TRPV2 channels are demonstrated here to play a key role in cation influx and dysregulation in dystrophin (显示 DMD 抗体) deficient cardiomyocytes, enhanced in stretching conditions

6. TRPV2KO mice show cold intolerance and impaired BAT (显示 BAAT 抗体) thermogenesis.

7. We conclude that TRPV2 is involved in osmosensation in skeletal muscle fibres, acting in concert with P-SPAK (显示 STK39 抗体)-activated NKCC1 (显示 SLC12A2 抗体)

8. TRPV2-knockout mice have impaired cardiac functional response to exercise.

9. TRPV2 channels play a predominant role in fluid flow mechanosensitivity over that of TRPC1 (显示 TRPC1 抗体) and TRP (显示 TYRP1 抗体) melastatin (显示 TRPM1 抗体) (TRPM) 7 (显示 TRPM7 抗体).

10. TRPV2 is involved in a stretch-activated calcium influx pathway in dystrophic cardiomyopathy, contributing to the defective cellular Ca2 (显示 CA2 抗体)+ handling in this disease.