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Our findings provide insight for the first time into the mechanisms involved in Rgs19 regulation of mouse ESC proliferation and differentiation.
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Notch signaling positively regulates phosphorylation of Akt, possibly via phosphorylation of RGS19
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RGS19 and its partner Galpha-inhibiting activity polypeptide 3 (GNAI3) are involved in zVAD-, but not TNF-, induced cell death.
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level of basal autophagy, autophagic induction, autophagic flux, autophagic degradation and the anti-autophagic action in macrophages that lacked Galphai3, AGS3, or RGS19; or had been treated with pertussis toxin, were similar to controls
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Rgs19 plays important roles in palatal fusion.
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Regulator of G protein signaling 19 enhances cell proliferation through binding to GAIP-interacting protein C-terminus.
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RGS19 influenced cardiac development and had negative effects on heart function.
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gene structure and transcription regulation
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RGS19 prefers regulating ORL1 receptor signaling over other opioid receptors, and the N-terminal domain of RGS19 plays a crucial role in its receptor preference
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Results suggest a complex role for RGS19 in regulating the ability of Wnt3a to signal to the level of beta-catenin and gene transcription.
