MAGE-Like 2 蛋白
(MAGE-Like 2 (MAGEL2))
Prader-Willi syndrome (PWS) is caused by the loss of expression of imprinted genes in chromosome 15q11-q13 region. Affected individuals exhibit neonatal hypotonia, developmental delay, and childhood-onset obesity. Necdin (NDN), a gene involved in the terminal differentiation of neurons, localizes to this region of the genome and has been implicated as one of the genes responsible for the etiology of PWS. This gene is structurally similar to NDN, is also localized to the PWS chromosomal region, and is paternally imprinted, suggesting a possible role for it in PWS. [provided by RefSeq, Oct 2010].
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4 results
custom-made
MAGEL2
宿主: 小鼠
宿主: Cell-free protein synthesis (CFPS)
Recombinant
approximately 70-80 % as determined by SDS PAGE, Western Blot and analytical SEC (HPLC).
ELISA, SDS, WB
custom-made
MAGEL2
宿主: 人
宿主: Cell-free protein synthesis (CFPS)
Recombinant
approximately 70-80 % as determined by SDS PAGE, Western Blot and analytical SEC (HPLC).
ELISA, SDS, WB
Aliases for MAGE-Like 2 蛋白
MAGE family member L2 (MAGEL2) 蛋白melanoma antigen, family L, 2 (Magel2) 蛋白
MAGE family member L2 (Magel2) 蛋白
Mage-l2 蛋白
MAGEL2 蛋白
NDNL1 蛋白
nM15 蛋白
ns7 蛋白
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