KCNJ10 抗体
(Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 (KCNJ10))
This gene encodes a member of the inward rectifier-type potassium channel family, characterized by having a greater tendency to allow potassium to flow into, rather than out of, a cell. The encoded protein may form a heterodimer with another potassium channel protein and may be responsible for the potassium buffering action of glial cells in the brain. Mutations in this gene have been associated with seizure susceptibility of common idiopathic generalized epilepsy syndromes. [provided by RefSeq, Jul 2008].
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Latest Publications for our KCNJ10 抗体
: "Identification and functional characterization of two novel mutations in KCNJ10 and PI4KB in SeSAME syndrome without electrolyte imbalance." in: Human genomics, Vol. 13, Issue 1, pp. 53, (2019) (PubMed).: "Development of the stria vascularis and potassium regulation in the human fetal cochlea: Insights into hereditary sensorineural hearing loss." in: Developmental neurobiology, Vol. 75, Issue 11, pp. 1219-40, (2015) (PubMed).
: "Free radical stress-mediated loss of Kcnj10 protein expression in stria vascularis contributes to deafness in Pendred syndrome mouse model." in: American journal of physiology. Renal physiology, Vol. 294, Issue 1, pp. F139-48, (2008) (PubMed).
Aliases for KCNJ10 抗体
potassium channel, inwardly rectifying subfamily J, member 10 L homeolog (kcnj10.L) 抗体potassium voltage-gated channel subfamily J member 10 (KCNJ10) 抗体
potassium channel, inwardly rectifying subfamily J, member 10 (kcnj10) 抗体
potassium inwardly-rectifying channel, subfamily J, member 10 (Kcnj10) 抗体
potassium voltage-gated channel subfamily J member 10 (Kcnj10) 抗体
BIR10 抗体
BIRK-1 抗体
birk-10 抗体
BIRK-10 抗体
KCNJ13-PEN 抗体
kir1.2 抗体
Kir1.2 抗体
KIR1.2 抗体
kir4.1 抗体
Kir4.1 抗体
KIR4.1 抗体
MGC145926 抗体
SESAME 抗体
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