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抗Mouse (Murine) Prokineticin Receptor 1 抗体:
抗Human Prokineticin Receptor 1 抗体:
抗Rat (Rattus) Prokineticin Receptor 1 抗体:
Human Polyclonal Prokineticin Receptor 1 Primary Antibody for FACS, IHC - ABIN347055
Brouillet, Murthi, Hoffmann, Salomon, Sergent, De Mazancourt, Dakouane-Giudicelli, Dieudonné, Rozenberg, Vaiman, Barbaux, Benharouga, Feige, Alfaidy: EG-VEGF controls placental growth and survival in normal and pathological pregnancies: case of fetal growth restriction (FGR). in Cellular and molecular life sciences : CMLS 2013
Show all 5 Pubmed References
The authors discovered that PKR1 regulates epicardial-mesenchymal transition (EMT (显示 ITK 抗体)) for epicardial-derived progenitor cell (EPDC), formation. This event affects at least three consequential steps during heart development: (i) EPDC and cardiomyocyte proliferation involved in thickening of an outer compact ventricular chamber wall, (ii) rhythmicity, (iii) formation of coronary circulation.
These results establish PKR1 via NFATc3 (显示 NFATC3 抗体) as a crucial modifier of mesenchymal-epithelial transition processing to the development of nephron.
show that MRAP2 significantly and specifically inhibits PKR1 signaling.
Data show that the prokineticins and their receptors PROK2 (显示 PROK2 抗体), PKR1 and PKR2 (显示 PROKR2 抗体) contributes to altered sensitivity in diabetic neuropathy and its inhibition blocked both allodynia and inflammatory events underlying disease.
These results suggest PKR1 to be a crucial player in the preadipocyte proliferation and differentiation.
Loss of PKR1 causes renal and cardiac structural and functional changes because of deficits in survival signaling, mitochondrial, and progenitor cell functions in found both organs.
The functional characteristics of coronary endothelial cells depend on the expression of PKR1 and PKR2 (显示 PROKR2 抗体) levels and the divergent signaling pathways used by these receptors.
Identification and molecular characterization of two closely related G protein-coupled receptors (prokineticin receptor)
PKR1 protein was localised to the labyrinth layer and showed the same pattern of expression as EG-VEGF (显示 Prok1 抗体) in mouse placenta.
Cardiomyocyte-PKR1 signaling upregulates its own ligand prokineticin-2 (显示 PROK2 抗体) that acts as a paracrine factor, triggering epicardial-derived progenitor cell proliferation/differentiation.
a significant association between PKR2 rs6053283 polymorphism and Recurrent pregnancy loss (RPL)(P=0.003), whereas no association was observed between PKR1 rs4627609 polymorphism and RPL (P=0.929) in the Chinese Han population.
Data suggest that prokineticins (PROK1 (显示 Prok1 抗体) and PROK2 (显示 PROK2 抗体)) and prokineticin receptors (PROKR1 and PROKR2 (显示 PROKR2 抗体)) act as main regulators of physiological functions of ovary, uterus, placenta, and testis. [REVIEW]
EG-VEGF (显示 Prok1 抗体) and its receptor PKR1 might play a role in the pathogenesis of adrenocortical tumors and could serve as prognostic markers for this rare malignant disease.
Expression of PROK1 (显示 Prok1 抗体) and PROKR1 was significantly higher in mid-gestation ovaries (17-20 wk) than at earlier gestations (8-11 and 14-16 wk).
Study corroborates the clinical relevance of the EG-VEGF (显示 Prok1 抗体) system in human early pregnancy, and provides evidence for the gene-gene interactions of EG-VEGF (显示 Prok1 抗体) and PROKR variants.
hCG (显示 CGA 抗体) increases EG-VEGF (显示 Prok1 抗体), PROKR1 and PROKR2 (显示 PROKR2 抗体) mRNA and protein expression in a dose- and time-dependent manner, demonstrating a new role for hCG (显示 CGA 抗体) in the regulation of EG-VEGF (显示 Prok1 抗体) and its receptors
Findings, together with the detection of sequence variants in PROKR1, PROK1 (显示 Prok1 抗体) and PROKR2 (显示 PROKR2 抗体) genes associated to HSCR (显示 EDNRB 抗体) and, in some cases in combination with RET (显示 RET 抗体) or GDNF mutations, provide evidence to consider them as susceptibility genes for HSCR (显示 EDNRB 抗体).
The number of PKR1 is not reduced in preeclampsia.
Data suggest that smoking targets human Fallopian tubes via nAChRalpha-7 to increase tubal PROKR1, leading to alterations in the tubal microenvironment that could predispose to ectopic pregnancy.
Data show that expression of PK1 (显示 PKLR 抗体) and PKR1 was detected in primary MM cells and myeloma cell lines.
PROK1 (显示 Prok1 抗体), acting via PROKR1, may be involved in the recruitment of monocytes to regressing CL and atretic follicles and their consequent activation therein.
G protein-coupled receptor for prokineticin
G protein-coupled receptor 73
, G-protein coupled receptor 73
, G protein-coupled receptor ZAQ
, G-protein coupled receptor ZAQ
, prokineticin receptor 1
, prokineticin receptor 1-like