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抗Mouse (Murine) ADCY1 抗体:
抗Human ADCY1 抗体:
抗Rat (Rattus) ADCY1 抗体:
Human Polyclonal ADCY1 Primary Antibody for ELISA, WB - ABIN2746148
Nakamura, Niimi, Yanagawa: Protection from sepsis-induced acute renal failure by adenoviral-mediated gene transfer of beta2-adrenoceptor. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2010
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data indicate that ST034307 is a selective small-molecule inhibitor of AC1 (显示 HRASLS 抗体) and suggest that selective AC1 (显示 HRASLS 抗体) inhibitors may be useful for managing pain.
Genetic deletion of AC8 (adenylyl cyclase 8 (显示 ADCY8 抗体)) but not AC1 (显示 HRASLS 抗体) abolished long-lasting anxiety.
Following ethanol administration, phosphorylation of TrkB (显示 NTRK2 抗体) is significantly increased in the striatum of Adcyl1 and Adcyl6 knock-out mice.
Findings provide evidence to link activation of alpha7 nAChRs to a cAMP rise via AC1 (显示 HRASLS 抗体), which defines a new signaling pathway employed by alpha7 nAChRs
Results indicate that thalamic AC1 (显示 HRASLS 抗体) plays a major role in patterning and refinement of the mouse thalamocortical circuitry
Adenylyl cyclase-mediated effects contribute to increased isoprenaline-induced cardiac contractility in TRPM4 (显示 TRPM4 抗体)-deficient mice.
Results demonstrate that ADCY1 has an evolutionarily conserved role in hearing.
Data indicate that genetic ablation of RGS6 (显示 RGS6 抗体) expression resulted in anxiolytic and antidepressant behavior by enhancing signaling through the 5-HT1A receptor (显示 CC2D1A 抗体)-adenylyl cyclase (AC) axis.
Contrast sensitivity is similarly reduced in Adcy1-/- mice.
These results suggest that AC1 (显示 HRASLS 抗体) in retinal ganglion cell axons mediates the development of retinotopy and eye-specific segregation in the superior colliculus and dorsal lateral geniculate nucleus.
data indicate that ST034307 is a selective small-molecule inhibitor of AC1 and suggest that selective AC1 inhibitors may be useful for managing pain.
STC1 (显示 STC1 抗体) interferes with CALCRL (显示 CALCRL 抗体) signaling during osteoblastogenesis via adenylate cyclase inhibition.
AC1 catalytic activity can be adjusted by mediating calmodulin activation of AC1 by reversible methionine oxidation in calmodulin.
Increased expression of AC1 in the forebrain leads to deficits in behavioral inhibition.
AGS3 (显示 GPSM1 抗体) reduced D(2L)DR-mediated sensitization of AC1 and AC2.
Compartmentalized AC1-CFTR (显示 CFTR 抗体) association is responsible for Ca(2 (显示 CA2 抗体)+)/cAMP cross-talk.
Tissue transglutaminase directly regulates adenylyl cyclase resulting in enhanced cAMP-response element-binding protein (CREB) activation.
Transgenic mice overexpressing type-1 adenylyl cyclase in the forebrain show elevated long-term potentiation (LTP (显示 SCP2 抗体)), increased memory for object recognition and slower rates of extinction for contextual memory.
Expression of sensitization of AC1 involves Galpha (显示 SUCLG1 抗体)(s)-adenylyl cyclase interactions.
This gene encodes a form of adenylate cyclase expressed in brain. A similar protein in mouse is involved in pattern formation of the brain.
brain adenylate cyclase 1
, adenylate cyclase 1 (brain)
, adenylate cyclase type 1-like
, ATP pyrophosphate-lyase 1
, Ca(2+)/calmodulin-activated adenylyl cyclase
, adenylate cyclase type 1
, adenylate cyclase type I
, adenylyl cyclase 1
, 3',5'-cyclic AMP synthetase
, adenyl cyclase
, ca(2+)/calmodulin-activated adenylyl cyclase