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Methamphetamine 抗体

M-Amp ELISA 宿主: 小鼠 Monoclonal 4D2 unconjugated
产品编号 ABIN723186
发货至: 中国
  • 抗原 See all Methamphetamine (M-Amp) products
    Methamphetamine (M-Amp)
    适用
    请咨询
    宿主
    • 15
    • 15
    • 2
    小鼠
    克隆类型
    • 17
    • 15
    单克隆
    标记
    • 11
    • 3
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    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
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    This Methamphetamine antibody is un-conjugated
    应用范围
    • 15
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    • 5
    • 3
    • 3
    • 2
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    ELISA
    交叉反应 (详细)
    Methamphetamine
    纯化方法
    Purified by Protein G.
    免疫原
    KLH conjugated Methamphetamine
    克隆位点
    4D2
    亚型
    IgG
  • 限制
    仅限研究用
  • 状态
    Liquid
    浓度
    1 μg/μL
    缓冲液
    0.01M TBS( pH 7.4) with 1 % BSA, 0.02 % Proclin300 and 50 % Glycerol.
    储存液
    Sodium azide
    注意事项
    This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    储存条件
    4 °C,-20 °C
    储存方法
    Shipped at 4°C. Store at -20°C for one year. Avoid repeated freeze/thaw cycles.
    有效期
    12 months
  • 抗原
    Methamphetamine (M-Amp)
    别名
    Methamphetamine (M-Amp 产品)
    物质类
    Chemical
    背景

    Synonyms: d-Desoxyephedrine hydrochloride, d-N, -Dimethylphenethylamine hydrochloride, Methylamphetamine hydrochloride, METH.

    Background: Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.

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