UL16 Binding Protein 2 (ULBP2) ELISA试剂盒

Ligand for the NKG2D receptor, together with at least ULBP1 and ULBP3. 再加上,我们可以发ULBP2 抗体 (45)ULBP2 蛋白 (13)和数多这个蛋白质的别的产品。

list all ELISA KIts 基因 基因ID UniProt
ULBP2 80328 Q9BZM5
ULBP2    
ULBP2    
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Showing 10 out of 16 products:

产品编号 适用 灵敏度 范围 图像 规格 交付 价格 详细
0.055 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests 13至16个工作日
$810.53
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小鼠 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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豚鼠 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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大鼠 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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Pig
  96 Tests 15至18个工作日
$707.14
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绵羊 1.0 ng/mL 2.5-50 ng/mL   96 Tests 15至18个工作日
$707.14
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0.1 ng/mL 0.5-10 ng/mL   96 Tests 15至18个工作日
$707.14
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1.3 pg/mL n/a   96 Tests 11至16个工作日
$617.38
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适于 ULBP2 相互作用对的更多 ELISA 试剂盒

Human UL16 Binding Protein 2 (ULBP2) interaction partners

  1. Data show that the response of NK cells activated by ULBP2 was augmented by an interaction between NKG2D ligand 2 protein (ULBP2)-bound natural killer group 2D (NKG2D) and IL- interleukin 15 receptor (IL-15R).

  2. NK cells are important cytotoxic cells in the immune system, and the activated NKG2D receptor on the NK cell surface can bind to NKG2D ligands expressed in tumor cells, enabling NK cells to activate and kill tumor cells. (Review)

  3. High ULBP2 expression is associated with lymphoma.

  4. IMP3 directly interacts with ULBP2 mRNA, leading to ULBP2 transcript destabilization and reduced ULBP2 surface expression and indirectly downregulates MICB with a mechanism functionally distinct from that of ULBP2.

  5. By suppressing AR and up-regulating ULBP2 in HCC, cisplatin could up-regulate cytotoxicity of NK cells to better target HCC.

  6. human tumor cells lost their surface expression of ULBP2, but not ULBP1 and ULBP3, during NK cell-mediated cytolysis.

  7. This study suggests that NKG2D ligands shedding of MICA, MICB and ULBP-2 is a novel pathway in endometriosis complex pathogenesis that impairs Natural Killer Cells cell function.

  8. Varicella-Zoster Virus differentially modulates expression of the NKG2D ligands by upregulating MICA expression and reducing ULBP2 and ULBP3 expression in the infected cells.

  9. A conserved WW domain-like motif regulates CD74 antigen-dependent cell-surface transport of the NKG2D ligand ULBP2.

  10. NKG2D and NKG2DL are involved in allergen-induced activation of dendritic epidermal T cells and the NKG2D/NKG2DL pathway might be a potential target for treatment of contact hypersensitivity.

  11. Human anaplastic thyroid carcinoma cells are sensitive to NK cell-mediated lysis via ULBP2/5/6 and chemoattract NK cells.

  12. Data inticate that heat shock protein 60 (HSP60) interacted constitutively with NKG2D ligand ULBP2 and phosphatase of regenerating liver 3 (PRL-3) regulated HSP60 tyrosine phosphorylation.

  13. Results suggest that ULBP2 is expressed and released from cervical cancer cells by CRF, which regulates NKG2D expression in natural killer cells.

  14. c-Cbl regulates MICA- but not ULBP2-induced NKG2D down-modulation in human NK cells.

  15. the NKG2D ligand ULBP2 is transported to the cell surface through an endosomal pathway dependent on protein kinase C and lysosomal integrity. ULBP2 surface transport is dependent on the invariant chain.

  16. This study provides for the first time, the c-Myc dependent regulation of NKG2D ligands, ULBP1/2/3 in acute myeloid leukemia.

  17. Vpr augments ULBP2 expression on both infected and uninfected bystander cells during HIV-1 infection of primary CD4+ T lymphocytes.

  18. Study shows that tumor-suppressive miR-34a and miR-34c act as ULBP2 repressors. Findings also implicate p53 in ULBP2 regulation, emphasizing the role of the specific NKG2DL in tumor immune surveillance.

  19. Data show that VSV infection caused an active suppression of NKG2D-ligand surface expression, affecting both endogenous and histone deacetylase (HDAC)-inhibitor induced MICA, MICB and ULBP-2 expression.

  20. Findings define the involvement of p53 in the regulation of ULBP1 and ULBP2 which enhance NK cell-mediated target recognition.

ULBP2 抗原简介

Antigen Summary

Ligand for the NKG2D receptor, together with at least ULBP1 and ULBP3. ULBPs activate multiple signaling pathways in primary NK cells, resulting in the production of cytokines and chemokines. Binding of ULBPs ligands to NKG2D induces calcium mobilization and activation of the JAK2, STAT5, ERK and PI3K kinase/Akt signal transduction pathway. In CMV infected cells, interacts with soluble CMV glycoprotein UL16. The interaction with UL16 blocked the interaction with the NKG2D receptor, providing a mechanism by which CMV infected cells might escape the immune system. UL16 also causes ULBP2 to be retained in the ER and cis- Golgi apparatus so that it does not reach the cell surface.

Gene names and symbols associated with ULBP2

  • UL16 binding protein 2 (ULBP2) 抗体
  • NKG2D ligand 2-like (LOC100355436) 抗体
  • N2DL2 抗体
  • RAET1H 抗体

Protein level used designations for ULBP2

ALCAN-alpha , N2DL-2 , NKG2D ligand 2 , NKG2DL2 , UL16-binding protein 2 , retinoic acid early transcript 1 H , retinoic acid early transcript 1H

GENE ID SPECIES
80328 Homo sapiens
100355436 Oryctolagus cuniculus
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