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TNFRSF6B belongs to the tumor necrosis factor receptor superfamily. 再加上，我们可以发TNFRSF6B 抗体 (81) 和 TNFRSF6B 蛋白 (10)和数多这个蛋白质的别的产品。
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Human TNFRSF6B ELISA Kit for Sandwich ELISA - ABIN1979508
Lancrajan, Schneider-Stock, Naschberger, Schellerer, Stürzl, Enz: Absolute quantification of DcR3 and GDF15 from human serum by LC-ESI MS. in Journal of cellular and molecular medicine 2015
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Compared with the control, miR (显示 MLXIP ELISA试剂盒)-340 was significantly lower in the serum of hepatocellular carcinoma patients (p<0.01). miR (显示 MLXIP ELISA试剂盒)-340 was lower in HCC tissues than in adjacent; however, DcR3, TGF-beta1 (显示 TGFB1 ELISA试剂盒) and Smad2 (显示 SMAD2 ELISA试剂盒) were higher.
DcR3 treatment caused HepG2 cell cytoskeleton remodeling, inhibited E-cadherin (显示 CDH1 ELISA试剂盒) expression, and promoted cell migration.
DcR3 gene polymorphisms are associated with sporadic breast infiltrating ductal carcinoma risk in Northeast Chinese females
Study found that both transcriptional expression and post-transcriptional DcR3 expression DcR3 expression were significantly up-regulated in colorectal cancer (CRC (显示 CALR ELISA试剂盒)) tissues. DcR3 was found to be playing a critical role in CRC (显示 CALR ELISA试剂盒) proliferation and migration in vitro and in tumorigenesis and metastasis in vivo.
Higher plasma DcR3 level was related to better coronary collateral circulation (CCC) formation and displayed potent predictive power for CCC status in patients with coronary artery disease.
High serum DcR3 levels are associated with disease severity in nonatopic asthma patients.
Results show that DcR3 engages TNF (显示 TNF ELISA试剂盒) ligands: FasL (显示 FASL ELISA试剂盒), LIGHT, and TL1A (显示 TNFSF15 ELISA试剂盒) in a structurally similar fashion and its promiscuity is due to recognition of backbone atoms and invariant side chains.
These results raise the possibility for involvement of TL1A (显示 TNFSF15 ELISA试剂盒)/DR3/DR3 (显示 TNFRSF25 ELISA试剂盒)-mediated mechanisms in epithelial-mesenchymal interactions and the development of inflammation-induced intestinal fibrosis in Crohn's disease.
when idiopathic pulmonary fibrosis cells interact with collagen matrix, aberrantly activated Akt (显示 AKT1 ELISA试剂盒) increases DcR3 expression via GSK-3beta-NFATc1 (显示 NFATC1 ELISA试剂盒) and protects IPF cells from the FasL (显示 FASL ELISA试剂盒)-dependent apoptotic pathway.
Coexpression of TNFRSF6B and PCDHGA3 was observed immunohistochemically in FL18 cells, suggesting potential cooperation in tumor cell maintenance.
This gene belongs to the tumor necrosis factor receptor superfamily. The encoded protein is postulated to play a regulatory role in suppressing FasL- and LIGHT-mediated cell death. It acts as a decoy receptor that competes with death receptors for ligand binding. Over-expression of this gene has been noted in gastrointestinal tract tumors. Read-through transcription into this gene from the neighboring upstream gene, which encodes regulator of telomere elongation helicase 1 (RTEL1), generates a non-coding transcript.
decoy receptor 3
, decoy receptor for Fas ligand
, tumor necrosis factor receptor superfamily member 6B
, tumor necrosis factor receptor superfamily, member 6b
, tumor necrosis factor receptor superfamily, member 6b, decoy