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TRPV5 is a member of the transient receptor family and the TrpV subfamily. 再加上，我们可以发TRPV5 蛋白 (7) 和 TRPV5 试剂盒 (2)和数多这个蛋白质的别的产品。
Showing 10 out of 81 products:
Human Polyclonal TRPV5 Primary Antibody for ICC, IF - ABIN408730
van der Eerden, Hoenderop, de Vries, Schoenmaker, Buurman, Uitterlinden, Pols, Bindels, van Leeuwen: The epithelial Ca2+ channel TRPV5 is essential for proper osteoclastic bone resorption. in Proceedings of the National Academy of Sciences of the United States of America 2005
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Cow (Bovine) Polyclonal TRPV5 Primary Antibody for IHC, WB - ABIN2783755
Cha, Wu, Huang: Protein kinase C inhibits caveolae-mediated endocytosis of TRPV5. in American journal of physiology. Renal physiology 2008
Human Polyclonal TRPV5 Primary Antibody for ELISA, ICC - ABIN263199
Kennedy, Torabi, Kurzawa, Echtenkamp, Mangini: Expression of transient receptor potential vanilloid channels TRPV5 and TRPV6 in retinal pigment epithelium. in Molecular vision 2010
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TRPV5 and TRPV6 (显示 TRPV6 抗体) lack a positively charged residue in the TM4 (显示 TPM4 抗体)-TM5 loop that was shown to interact with PI(4,5)P2 in TRPV1 (显示 TRPV1 抗体), which shows high affinity for this lipid
The alterations may lead to misfolding of TRPV5, reduction in translocation of the channel to the plasma membrane and/or impaired Ca(2 (显示 CA2 抗体)+) transport function of the channel, and ultimately disrupt TRPV5-mediated Ca(2 (显示 CA2 抗体)+) reabsorption.
These findings indicate that the A563T variation induces structural, dynamical, and electrostatic changes in the TRPV5 pore, providing structural insight into the functional alterations associated with the A563T variation.
Data found that a TRPV5 polymorphism (rs4236480) was observed to be associated with stone multiplicity of calcium nephrolithiasis, as the risk of stone multiplicity was higher in patients with the TT+CT genotype than in patients with the CC genotype
High TRPV5 expression is aassociated with adenoma of parathyroid glands.
Klotho (显示 KL 抗体) up-regulates TRPV5 from both the inside and outside of cells.
Decreased expression of TRPV5 is associated with non-small cell lung cancer.
Upregulating the expression of TRPV5 can be utilized to manipulate transmembrane Ca2 (显示 CA2 抗体)+ transport, and may serve as an alternative for the treatment of Ca2 (显示 CA2 抗体)+ balance-related diseases.
These results suggest that TRPV5 and TRPV6 (显示 TRPV6 抗体) are crucial gates controlling cadmium and zinc levels in the human body.
these data showed that TRPV5/TRPV6 (显示 TRPV6 抗体) in human lymphocytes are functionally active, and their activity is associated with proliferative status of blood cells.
life-long challenge of Ca2 (显示 CA2 抗体)+ homeostasis present in Trpv5-/- mice causes accelerated bone aging and a low cortical and trabecular bone mass phenotype
beta-gal (显示 GLB1 抗体) is present in the pro-urine from where it is thought to stimulate TRPV5 activity.
A novel primary cell model with TRPV5-dependent Ca(2 (显示 CA2 抗体)+) transport characteristics was successfully established.
Phosphorylation of beta1-AR by PKA stimulates active Ca(2 (显示 CA2 抗体)+)influx through TRPV5.
The expression of TRPV5 channel is regulated by estrogen via estrogen receptor (显示 ESR1 抗体) in osteoblasts.
Uromodulin (显示 UMOD 抗体) upregulates TRPV5 by decreasing caveolin-1 (显示 CAV1 抗体) dependent endocytosis of TRPV5.
In colitis in mice, tumor necrosis factor (显示 TNF 抗体) and interferon-gamma (显示 IFNG 抗体) reduced expression of Klotho (显示 KL 抗体), which otherwise would protect TRPV5 from hypersialylation and cytokine-induced TRPV5 endocytosis, UBR4 (显示 UBR4 抗体)-dependent ubiquitination, degradation, and Ca(2 (显示 CA2 抗体)+) wasting.
The TRPV5 S682P mutant is a functionally significant factor in autosomal dominant hypercalciuria.
promoter driven Cre recombinase (显示 RAG1 抗体) expression will be useful for inducing DCT2 and CNT specific gene silencing of various channels, pumps, carriers, and receptors
Calcitonin (显示 CALCA 抗体) augments the renal reabsorptive capacity for Ca(2 (显示 CA2 抗体)+). This increase is likely to occur independently of TRPV5.
mutation of a pore helix residue glutamate (显示 GRIN2A 抗体)-535 to glutamine (显示 GFPT2 抗体) (E535Q) enhances the sensitivity of the channel to inhibition by extracellular protons
Results suggest that intracellular acidification directly inhibits TRPV5 by causing a conformational change(s) leading to a decrease of open probability of TRPV5 as well as of the single-channel conductance.
Activation of the CaR stimulates TRPV5-mediated Ca(2+) influx via a PMA-insensitive PKC isoform pathway.
TRPV5 and TRPV6 (显示 TRPV6 抗体) were upregulated with time and passage in culture suggesting that a shift in the phenotype of the cells in monolayer culture alters the expression of these channels.
In Equus caballus, TRPV5 mRNA is highly expressed in the kidney but scarce in the duodenum and heart. Protein expression followed a similar pattern.
This gene is a member of the transient receptor family and the TrpV subfamily. The calcium-selective channel encoded by this gene has 6 transmembrane-spanning domains, multiple potential phosphorylation sites, an N-linked glycosylation site, and 5 ANK repeats. This protein forms homotetramers or heterotetramers and is activated by a low internal calcium level.
, calcium transporter 2
, epithelial calcium channel 1
, osm-9-like TRP channel 3
, transient receptor potential cation channel subfamily V member 5
, calcium transport protein 2
, transient receptor potential cation channel, subfamily V, member 5
, calcium transporter 2 L homeolog