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SULT4A1 encodes a member of the sulfotransferase family.
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These results show that SULT4A1 is widely expressed in human tissues, but mostly as a splice variant that produces a rapidly degraded protein. Dimerization protects the protein from degradation.
Across three psychiatric disorders (n=2815 patients), we observed no consistent association between SULT4A1-1 status and atypical antipsychotic effect
This study provides a second replication of superior olanzapine response in SULT4A1-1-positive subjects compared with SULT4A1-1-negative subjects.
determination of SULT4A1-1 haplotype status might be useful for identifying patients who show an enhanced response to long-term olanzapine treatment.
the first immunohistochemical localization of SULT4A1 in human brain
These results provide the first evidence of how genetic variation in Sult4A1 may be related to clinical symptoms and cognitive function in schizophrenia
The lack of polymorphisms in the coding region of the SULT4A1 gene is highly unusual and, along with its high conservation between species, suggests that SULT4A1 may have an important function in vivo.
Cytosolic SULT4A1 interacts with PIN1 (显示 PIN1 抗体).
casual observations were made that sult4a1 mutant zebrafish were exhibiting excessively sedentary behavior during the day
This study is the first identification of a cellular process whose regulation appears to be associated with SULT4A1 expression.
This gene encodes a member of the sulfotransferase family. The encoded protein is a brain-specific sulfotransferase believed to be involved in the metabolism of neurotransmitters. Polymorphisms in this gene may be associated with susceptibility to schizophrenia.
, brain sulfotransferase-like protein
, nervous system cytosolic sulfotransferase
, nervous system sulfotransferase
, sulfotransferase 4A1
, sulfotransferase-related protein
, sulfotransferase family 4A, member 1
, brain sulfotransferase 4A1
, putative sulfotransferase family 4A member 1