anti-Solute Carrier Family 2 (Facilitated Glucose Transporter), Member 3 (SLC2A3) 抗体

Facilitative glucose transporter. 再加上,我们可以发SLC2A3 试剂盒 (30)SLC2A3 蛋白 (7)和数多这个蛋白质的别的产品。

列出全部抗体 基因 基因ID UniProt
SLC2A3 6515 P11169
SLC2A3 20527 P32037
SLC2A3 25551 Q07647
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antibodies-online.cn销售最多的anti-SLC2A3 抗体

Showing 10 out of 100 products:

产品编号 适用 宿主 标记 应用范围 图像 规格 交付 价格 详细
Cow 非结合性 WB WB Suggested Anti-SLC2A3 Antibody Titration: 1 ug/mlPositive Control: Cultured mouse primary cortex neuron, mouse cerebellum tissue sample, cultured mouse primary cortex astrocyte, 100 μL 2至3个工作日
$319.00
详细
非结合性 WB Western blot analysis of GLUT3 expression in COLO205 (A), HepG2 (B) whole cell lysates. 200 μL 13至14个工作日
$487.50
详细
非结合性 ELISA, ICC, IF, IHC, WB IHC analysis of human colon tissue, using GLUT3 Antibody at 1/100. ABIN6266554 at 1/100 staining Human liver cancer tissue by IHC-P. The sample was formaldehyde fixed and a heat mediated antigen retrieval step in citrate buffer was performed. The sample was then blocked and incubated with the antibody for 1.5 hours at 22°C. An HRP conjugated goat anti-rabbit antibody was used as the secondary. 100 μL 11至12个工作日
$390.77
详细
非结合性 IHC, ELISA, WB Western blot analysis of extracts from LOVO cells, using GLUT3 Antibody. The lane on the right is treated with the synthesized peptide. Immunohistochemistry analysis of paraffin-embedded human brain tissue, using GLUT3 Antibody. The picture on the right is treated with the synthesized peptide. 100 μg 2至3个工作日
$302.50
详细
非结合性 ELISA, IHC, WB Western Blot (WB) analysis of Mouse Kidney lysis using Glut3 antibody. 100 μL Available
$363.46
详细
非结合性 ELISA, IHC, IHC (p), WB Anti-SLC2A3 / GLUT3 antibody IHC staining of human tonsil, germinal center. Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval. Antibody  ABIN959380 concentration 10 ug/ml. 50 μL 11至14个工作日
$484.00
详细
Cow 非结合性 WB 100 μL 11至14个工作日
$581.17
详细
非结合性 ELISA, ICC, IF, IHC, WB ABIN6269009 at 1/100 staining Human liver cancer tissue by IHC-P. The sample was formaldehyde fixed and a heat mediated antigen retrieval step in citrate buffer was performed. The sample was then blocked and incubated with the antibody for 1.5 hours at 22°C. An HRP conjugated goat anti-rabbit antibody was used as the secondary. ABIN6269009 staining LOVO by IF/ICC. The sample were fixed with PFA and permeabilized in 0.1% Triton X-100,then blocked in 10% serum for 45 minutes at 25°C. The primary antibody was diluted at 1/200 and incubated with the sample for 1 hour at 37°C. An Alexa Fluor 594 conjugated goat anti-rabbit IgG (H+L) Ab, diluted at 1/600, was used as the secondary antibody. 100 μL 11至12个工作日
$390.77
详细
非结合性 WB Immunohistochemistry of paraffin-embedded mouse heart using SLC2A3 antibody. 100 μL 11至13个工作日
$366.77
详细
非结合性 IHC, WB   100 μg 11至14个工作日
$1,277.83
详细

引用最多的anti-SLC2A3 抗体

  1. Human Monoclonal SLC2A3 Primary Antibody for CyTOF, FACS - ABIN4900764 : Dimitriadis, Maratou, Boutati, Kollias, Tsegka, Alevizaki, Peppa, Raptis, Hadjidakis: IGF-I increases the recruitment of GLUT4 and GLUT3 glucose transporters on cell surface in hyperthyroidism. in European journal of endocrinology 2008 (PubMed)
    Show all 3 Pubmed References

  2. Human Polyclonal SLC2A3 Primary Antibody for IHC, IHC (p) - ABIN4314618 : Wang, Xiao, Li, Aziz, Gan, Johnson, Chen: AMPK modulates Hippo pathway activity to regulate energy homeostasis. in Nature cell biology 2015 (PubMed)
    Show all 2 Pubmed References

更多抗SLC2A3的相互作用对抗体

Human Solute Carrier Family 2 (Facilitated Glucose Transporter), Member 3 (SLC2A3) interaction partners

  1. GLUT3 plays a fundamental role in the survival and resistance of acute myeloid leukemia (AML) cells to vincristine.

  2. Results show that upregulation of the SLC2A3 gene is associated with decreased overall survival and disease-free survival in colorectal cancer patients. SLC2A3 gene expression analysis may be useful for predicting prognosis and survival of CRC patients.

  3. GLUT3 was upregulated in bevacizumab-resistant glioblastoma versus sensitive xenografts and patient specimens.

  4. 1) that decrease of GLUT3 is associated with the reduction of protein O-GlcNAcylation in Alzheimer's disease (AD)brain, 2) that GLUT3 level is negatively correlated with calpain I activation in human brain, 3) that calpain I proteolyzes GLUT3 at the N-terminus in vitro, and 4) that activation of calpain I is negatively correlated with protein O-GlcNAcylation in AD brain.

  5. Study provides evidence that rs7309332 genetic variant in GLUT3 may be useful in predicting survival of patients with early stage non-small cell lung cancer.

  6. We also showed that IL-8 stimulation of colon and lung cancer cells-induced glucose uptake and expressions of glucose transporter 3 (GLUT3) and glucosamine fructose-6-phosphate aminotransferase (GFAT), a regulator of glucose flux to the hexosamine biosynthetic pathway, resulting in enhancement of protein O-GlcNAcylation

  7. means of immunolabeled cells for GLUT-3 varied approximately from 19% to 73%

  8. PPAR-gamma and Akt regulate GLUT1 and GLUT3 surface localization during Mycobacterium tuberculosis infection

  9. miR-29c was down-regulated in Prostate cancer samples. SLC2A3, a regulator of glycolysis, was validated as a direct target of miR-29c. Moreover, functional studies showed miR-29c could inhibit cell growth, induce apoptosis and deceased the rate of glucose metabolism.

  10. Both common and rare SLC2A3 variation impacting regulation of neuronal glucose utilization and energy homeostasis may result in neurocognitive deficits known to contribute to ADHD risk.

  11. N-methyl-D aspartate (NMDA) receptor activity increases GLUT3 expression.

  12. identify a subset of tumors within the "proneural" and "classical" subtypes that are addicted to aberrant signaling from integrin alphavbeta3, which activates a PAK4-YAP/TAZ signaling axis to enhance Glut3 expression

  13. GLUT3-mediated glucose utilization and glycogenolysis in platelets promotes alpha-granule release, platelet activation, and postactivation functions.

  14. Studies suggest that a combination of glucose transporter s (GLUTs) 1 and 3 might help predict malignancy of cancers and direct effective cancer therapy.

  15. We found that insulin increases the cytotoxic effect of 5-fluorouracil and cyclophosphamide in vitro up to two-fold. The effect was linked to enhancement of apoptosis, activation of apoptotic and autophagic pathways, and overexpression of glucose transporters 1 and 3 as well as inhibition of cell proliferation and motility

  16. effects of physiologically relevant phospholipids on glucose transport in liposomes containing purified GLUT4 and GLUT3. The anionic phospholipids, phosphatidic acid, phosphatidylserine, phosphatidylglycerol, and phosphatidylinositol, were found to be essential for transporter function by activating it and stabilizing its structure.

  17. High Glut3 expression is associated with gastric cancer.

  18. Both Glut1 and GLUT3 are strongly expressed by papillary thyroid carcinomas, and their expressions were significantly associated with the presence of the BRAF V600E mutation.

  19. GLUT3 and OCT4 expression were correlated suggesting that Human embryonic stem cells self-renewal is regulated by the rate of glucose uptake.

  20. Using lipidic cubic phase crystallization and microfocus X-ray diffraction, we determined the structure of human GLUT3 in complex with D-glucose at 1.5 A resolution in an outward-occluded conformation

Mouse (Murine) Solute Carrier Family 2 (Facilitated Glucose Transporter), Member 3 (SLC2A3) interaction partners

  1. Lack of Glut3 during early postnatal life, results in a reduction in dendritic spines and brain size, along with a shortened lifespan.

  2. 1) that decrease of GLUT3 is associated with the reduction of protein O-GlcNAcylation in Alzheimer's disease (AD)brain, 2) that GLUT3 level is negatively correlated with calpain I activation in human brain, 3) that calpain I proteolyzes GLUT3 at the N-terminus in vitro, and 4) that activation of calpain I is negatively correlated with protein O-GlcNAcylation in AD brain.

  3. Study found no differences in mRNA or protein levels of neuronal monocarboxylate transporters (MCTs). Functional analyses revealed that neuronal MCT2 had high catalytic efficiency in Huntington's disease (HD) cells. Ascorbic acid did not stimulate lactate uptake in HD cells; and was unable to inhibit glucose transport in HD cells because they exhibit decreased expression of the neuronal glucose transporter GLUT3.

  4. Neonatal hypothyroridism downregulates the expressions of GLUT3 and GLUT8 in the testis of perpubertal mice.

  5. GLUT3-mediated glucose utilization and glycogenolysis in platelets promotes alpha-granule release, platelet activation, and postactivation functions.

  6. Morphological changes and GLUT1, GLUT3, and GLUT4 expression were evaluated in placentas by immunohistochemical and image analysis and correlated with iAs and arsenical species concentration, which were quantified by atomic absorption spectroscopy

  7. Results define Glut3 to be a rab11-dependent trafficking cargo and suggest that impaired Glut3 trafficking arising from rab11 dysfunction underlies the glucose hypometabolism observed in Huntington's disease

  8. Glut3 is a downstream target of mTORC1, and it is critical for oncogenic mTORC1-mediated aerobic glycolysis and tumorigenesis.

  9. GLUT3 levels in the brains of scrapie-infected animals was significantly downregulated.

  10. Placental endoplasmic reticulum stress by administration of Tun causes downregulation of Slc2a1(GLUT1) and upregulation of Slc2a3(GLUT3) mRNA expression.

  11. knock-down of glucose transporter 3 in embryonic stem cells impaired the beating function of ESC-derived cardiomyocytes, suggesting its potential role in mediating stem cell differentiation

  12. Data suggest that co-activation of CREST (calcium-responsive transactivator) and CBP (CREB-binding protein) enhances signaling between p-Creb/AP-1 and p-HIF-1/HRE resulting in up-regulation of Glut3 gene; here, stimulus was cell hypoxia.

  13. Recruitment of Creb1-Mecp2 by glut3-(m)CpG contributes towards transactivation, formulating an escape from (m)CpG-induced gene suppression, and thereby promoting developmental neuronal glut3 gene transcription and expression.

  14. These observations collectively support a temporal contribution by transcription toward ensuring adequate tissue-specific, developmental (placenta and embryonic brain), and postnatal hypoxic brain GLUT3 expression.

  15. In glut3(+/-) mice, a key role of placental Glut3 in mediating transplacental and intraplacental glucose transport was established.

  16. Hesperetin and hesperidin downregulate Akt, GLUT3, and GLUT4 of the insulin signaling pathway in Abeta1-42-induced Neuro-2A cells.

  17. This study demonistrated that Glut3 haploinsufficiency does not impair brain glucose uptake or utilization.

  18. This is the first description of GLUT3 deficiency that forms a possible novel genetic mechanism for pervasive developmental disorders, such as the neuropsychiatric autism spectrum disorders

  19. phosphorylated CREB and Sp3 induce GLUT 3 expression in response to development/cell differentiation and neurotransmission

  20. crosstalk between an imprinted growth demand gene (Igf2) and placental supply transporter genes (Slc38a4, Slc38a2, and Slc2a3) may be a component of the genetic control of nutrient supply and demand during mammalian development

Pig (Porcine) Solute Carrier Family 2 (Facilitated Glucose Transporter), Member 3 (SLC2A3) interaction partners

  1. These findings indicated that heat shock (HS)-induced autophagy regulates lactate secretion by inhibiting apoptosis and increasing mRNA transcript and protein levels of SLC2A3, LDHA, and SLC16A1, which suggests that HS-induced autophagy may enhance lactate secretion by sertoli cells.

  2. Anti-GLUT3 predominantly labels axonal bundles. TEM immunolabeling with colloidal gold displays a very specific distribution of GLUT-1 in the membranes of vascular endothelial cells and in periaxonal astrocyte expansions

Cow (Bovine) Solute Carrier Family 2 (Facilitated Glucose Transporter), Member 3 (SLC2A3) interaction partners

  1. Low GLUT1 and GLUT3 expression in nonclassical monocytes was unaltered during differentiation into macrophages. GLUT4 mRNA was only detectable in unstimulated macrophages. Neither monocytes nor macrophages were insulin responsive.

  2. GLUT3 gene expression increased during late lactation.

  3. distinct regulation by glucose deprivation in chromaffin cells

SLC2A3 抗原简介

蛋白简介

Facilitative glucose transporter. Probably a neuronal glucose transporter.

Gene names and symbols associated with SLC2A3

  • solute carrier family 2 (facilitated glucose transporter), member 3a (slc2a3a) 抗体
  • solute carrier family 2 member 3 (SLC2A3) 抗体
  • solute carrier family 2 (facilitated glucose transporter), member 3 (Slc2a3) 抗体
  • solute carrier family 2 member 14 (SLC2A14) 抗体
  • solute carrier family 2, facilitated glucose transporter member 3 (LOC100125981) 抗体
  • solute carrier family 2 (facilitated glucose transporter), member 3 (SLC2A3) 抗体
  • AA408729 抗体
  • AL023014 抗体
  • AL024341 抗体
  • AU040424 抗体
  • C78366 抗体
  • CEF-GT3 抗体
  • GLUT-3 抗体
  • Glut3 抗体
  • SLC2A3 抗体
  • SLC2A14 抗体
  • wu:fb92c06 抗体
  • zgc:92476 抗体

Protein level used designations for SLC2A3

solute carrier family 2 (facilitated glucose transporter), member 3 , solute carrier family 2, facilitated glucose transporter member 3 , zglut3 , GLUT-3 , glucose transporter type 3, brain , Solute carrier family 2 A3 (neuron glucose transporter) , solute carrier family 2, member 2 , solute carrier family 2, member 3 , glucose transporter type 3 , neuron glucose transporter 3 , Solute carrier family 2, facilitated glucose transporter member 3 , glucose transporter 3

GENE ID SPECIES
436916 Danio rerio
451818 Pan troglodytes
6515 Homo sapiens
20527 Mus musculus
25551 Rattus norvegicus
396517 Gallus gallus
403997 Canis lupus familiaris
100626468 Sus scrofa
282358 Bos taurus
100125981 Oryctolagus cuniculus
443308 Ovis aries
100174668 Pongo abelii
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