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Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases.
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Conclusively, these data demonstrate the MCPIP1 (显示 ZC3H12A 蛋白) contributes to attenuate influenza A virus-induced host antiviral response by suppressing RIG-I (显示 DDX58 蛋白) expression.
Oncostatin M (显示 OSM 蛋白) induces RIG-I (显示 DDX58 蛋白) and MDA5 (显示 IFIH1 蛋白) expression and enhances the double-stranded RNA response in fibroblasts.
Identified the tumor suppressor RARRES3 as a critical target of G9a (显示 EHMT2 蛋白). Epigenetic silencing of RARRES3 contributed to the tumor-promoting function of G9a (显示 EHMT2 蛋白)
Results provide evidence that RIG-I (显示 DDX58 蛋白) as an essential mediator for influenza A virus-induced COX-2 expression.
perifascicular pattern of RIG-I (显示 DDX58 蛋白) expression supports the diagnosis of dermatomyositis
The current study establishes that hypoxia can profoundly influence the inducible RIG-I (显示 DDX58 蛋白) protein expression in malignant cells of both human and murine origin, whereas this phenomenon was not identified in nonmalignant cell lines or primary cells.
our study demonstrates that the novel pathway lncRNA Ftx/miR (显示 MLXIP 蛋白)-545/RIG-I (显示 DDX58 蛋白) promotes hepatocellular carcinoma development
this review describes antiviral activities of RIG-I (显示 DDX58 蛋白) against influenza viruses (standing on three legs)
Study uncovered a novel aspect of Rig-I (显示 DDX58 蛋白) in monitoring gut (显示 GUSB 蛋白) microbiota through regulating IgA and IL6 (显示 IL6 蛋白)-STAT3 (显示 STAT3 蛋白)-dependent Reg3gamma pathway. Besides, Rig-I (显示 DDX58 蛋白) loss could also promote colorectal cancer progression both in the presence and absence of intestinal bacteria.
this study indicated that increased expression of TIG3 in primary glioblastoma is a novel biomarker for predicting poor outcome of patients. We then hypothesize that TIG3 may function in a different pattern in glioblastoma
Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. These effects are mediated by specific nuclear receptor proteins that are members of the steroid and thyroid hormone receptor superfamily of transcriptional regulators. RARRES1, RARRES2, and RARRES3 are genes whose expression is upregulated by the synthetic retinoid tazarotene. RARRES3 is thought act as a tumor suppressor or growth regulator.
HRAS-like suppressor 4
, RAR-responsive protein TIG3
, retinoic acid receptor responder protein 3
, retinoic acid-inducible gene 1
, retinoid-inducible gene 1 protein
, tazarotene-induced gene 3 protein
, retinoic acid receptor responder (tazarotene induced) 3