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Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. 再加上，我们可以发Mago-Nashi Homolog, Proliferation-Associated (Drosophila) 蛋白 (14)和数多这个蛋白质的别的产品。
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Human Polyclonal MAGOH Primary Antibody for ELISA, WB - ABIN268734
Zhao, Colaizzo-Anas, Nowak, Shows, Elliott, Aplan: The mammalian homologue of mago nashi encodes a serum-inducible protein. in Genomics 1998
findings show 2 genes MAGOH and MAGOHB (显示 MAGOHB 抗体) are expressed in mammals; in macrophages, expression of MAGOHB (显示 MAGOHB 抗体) but not MAGOH mRNA increases after LPS (显示 IRF6 抗体) stimulation; both MAGOH proteins interact with other exon junction complex (EJC) components, incorporate into mRNA-bound EJCs and activate nonsense-mediated decay
The stable association of multiprotein exon junction complex core with RNA is maintained by inhibition of eIF4AIII (显示 EIF4A3 抗体) ATPase (显示 DNAH8 抗体) activity by MAGOH-Y14 (显示 RBM8A 抗体).
crystal structure of a tetrameric exon junction core complex containing the DEAD-box adenosine triphosphatase eukaryotic initiation factor 4AIII (显示 EIF4A3 抗体) bound to an ATP analog, MAGOH, Y14 (显示 RBM8A 抗体), a fragment of MLN51 (显示 CASC3 抗体), and a polyuracil mRNA mimic
These results indicate that MAGOH regulates the transcriptional activation of STAT3 (显示 STAT3 抗体) by interfering complex formation between STAT3 (显示 STAT3 抗体) and Y14 (显示 RBM8A 抗体).
we first show that Eif4a3 (显示 EIF4A3 抗体) haploinsufficiency phenocopies aberrant neurogenesis and microcephaly of Magoh and Rbm8a (显示 RBM8A 抗体) mutant mice.we show that genetic ablation of one downstream pathway, p53 (显示 TP53 抗体), significantly rescues microcephaly of all 3 EJC mutant
findings show 2 genes MAGOH and MAGOHB (显示 MAGOHB 抗体) are expressed in mammals; in macrophages, expression of MAGOHB (显示 MAGOHB 抗体) but not MAGOH mRNA increases after LPS (显示 TLR4 抗体) stimulation; both MAGOH proteins interact with other exon junction complex (EJC) components, incorporate into mRNA-bound EJCs and activate nonsense-mediated decay
These results point to a central role for Magoh in melanocyte development.
mouse magoh is involved in cyclin-dependent kinase (显示 CDK1 抗体) regulation.
Magoh haploinsufficiency causes microcephaly because of intermediate neural progenitors depletion and neuronal apoptosis.
Zygotic mago nashi is expressed into the dorsal-marginal region during gastrulation.
MAPK (显示 MAPK1 抗体) is the primary functional target of mago in eye development; in cultured cells, Mago knockdown disproportionately affects other large genes located in heterochromatin
Mago Nashi, Tsunagi/Y14 (显示 RBM8A 抗体), and Ranshi form a complex that influences oocyte differentiation in Drosophila melanogaster
We have determined the crystal structure of the complex between Drosophila melanogaster Y14 (显示 RBM8A 抗体) and Mago at a resolution of 2.5 A. The structure reveals an atypical mode of protein-protein recognition mediated by an RNA-binding domain (RBD (显示 CACNA1D 抗体)).
mago nashi is required for germline stem cell differentiation, but surprisingly mago nashi functions independently of tsunagi/Y14 (显示 RBM8A 抗体) in this process
Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. This gene encodes the mammalian mago nashi homolog. In mammals, mRNA expression is not limited to the germ plasm, but is expressed ubiquitously in adult tissues and can be induced by serum stimulation of quiescent fibroblasts.
protein mago nashi homolog
, mago-nashi-like proliferation-associated protein
, mago-nashi homolog, proliferation-associated
, mago-nashi homolog, proliferation-associated (Drosophila)
, mago nashi
, CG9401 gene product from transcript CG9401-RA
, mago nishi
, mago nashi homolog