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Leukocyte Ig-like receptors (LIRs) are a family of immunoreceptors expressed predominantly on monocytes and B cells and at lower levels on dendritic cells and natural killer (NK) cells. 再加上，我们可以发Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 蛋白 (14) 和 Leukocyte Immunoglobulin-Like Receptor, Subfamily A (Without TM Domain), Member 3 试剂盒 (7)和数多这个蛋白质的别的产品。
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Human Monoclonal LILRA3 Primary Antibody for ELISA, WB - ABIN564777
An, Chandra, Piraino, Borges, Geczy, McNeil, Bryant, Tedla et al.: Soluble LILRA3, a potential natural antiinflammatory protein, is increased in patients with rheumatoid arthritis and is tightly regulated by interleukin 10, tumor necrosis factor-alpha, and ... in The Journal of rheumatology 2010
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Human Polyclonal LILRA3 Primary Antibody for WB - ABIN1881498
Arm, Nwankwo, Austen: Molecular identification of a novel family of human Ig superfamily members that possess immunoreceptor tyrosine-based inhibition motifs and homology to the mouse gp49B1 inhibitory receptor. in Journal of immunology (Baltimore, Md. : 1950) 1997
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Mutations in the genes glucokinase regulatory protein (GCKR (显示 GCKR 抗体)), RNase L (RNASEL (显示 RNASEL 抗体)), leukocyte immunoglobulin-like receptor 3 (显示 LILRB3 抗体) (LILRA3), and dynein axonemal heavy chain 10 (DNAH10 (显示 DNAH10 抗体)) segregated with elevated HDLc levels in families, while no mutations associated with low HDLc.
The homozygous LILRA3 deletion is associated with a higher susceptibility for HIV disease and with a faster disease progression.
Letter: LILRA3 gene deletion is not involved in the giant cell arteritis and systemic sclerosis predisposition in Spanish patients.
LILRA3 significantly reversed Nogo (显示 RTN4 抗体)-66-mediated inhibition of neurite outgrowth and promoted synapse formation in primary cortical neurons through regulation of the ERK (显示 EPHB2 抗体)/MEK (显示 MAP2K1 抗体) pathway.
Experiments point towards a beneficial role for LILRA3 in virus infections, especially in ssRNA viruses, like HIV, that engage TLR8 (显示 TLR8 抗体); however, the potentially beneficial role of LILRA3 is abrogated during a HIV infection.
LILRA3 gene deletion was not associated with Multiple Sclerosis susceptibility and did not affect the age of disease onset, clinical subtype or disease severity.
Evidence showed lack of significant association between LILRA3 deletion and multiple sclerosis pathogenesis. [meta-analysis]
LILRA3 is a new susceptibility factor for systemic lupus erythematosus (SLE) and primary Sjogren's syndrome (pSS (显示 CDSN 抗体)). It predisposes to certain phenotypes such as leucopenia, thrombocytopenia, autoantibody positivity and increased disease activity.
The current study was conducted to investigate the association of rs103294 of LILRA3 with benign prostatic hyperplasia risk.
ILT6 deletion polymorphism does not appear to be a lupus susceptibility gene in South Indian Tamils, but may behave as a genetic modifier of autoantibody phenotype by influencing the production of anti-Ro60 (显示 TROVE2 抗体) and anti-Ro52 (显示 TRIM21 抗体) autoantibodies
Leukocyte Ig-like receptors (LIRs) are a family of immunoreceptors expressed predominantly on monocytes and B cells and at lower levels on dendritic cells and natural killer (NK) cells. All LIRs in subfamily B have an inhibitory function (see, e.g., LILRB1, MIM 604811). LIRs in subfamily A, with short cytoplasmic domains lacking an immunoreceptor tyrosine-based inhibitory motif (ITIM) and with transmembrane regions containing a charged arginine residue, may initiate stimulatory cascades (see LILRA1, MIM 604810). One member of subfamily A (LILRA3) lacks a transmembrane region and is presumed to be a soluble receptor (Borges et al., 1997
CD85 antigen-like family member E
, immunoglobulin-like transcript 6
, leucocyte immunoglobulin-like receptor
, leukocyte immunoglobulin-like receptor 4
, leukocyte immunoglobulin-like receptor A3
, leukocyte immunoglobulin-like receptor subfamily A member 3
, monocyte inhibitory receptor HM43/HM31