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Transcription factor involved in regulating gene activity following the primary growth factor response. 再加上，我们可以发JUNB 抗体 (251) 和 JUNB 蛋白 (8)和数多这个蛋白质的别的产品。
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It has been shown that the AP-1 (显示 FOSB ELISA试剂盒) family member JunB and retinoic acid receptor alpha (RARa (显示 RARA ELISA试剂盒)) mediate catalase (显示 CAT ELISA试剂盒) transcriptional activation and repression, respectively, by controlling chromatin remodeling through a histone deacetylases-dependent mechanism.
JunB neddylation mediated by Itch promotes its ubiquitination-dependent degradation.
a specific role for AP-1/JunB in multiple myeloma cell proliferation, survival and drug resistance.
BATF/JUN (显示 JUN ELISA试剂盒)-B and BATF/C-JUN (显示 JUN ELISA试剂盒) complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.
VEGF (显示 VEGFA ELISA试剂盒)-induced endothelial migration is mediated primarily by induction of JunB whereas the promotion of endothelial proliferation by VEGF (显示 VEGFA ELISA试剂盒) is mediated by JunB-independent AP-1 (显示 FOSB ELISA试剂盒) family members.
Results suggested that JunB could play an important role in promoting cell invasion, migration and distant metastasis in head and neck squamous cell carcinoma via pathways other than epithelial-to-mesenchymal transition.
Highly recurrent mutation of JUNB is associated with nodular lymphocyte predominant Hodgkin lymphoma.
ETS2 (显示 ETS2 ELISA试剂盒), HNF4A (显示 HNF4A ELISA试剂盒) and JUNB are synergistic master regulators of epithelial-to-mesenchymal transition in cancer.
CARMA1 (显示 CARD11 ELISA试剂盒)- and MyD88 (显示 MYD88 ELISA试剂盒)-dependent activation of Jun (显示 JUN ELISA试剂盒)/ATF-type AP-1 (显示 FOSB ELISA试剂盒) complexes is a hallmark of ABC (显示 ABCB6 ELISA试剂盒) diffuse large B-cell lymphomas.
PDK1 (显示 PDK1 ELISA试剂盒) functions as a tumor promoter in human gallbladder cancer by upregulating JunB, promoting epithelial mesenchymal transformation, and cell migration.
Data demonstrate for the first time an essential role of JunB-CBFbeta (显示 CBFB ELISA试剂盒) signaling for maintaining sarcomere architecture and function.
JunB is a regulator of tail organization possibly through integration of several morphogen (显示 SHH ELISA试剂盒) signaling pathways.
The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 (显示 JUN ELISA试剂盒) DNA-binding activity and expression of several transcriptions factors in breast cancer cells.
Data show that JunB is a nonredundant regulator of transcriptional programs that support Th17 cell identity and restrain alternative Th1 (显示 HAND1 ELISA试剂盒) and Treg cell fates.
the present data demonstrates for the first time that JunB plays an important role in the formation of embryonic vascular networks.
JUNB is a significant modulator of both classical and alternative macrophage activation.
Study shows that myeloid deletion of JUNB dampens immune polarization and reshapes disease outcomes during infection with both P. berghei and N. brasiliensis by limiting type 1 and type 2 responses, respectively. Thus, JUNB is an important regulator of myeloid responses to both type 1 and type 2 infections in vivo.
Loss of JunB expression led to increased proliferation and decreased senescence, likely owing to decreased p16(Ink4a) and p21(CIP1 (显示 CDKN1A ELISA试剂盒)) in epithelial cells.
JunB and c-Jun (显示 JUN ELISA试剂盒) expression in post-mitotic oligodendrocytes is mostly dispensable for the maintainance of white matter tracts throughout adult life, even under demyelinating conditions.
JunB controls epidermal growth, barrier formation, and proinflammatory responses through direct and indirect mechanisms, pinpointing SQSTM1 (显示 SQSTM1 ELISA试剂盒) as a key mediator of JunB suppression of NF-kappaB (显示 NFKB1 ELISA试剂盒)-dependent inflammation
our results suggest a functional cooperation between NFAT1 (显示 NFAT1 ELISA试剂盒) and JunB in mediating IL-31 (显示 IL31 ELISA试剂盒) gene expression in CD4 (显示 CD4 ELISA试剂盒)(+) T cells
an important role of the A2B (显示 ADORA2B ELISA试剂盒) receptor-dependent upregulation of JunB in VEGF (显示 VEGFA ELISA试剂盒) production and possibly other AP-1 (显示 JUN ELISA试剂盒)-regulated events.
Transcription factor involved in regulating gene activity following the primary growth factor response. Binds to the DNA sequence 5'-TGATCA-3'.
activator protein 1
, transcription factor jun-B
, Jun-B oncogene
, jun B proto-oncogene
, jun-B proto-oncogene