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The protein encoded by IL22RA1 belongs to the class II cytokine receptor family, and has been shown to be a receptor for interleukin 22 (IL22). 再加上，我们可以发IL22 Receptor alpha 1 蛋白 (13) 和 IL22 Receptor alpha 1 试剂盒 (4)和数多这个蛋白质的别的产品。
Showing 10 out of 116 products:
Mouse (Murine) Monoclonal IL22 Receptor alpha 1 Primary Antibody for CyTOF, FACS - ABIN4899326
Li, Burns, Miller, Smith: CCL20, γδ T cells, and IL-22 in corneal epithelial healing. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2011
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Human Monoclonal IL22 Receptor alpha 1 Primary Antibody for CyTOF, FACS - ABIN4899325
Perriard, Mathias, Enz, Canales, Schluep, Gentner, Schaeren-Wiemers, Du Pasquier: Interleukin-22 is increased in multiple sclerosis patients and targets astrocytes. in Journal of neuroinflammation 2015
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Human Monoclonal IL22 Receptor alpha 1 Primary Antibody for FACS - ABIN4895922
Maarof, Bouchet-Delbos, Gary-Gouy, Durand-Gasselin, Krzysiek, Dalloul: Interleukin-24 inhibits the plasma cell differentiation program in human germinal center B cells. in Blood 2010
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Human Polyclonal IL22 Receptor alpha 1 Primary Antibody for IF (p), IHC (p) - ABIN748178
Guo, Wu, Cen, Li, Kong, Zhou: IL-22-producing Th22 cells play a protective role in CVB3-induced chronic myocarditis and dilated cardiomyopathy by inhibiting myocardial fibrosis. in Virology journal 2014
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Human Monoclonal IL22 Receptor alpha 1 Primary Antibody for FACS - ABIN4895924
Tohyama, Hanakawa, Shirakata, Dai, Yang, Hirakawa, Tokumaru, Okazaki, Sayama, Hashimoto: IL-17 and IL-22 mediate IL-20 subfamily cytokine production in cultured keratinocytes via increased IL-22 receptor expression. in European journal of immunology 2009
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increased expression of IL-22Ralpha therefore promotes keratinocyte proliferation and pro-inflammatory cytokine production during UVB-induced skin inflammation, suggesting that UVB facilitates skin inflammation by increasing the responsiveness of keratinocytes to IL-22 (显示 IL22 抗体)
IL-22 (显示 IL22 抗体) and its receptor have a crucial role in the development and pathogenesis of uveitis by facilitating inflammatory cell infiltration.
phosphorylation of STAT3 (显示 STAT3 抗体) and AKT (显示 AKT1 抗体) was highly induced by treatment with IL-22 (显示 IL22 抗体) via IL-22R1. IL-22R1 was also consistently overexpressed in recurrent NSCLC tissues
These data suggest that imbalance of IL-22 (显示 IL22 抗体)/IL-22R1 signaling regulates the pathogenesis of chronic rhinosinusitis with nasal polyps, including local eosinophilia, via alteration of MUC1 (显示 MUC1 抗体) expression.
FBXW12 (显示 FBXW12 抗体) functions as an E3 ligase constituent to ubiquitinate and degrade IL-22R and that therapeutic FBXW12 (显示 FBXW12 抗体) inhibition may enhance IL-22 (显示 IL22 抗体) signaling and bolster mucosal host defense and infection containment.
IL-22R1 is over-expressed in primary Sjogren's syndrome and Sjogren-associated non-Hodgkin lymphomas and is regulated by IL-18 (显示 IL18 抗体).
IL-22 (显示 IL22 抗体) has restricted tissue specificity as its unique receptor IL-22R1 is exclusively expressed on epithelial and tissue cells, but not immune cells.
serum IL-22 (显示 IL22 抗体) levels are significantly higher in patients with pituitary macroadenomas than in healthy controls and IL-22R is variably expressed in both prolactinoma and non-functioing pituitary adenoma cells.
High expression of interleukin-22 (显示 IL22 抗体) receptor is associated with pancreatic ductal adenocarcinoma.
Letter: calcipotriol inhibits the IL-22 (显示 IL22 抗体)-induced epidermal hyperplasia by decreasing keratinocyte proliferation and down-modulating IL-22R expression.
Endogenous IL-22 (显示 IL22 抗体) and hepatic IL-22R signaling play critical roles in controlling pneumococcal lung burden, and systemic IL-22 (显示 IL22 抗体) decreases bacterial burden in the lungs and peripheral organs by potentiating C3 opsonization on bacterial surfaces, through the increase of hepatic C3 expression.
our data suggest that IL-20 (显示 IL20 抗体) subfamily cytokines, particularly IL-20 (显示 IL20 抗体), IL-22 (显示 IL22 抗体), and IL-24 (显示 IL24 抗体), might provide therapeutic benefit for patients with Diabetic foot ulcers (DFU) .
this study shows that IL-22 (显示 IL22 抗体) plays role in acute graft versus host disease via IL-22R-STAT3 (显示 STAT3 抗体) pathway
RNA sequencing analyses of primary colonic organoids showed that IL-22RA1 signaling promotes intestinal fucosylation via induction of the fucosyltransferase Fut2 (显示 FUT2 抗体).
These findings reveal the ability of GSK-3beta to modulate IL-22R protein stability that might have significant implications for cytoprotective functions and therapeutic targeting of the IL-22 (显示 IL22 抗体) signaling axis.
IL-22R was up-regulated in lymphoid organs and joints during the course of arthritis.
This study documents a previously unknown role of IL-22R1 in inflammation and identifies the involvement of IL-22R1/IL-22 (显示 IL22 抗体) in anaplastic lymphoma kinase-positive anaplastic large cell lymphoma
Genomic structure and inducible expression of the IL-22 (显示 IL22 抗体) receptor alpha chain (显示 FCGRT 抗体).
SNPs in IFNG (显示 IFNG 抗体), IFNGR1 (显示 IFNGR1 抗体) and R2, IL22 (显示 IL22 抗体), and IL22RA1 were analyzed for an association to Estimated breeding values for somatic cell score in Canadian Holstein bulls; no significant associations were found.
The protein encoded by this gene belongs to the class II cytokine receptor family, and has been shown to be a receptor for interleukin 22 (IL22). IL22 receptor is a protein complex that consists of this protein and interleukin 10 receptor, beta (IL10BR/CRFB4), a subunit also shared by the receptor complex for interleukin 10 (IL10). This gene and interleukin 28 receptor, alpha (IL28RA) form a cytokine receptor gene cluster in the chromosomal region 1p36.
interleukin 22 receptor, alpha 1
, IL-22 receptor subunit alpha-1
, cytokine receptor class-II member 9
, cytokine receptor family 2 member 9
, interleukin-22 receptor subunit alpha-1
, interleukin-22 receptor alpha chain