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The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. 再加上，我们可以发HAVCR1 试剂盒 (84) 和 HAVCR1 蛋白 (36)和数多这个蛋白质的别的产品。
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Human Monoclonal HAVCR1 Primary Antibody for CyTOF, FACS - ABIN4899580
Ivie, Fennessey, Sheng, Rubin, McClain: Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin. in PLoS ONE 2011
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Human Monoclonal HAVCR1 Primary Antibody for CyTOF, FACS - ABIN4899579
Kuroda, Fujikura, Noyori, Kajihara, Maruyama, Miyamoto, Yoshida, Takada: A polymorphism of the TIM-1 IgV domain: implications for the susceptibility to filovirus infection. in Biochemical and biophysical research communications 2014
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Mouse (Murine) Polyclonal HAVCR1 Primary Antibody for ICC, IHC - ABIN1077715
Schindler, Bondeva, Schindler, Claus, Franke, Wolf: Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2016
Data suggest that T-cell immunoglobulin domain and mucin (显示 SLC13A2 抗体) domain containing protein 1 (TIM1 (显示 TIMELESS 抗体)) to be under positive natural selection in primates.
By preventing ERK1/2 (显示 MAPK1/3 抗体) phosphorylation following renal injury, STAT3 (显示 STAT3 抗体) phosphorylation is decreased, leading to less phosphorylated STAT3 (显示 STAT3 抗体) within the nucleus, and subsequently less KIM-1 mRNA increases post injury
Study used a previously described highly mobile membrane mimetic membrane in combination with a conventional lipid bilayer model to generate a membrane-bound configuration of Tim1 (显示 ARHGEF5 抗体) in silico, identified two possible states for a membrane-bound form of Tim1 (显示 ARHGEF5 抗体).
Our data reveal a previously unknown role for Galpha12 (显示 GNA12 抗体) in regulating efferocytosis and that renal tubular epithelial cells require KIM-1 to mediate this process.
Urinary L-FABP (显示 FABP1 抗体), NGAL (显示 LCN2 抗体), Kim-1 and albumin (显示 ALB 抗体) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (显示 HAND1 抗体)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (显示 LDLR 抗体) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
data suggest that TIM-1 signaling plays a direct role in Breg maintenance and induction both under physiological conditions (in response to ACs (显示 Acsl1 抗体)) and in response to therapy through TIM-1 ligation
Deletion of the mucin (显示 SLC13A2 抗体) domain impaired KIM-1-mediated phagocytic function, resulting in increased proinflammatory cytokine production, decreased antiinflammatory growth factor secretion by proximal epithelial cells, and an increase in tissue macrophages.
Tim-1 is critical for maintaining self-tolerance by regulating IL-10 (显示 IL10 抗体) production in Bregs
Tim-1 expression was lower in a herpes simplex virus-induced Behcet's disease (BD) mouse model compared to that in asymptomatic BD normal (BDN) mice.
Report detection of drug-induced acute kidney injury in humans by combining the sensitivity of urinary KIM-1 along with urinary miR (显示 MLXIP 抗体)-21, -200c, and -423.
measurement of urinary and renal KIM-1 level may be helpful to evaluate severity of renal pathological damage and prognosis in adult Henoch-Schonlein purpura patients with nephritis
Urine KIM-1 level in acute kidney injury patients was significantly higher than that in the healthy controls.
PCNSL is characterized by frequent Tim-1 (显示 ARHGEF5 抗体) expression, and its soluble form in CSF (显示 CSF2 抗体) may become a useful biomarker for PCNSL.
TIM-1 (显示 ARHGEF5 抗体) is a unique marker for the identification of a human IL-10 (显示 IL10 抗体)(+) Breg subpopulation which is partially superimposed with transitional B cells
in the current meta-analysis, based on ten prospective studies involving 29366 participants, we evaluated the role of urinary tubular injury markers (NGAL (显示 LCN2 抗体), KIM-1 and NAG (显示 NAGLU 抗体)) in predicting clinical outcomes including CKD stage 3, end stage renal disease and mortality.
Urinary KIM-1 levels in the acute kidney injury (AKI) preterm infant group were not significantly higher than in no-AKI group on day of life 1, 3 and 7.
We conclude that KIM-1 might serve as a sensitive biomarker to screen children for kidney damage induced by environmental toxic agents, such as chromium and arsenic
These results suggest that a 6-amino acid deletion polymorphism in the mucin (显示 SLC13A2 抗体) domain of TIM-1 (显示 ARHGEF5 抗体) protects from HIV-1 infection with a recessive effect.
The present study shows that serum and urine levels of NGAL (显示 LCN2 抗体) and KIM-1 are higher in patients with acute kidney injury than in those without acute kidney injury, and that serum NGAL (显示 LCN2 抗体) and the presence of chronic kidney disease are significant predictors of acute kidney injury in scrub typhus.
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, hepatitis A virus cellular receptor 1 homolog
, kidney injury molecule 1
, t cell immunoglobulin and mucin domain-containing protein 1
, t cell membrane protein 1
, T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, rho guanine nucleotide exchange factor 5