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ETFB encodes electron-transfer-flavoprotein, beta polypeptide, which shuttles electrons between primary flavoprotein dehydrogenases involved in mitochondrial fatty acid and amino acid catabolism and the membrane-bound electron transfer flavoprotein u. 再加上，我们可以发Electron-Transfer-Flavoprotein, beta Polypeptide 试剂盒 (38) 和 Electron-Transfer-Flavoprotein, beta Polypeptide 蛋白 (20)和数多这个蛋白质的别的产品。
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Human Polyclonal ETFB Primary Antibody for ELISA, IHC - ABIN4309651
Ohkuma, Noguchi, Sugie, Malicdan, Fukuda, Shimazu, López, Hirano, Hayashi, Nonaka, Nishino: Clinical and genetic analysis of lipid storage myopathies. in Muscle & nerve 2009
ETF (显示 TEAD2 抗体)-beta has a role as part of the electron transfer flavoprotein complex in the catabolism of leucine and involvement in the chlorophyll degradation pathway activated during dark-induced carbohydrate deprivation.
The results identify new protein-protein interactions between AIF (显示 AIFM1 抗体)-Cx43 (显示 GJA1 抗体), ETFB-Cx43 (显示 GJA1 抗体) and AIF (显示 AIFM1 抗体)-ETFB as possible players in the regulation of the mitochondrial redox state.
These results suggested that increasing levels of ATP5B (显示 ATP5B 抗体) and ETFB were associated with worsening renal injury.
the findings of our present study suggest that ETFbeta plays an important role in renal tubular cell apoptosis during the progression of DN.
Electron Transfer Flavoprotein Subunit Beta Is a Candidate Endothelial Cell Autoantigen in Behcet's Disease
Human METTL20 (显示 C12orf72 抗体) is a mitochondrial lysine methyltransferase that targets ETFbeta and modulates its activity.
ETFB knockdown can affect TGF-beta (显示 TGFB1 抗体)-induced tissue remodeling and/or fibrotic processes in vitro.
Point mutations in electron-transfer-flavoprotein beta is associated with destabilized conformations and defective protein:protein interactions leading to mitochondrial dysfunction.
ETFB participates in the mechanoregulation of fibroblast cell number in collagen gel culture.
Data established structural hotspots within the ETF (显示 TEAD2 抗体) fold, and provided a rationale for the prediction of effects of mutations in ETF (显示 TEAD2 抗体).
Tissue samples from 16 unrelated patients with ETF (显示 TEAD2 抗体) deficiency were analysed, two of them harboured mutations in the ETFB gene.
conformational and stability properties of the disease-causing variant ETFbeta-D128N, and our findings on the effect of flavinylation in modulating protein conformational stability and activity.
This gene encodes electron-transfer-flavoprotein, beta polypeptide, which shuttles electrons between primary flavoprotein dehydrogenases involved in mitochondrial fatty acid and amino acid catabolism and the membrane-bound electron transfer flavoprotein ubiquinone oxidoreductase. The gene deficiencies have been implicated in type II glutaricaciduria. Alternatively spliced transcript variants have been found for this gene.
electron transfer flavoprotein subunit beta
, electron transfer flavoprotein beta subunit
, electron transfer flavoprotein beta-subunit
, electron transfer flavoprotein, beta polypeptide
, electron-transferring-flavoprotein, beta polypeptide
, electron transferring flavoprotein, beta polypeptide