Distal-Less Homeobox 3 (DLX3) ELISA试剂盒

Many vertebrate homeo box-containing genes have been identified on the basis of their sequence similarity with Drosophila developmental genes. 再加上,我们可以发DLX3 抗体 (58)DLX3 蛋白 (8)和数多这个蛋白质的别的产品。

list all ELISA KIts 基因 基因ID UniProt
DLX3 1747 O60479
DLX3 13393 Q64205
大鼠 DLX3 DLX3 287638  
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Human Distal-Less Homeobox 3 (DLX3) interaction partners

  1. miR-675 regulates the odontogenic differentiation of human dental pulp cells by epigenetic regulating DLX3.

  2. our studies demonstrate that DLX3 physically interacts with GCM1 and inhibits its transactivation activity, suggesting that DLX3 and GCM1 may form a complex to functionally regulate placental cell function through modulation of target gene expression.

  3. Our research of DLX3 mutation protecting aging-related bone loss opens the possibility of its therapeutic potential in bone regeneration and bone loss disease.

  4. DLX3 expression specifically modulates regulatory networks such as Wnt signaling, phosphatase activity and cell adhesion.

  5. Novel de novo mutation of DLX3 significantly decreases the proliferation rate and inhibits the odontogenic differentiation and mineralization of hDPCs, suggesting that this novel mutation of DLX3 can influence the dentinogenesis in TDO syndrome.

  6. DLX3 regulates bone marrow mesenchymal stem cell proliferation through H19/miR-675 axis.

  7. Data establish the DLX3-p53 interplay as a major regulatory axis in epidermal differentiation and suggest that DLX3 is a modulator of skin carcinogenesis.

  8. Identify a novel cis-acting sequence (-369 to -320) at the placental growth factor promoter, which was critical for mediating the basal and DLX3/GCM1-dependent PGF promoter activities.

  9. We showed that the supplementation of the osteogenic differentiation medium with PTHrP inhibited the alkaline phosphatase activity and the expression of the transcription factor DLX3, but the depletion of PTHrP did not support the differentiation of DFCs.We showed that SUFU (Suppressor Of Fused Homolog) was not regulated during the osteogenic differentiation in DFCs

  10. we identified a recurrent 2-bp deletion in the DLX3 gene in a new family and described their mild clinical phenotype related to the DLX3 mutation.

  11. genetic analysis revealed a novel de novo missense mutation c.533A>G (p.Q178R) in the conserved homeodomain of the DLX3 gene. This DLX3 mutation is the sixth causative mutation for TDO to be identified so far.

  12. ER-alpha regulates the osteoblast differentiation through modulation of Dlx3 expression and/or interaction with Dlx3.

  13. Results suggest that Dlx3 is a novel target of PKA, and that PKA mediates BMP signaling during osteoblast differentiation, at least in part, by phosphorylating Dlx3 and modulating its stability and function.

  14. rs2278163 SNP of DLX3 might be associated with dental caries susceptibility in Japanese children. T and C alleles of rs2278163 SNP may potentially be involved in caries susceptibility and caries protection respectively.

  15. In conclusion, results of our study suggest that the NOTCH-signaling pathway, which is activated during the osteogenic differentiation of DFCs.

  16. DLX3 orchestrates the expression of multiple regulators of trophoblast differentiation and that expression of these regulatory genes is abnormal in fetal growth restriction.

  17. DLX3 stimulates osteogenic differentiation via BMP2 dependent pathway.

  18. Increased DLX3 expression in idiopathic fetal growth restricion (FGR) may contribute to trophoblast dysfunction observed in FGR.

  19. DLX3 acts upstream of syncytin, 3beta-hydroxysteroid dehydrogenase, and the human gonadotropin beta-subunit to play a regulatory role in villous cytotrophoblast differentiation.

  20. DLX3 homeodomain mutations cause tricho-dento-osseous syndrome with novel phenotypes

Mouse (Murine) Distal-Less Homeobox 3 (DLX3) interaction partners

  1. DLX3 is indispensable for the regulation of ion transporters and carbonic anhydrases during the maturation stage of amelogenesis, exerting a crucial regulatory function on pH oscillations during enamel mineralization.

  2. ER-alpha regulates the osteoblast differentiation through modulation of Dlx3 expression and/or interaction with Dlx3.

  3. indicate that Distal-less-3 negatively regulates osteoclastic differentiation through microRNA-124

  4. our data show that DLX3 promotes the expression of the EMP genes Amelx, Enam, Klk4, and Odam in amelogenesis.

  5. Results suggest that Dlx3 is a novel target of PKA, and that PKA mediates BMP signaling during osteoblast differentiation, at least in part, by phosphorylating Dlx3 and modulating its stability and function.

  6. The DLX3 regulates transcription factors crucial for bone formation and DLX3 attenuates bone mass accrual to support bone homeostasis by osteogenic gene pathway regulation.

  7. We show that the deletion of Dlx3 in epidermis and isthums/infundibulum area leads to hair shaft differentiation but not hair growth.

  8. we provide a novel insight that BMP-2-induced Dlx3 expression is regulated by p38/Smad5 signaling pathway in osteoblasts.

  9. used a conditional knockout approach to analyze the effects of neural crest deletion of Dlx3 on craniofacial bones development

  10. regulation of Dlx3 by HR affects the IRS keratin expression, thus modulating the formation of IRS of hair follicle.

  11. The transcription factor Dlx3 is essential in dentin formation by directly regulating a crucial matrix protein.

  12. Dlx3 ablation in epidermis is linked to altered epidermal differentiation, barrier development, and IL-17-associated skin inflammation.

  13. Mutant Dlx3 disrupts odontoblast polarization and dentin formation.

  14. AP-2 gamma and Dlx 3, together with an additional transcription factor(s) that are conserved between humans and mice, are required for trophoblast-specific expression of 3 beta-HSD VI.

  15. Bone morphogenetic protein-2 (BMP-2) transactivates Dlx3 through Smad1 and Smad4: alternative mode for Dlx3 induction in mouse keratinocytes

  16. Genomic structure and functional control of the Dlx3-7 bigene cluster

  17. Gene expression regulation of this protein in visceral arches is evolutionarily conserved.

  18. Results describe gene regulation by Dlx3 in relation to that of Msx2 and Dlx5 during osteoblast differentiation.

  19. DLX3 is one of 3 homeodomain proteins that provide a key series of molecular switches that regulate expression of Runx2 throughout bone formation.

  20. Dlx3 is sufficient to directly modulate expression of the Placental growth factor gene promoter in placental cells.

Xenopus laevis Distal-Less Homeobox 3 (DLX3) interaction partners

  1. these findings, strongly implicate Dlx3 in the regulation of non-neural competence, and show that GATA2 contributes to non-neural competence but is not sufficient to promote it ectopically.

Cow (Bovine) Distal-Less Homeobox 3 (DLX3) interaction partners

  1. study demonstrates the co-expression of DLX3, PPARG and SP1 in trophoblast binucleated cell (BNC)nuclei; this suggests a possible role of these transcription factors through BNC specific genes at the time of pre-placental differentiation

  2. DLX3 has a central role in controlling IFNT gene expression by associating with ETS2 on the IFNT promoter.

Zebrafish Distal-Less Homeobox 3 (DLX3) interaction partners

  1. Foxi1 and Dlx3b/4b regulate the neuronal and sensory lineages of the inner ear.

  2. the expression and function of hand2 and dlx3b/4b/5a genes specify major patterning domains along the dorsoventral axis of zebrafish pharyngeal arches

  3. Data show that compromising Fgf3 and Fgf8 signaling or removing dlx3b, dlx4b and sox9a genes together blocks ear development.

  4. Data suggest that the contribution of dlx3b and dlx4b to neural plate border formation is partially non-cell-autonomous acting via bone morphogenic protein activity.

  5. Dlx3b provide competence for cells to respond to Fgf and form an otic placode

  6. These results provide insight into the mechanisms of preplacodal region (PPR) specification as well as the role of dlx3b/4b function in PPR and otic placode induction.

DLX3 抗原简介

Antigen Summary

Many vertebrate homeo box-containing genes have been identified on the basis of their sequence similarity with Drosophila developmental genes. Members of the Dlx gene family contain a homeobox that is related to that of Distal-less (Dll), a gene expressed in the head and limbs of the developing fruit fly. The Distal-less (Dlx) family of genes comprises at least 6 different members, DLX1-DLX6. Trichodentoosseous syndrome (TDO), an autosomal dominant condition, has been correlated with DLX3 gene mutation. This gene is located in a tail-to-tail configuration with another member of the gene family on the long arm of chromosome 17. Mutations in this gene have been associated with the autosomal dominant conditions trichodentoosseous syndrome and amelogenesis imperfecta with taurodontism.

Gene names and symbols associated with DLX3

  • distal-less homeobox 3 (DLX3) 抗体
  • distal-less homeobox 3 (Dlx3) 抗体
  • distal-less homeobox 3 (dlx3) 抗体
  • distal-less homeobox 3 L homeolog (dlx3.L) 抗体
  • distal-less homeobox 3b (dlx3b) 抗体
  • ai4 抗体
  • AV237891 抗体
  • dll2 抗体
  • dlx-3 抗体
  • dlx3 抗体
  • id:ibd3531 抗体
  • MGC69301 抗体
  • tdo 抗体
  • wu:fb83f11 抗体
  • xdlx3 抗体
  • zgc:91827 抗体

Protein level used designations for DLX3

homeobox protein DLX-3 , DII C , DLX3 homeodomain containing protein , distal-less homeobox 3 , distal-less homeo box 3 , XDLL-2 , homeobox protein DLL-2 , LOW QUALITY PROTEIN: homeobox protein DLX-3 , distal-less homeobox gene 3b , distal-less homeobox protein 3b , homeobox protein Dlx3b

GENE ID SPECIES
1747 Homo sapiens
13393 Mus musculus
287638 Rattus norvegicus
395590 Gallus gallus
455110 Pan troglodytes
491073 Canis lupus familiaris
594954 Xenopus (Silurana) tropicalis
700831 Macaca mulatta
779428 Xenopus laevis
784366 Bos taurus
100342325 Oryctolagus cuniculus
100735447 Cavia porcellus
101109372 Ovis aries
30585 Danio rerio
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