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AGGF1 encodes an angiogenic factor that promotes proliferation of endothelial cells. 再加上，我们可以发AGGF1 抗体 (91) 和 AGGF1 蛋白 (8)和数多这个蛋白质的别的产品。
Showing 9 out of 31 products:
Human AGGF1 ELISA Kit for Sandwich ELISA - ABIN572416
Xu, Zhou, Wang, Zhao, Li, Zhou, Su, Xu, Xia, Qian, Tu, Xiao, Chen, Chen, Wang: Role of microRNA-27a in down-regulation of angiogenic factor AGGF1 under hypoxia associated with high-grade bladder urothelial carcinoma. in Biochimica et biophysica acta 2014
Bmp-induced activation of beta-catenin (显示 CTNNB1 ELISA试剂盒) through Aggf1 regulates caudal (显示 CAD ELISA试剂盒) vein development by promoting the Nr2f2 (显示 NR2F2 ELISA试剂盒)-dependent differentiation of venous endothelial cells and their survival.
aggf1 is involved in differentiation of both hematopoietic and endothelial lineages and acts upstream of scl (显示 TAL1 ELISA试剂盒), fli1 (显示 FLI1 ELISA试剂盒), and etsrp in specification of hemangioblasts
Our study establishes AGGF1 as an angiogenic factor (显示 VEGFA ELISA试剂盒) with an important role in the specification of vein identity and suggests that AGGF1-mediated AKT (显示 AKT1 ELISA试剂盒) signaling is responsible for establishing venous cell fate
Aggf1 has a role in regulating vascular injury.
High expression of AGGF1 predicts poor prognosis in gastric cancer patients.
AGGF1 gene polymorphism does not affect the risk of varicose veins of the legs in ethnic Russians.
Overexpression of AGGF1 is correlated with angiogenesis in hepatocellular carcinoma.
AGGF1 reduces myocardial apoptosis and inflammation and enhances angiogenesis, leading to decreased infarct size after I/R injury.
results of this study indicate that hypoxia down-regulates expression of the AGGF1 protein, but not AGGF1 mRNA, by inducing expression of miR (显示 MLXIP ELISA试剂盒)-27a
AGGF1 potently attenuated TNF-alpha (显示 TNF ELISA试剂盒) triggered NF-kappaB (显示 NFKB1 ELISA试剂盒) pathway, as indicated by the decreased promoter activity, nuclear distribution and phosphorylation of NF-kappaB (显示 NFKB1 ELISA试剂盒) p65 (显示 GORASP1 ELISA试剂盒) subunit as well as the increased protein level of IkappaBalpha (显示 NFKBIA ELISA试剂盒).
analysis of sphingolipid modulation of angio (显示 VEGFA ELISA试剂盒)genic factor expression in n (显示 ARHGEF16 ELISA试剂盒)euroblastoma
results define VG5Q as an angiogenic factor (显示 VEGFA ELISA试剂盒), establish VG5Q as a susceptibility gene for Klippel-Trenaunay syndrome, and show that increased angiogenesis is a molecular pathogenic mechanism of KTS
VG5Q, E133K is a mutation that causes Klippel-Trenaunay and other overgrowth syndromes.
Together with recent identification of somatic mutations in p110a (encoded by PIK3CA (显示 PIK3CA ELISA试剂盒)), our data establish a potential mechanistic link between AGGF1 and PIK3CA (显示 PIK3CA ELISA试剂盒), the two genes identified for Klippel-Trenaunay syndrome (KTS)
Data indicate that angiogenic factor (显示 VEGFA ELISA试剂盒) Aggf1+/- knockout (KO) mice show reduced autophagy, which was associated with inhibition of angiogenesis and larger infarct areas.
Mechanistically, Aggf1 activated the expression of gluconeogenesis gene and skewed the insulin (显示 INS ELISA试剂盒) signaling pathway to induce insulin (显示 INS ELISA试剂盒) resistance. Taken together, our data suggest that Aggf1 plays a role in steatosis in vivo and as such may be a new target in the development of therapeutics solutions against steatosis.
Sphingolipid modulation of angiogenic factor (显示 VEGFA ELISA试剂盒) expression in neuroblastoma (显示 ARHGEF16 ELISA试剂盒)
This gene encodes an angiogenic factor that promotes proliferation of endothelial cells. Mutations in this gene are associated with a susceptibility to Klippel-Trenaunay syndrome. Pseudogenes of this gene are found on chromosomes 3, 4, 10 and 16.
angiogenic factor with G patch and FHA domains 1
, angiogenic factor VG5Q
, G patch domain-containing protein 7
, vasculogenesis gene on 5q protein